Nicolaus

Member
Hello all

I have been looking into anabolic steroids for about a few years now, well not consistently and I want to take my body to the next level.

I got my lab work done and I have low testosterone, low fsh, and low lh levels. Very low actually, too embarassed to share with the public. Which is odd to me because I think I have a good amount of muscle on me, which was obtained naturally.

I was thinking about taking clomid and arimidex to boost my natural T, but I have come across conflicting information regarding its benefits and side effects of these drugs.

Could anyone shed some hard facts on this? I know general information about these drugs and the effects on the physiology of the body, but could anyone recommend what I should do?

Also, I would like to use anabolic steroids but do not know where to find "gtg" sources. Only one I know of is Onyx but people have convinced me @musclehead320 runs it and is very shady about it. Oh and also where to find SERMs and SARMs.

Do I need to become more of an active member and ask people around me so-rx or what should I do?

Thank you very much
 
1) gtg sources, dude your on a forum where sources can be found, why don't you take a look at the steroid underground sub forum?

2) I'm on Clomid right now, for the sole purpose of boosting T levels. Taking 35mg/eod and just had blood work cameback with LH and fsh above reference range limits as well as T levels being double reference range limits. AKA works great. I feel stronger than I normally do, lifts are going up on the daily despite no changes in diet, in fact my diets been subpar the past week.

If you find Clomid gives you bad side effects you can try tamoxifen too.
 

guyzcool

Member
Hello all

I have been looking into anabolic steroids for about a few years now, well not consistently and I want to take my body to the next level.

I got my lab work done and I have low testosterone, low fsh, and low lh levels. Very low actually, too embarassed to share with the public. Which is odd to me because I think I have a good amount of muscle on me, which was obtained naturally.

I was thinking about taking clomid and arimidex to boost my natural T, but I have come across conflicting information regarding its benefits and side effects of these drugs.

Could anyone shed some hard facts on this? I know general information about these drugs and the effects on the physiology of the body, but could anyone recommend what I should do?

Also, I would like to use anabolic steroids but do not know where to find "gtg" sources. Only one I know of is Onyx but people have convinced me @musclehead320 runs it and is very shady about it. Oh and also where to find SERMs and SARMs.

Do I need to become more of an active member and ask people around me so-rx or what should I do?

Thank you very much

Check out the steroid underground area to find sources and you can find pharmaceutical grade ancillaries from the @pharmacist here, check out his thread. What are you thinking about for a first cycle? And why not just try to get.your test levels up first,.don't kill yourself with aas yet.
 

Nicolaus

Member
1) gtg sources, dude your on a forum where sources can be found, why don't you take a look at the steroid underground sub forum?

2) I'm on Clomid right now, for the sole purpose of boosting T levels. Taking 35mg/eod and just had blood work cameback with LH and fsh above reference range limits as well as T levels being double reference range limits. AKA works great. I feel stronger than I normally do, lifts are going up on the daily despite no changes in diet, in fact my diets been subpar the past week.

If you find Clomid gives you bad side effects you can try tamoxifen too.

Thank you for the response. Geez people are nice here lol.

I don't know where to look because it seems like posting sources in public forums is prohibited. Would you happen to know any? Or any threads I could look at.

And that's great news about the clomid. If it's not too much to ask, could you tell me where you purchased the SERMs?

Thanks man!
 

Nicolaus

Member
Check out the steroid underground area to find sources and you can find pharmaceutical grade ancillaries from the @pharmacist here, check out his thread. What are you thinking about for a first cycle? And why not just try to get.your test levels up first,.don't kill yourself with aas yet.

Appreciate the response man. What is a pharmaceutical ancillary? Like, serms and AIs? Is this guy legit. You're right man, I'd like to try out clomid and other things before I try AAS. Do you happen to be on AAS? Also, what were your experiences with SERMs like clomid?

Thank you
 

Nicolaus

Member
1) gtg sources, dude your on a forum where sources can be found, why don't you take a look at the steroid underground sub forum?

2) I'm on Clomid right now, for the sole purpose of boosting T levels. Taking 35mg/eod and just had blood work cameback with LH and fsh above reference range limits as well as T levels being double reference range limits. AKA works great. I feel stronger than I normally do, lifts are going up on the daily despite no changes in diet, in fact my diets been subpar the past week.

If you find Clomid gives you bad side effects you can try tamoxifen too.
Oh also, I forgot to add. I read that clomid could increase ones SGBH? Thereby lowering bioavailabile test. Have you experienced anything like this? Oh and also risk of gyno. I read taking nolvadex helps reduce gyno risk, but isn't clomid and nolvadex essentially the same thing, so I was thinking arimidex and clomid would be optimal. What are your thoughts?
 

Dw725

Member
Post your lab work. I dont want to jump to conclusions, but if you don't post it it just seems like you're one of the many who claim "low t" in hopes of not getting flamed while searching for advice on aas or how to work around just doing them and still get some benefits. No reason to be embarrassed, theyre just numbers. And don't forget to edit out your personal info if you post them. Seen many make the mistake of not hiding it lately.
 
Oh also, I forgot to add. I read that clomid could increase ones SGBH? Thereby lowering bioavailabile test. Have you experienced anything like this? Oh and also risk of gyno. I read taking nolvadex helps reduce gyno risk, but isn't clomid and nolvadex essentially the same thing, so I was thinking arimidex and clomid would be optimal. What are your thoughts?

Best,
Charles


Not arimidex. I'd recommend Aromasin (chemical name exemestane). I tried to stack exemestane with my Clomid but my blood work indicates my exemestane is bunk stuff so I'm in the process of sourcing it from elsewhere now. Ideally I'd be running Clomid + exemestane.

Exemestane does not cause estrogen rebound effect when you stop taking it. It also is shown to lower SHBG levels. Furthermore it has a metabolite that binds to androgen receptors and seems to elicit androgenic effects. In a study where women were given exemestane for 2 years, the women gained an average of 4.4kg of lean mass while losing nearly 1kg of fat verse the control group if I recall correctly. It's theorized this was caused by the androgenic metabolite. These women were NOT weight training nor doing specific dieting.

Clomid and nolvadex are similar but not the same. They have different preferential binding targets for estrogen receptors and different agonist/antagonist signalling effects. But studies show that both do increase testosterone levels substantially. Some ppl get emotional side effects and/or blurred vision from Clomid, if that happens to you I'd switch over to nolvadex (tamoxifen). Also there's at least 1 study out there that claims Clomid causes a desensitization to LHRH and its effects could dwindle, but there are doctors using Clomid for TRT in patients for years not encountering that, so... *shrug* but supposedly this won't happen with nolvadex. However Clomid seems to cause higher t levels than tamoxifen from what I could tell reading literature.

Personally I'm on Clomid right now but have tamoxifen on hand and may swap over just as an experiment.
 

Nicolaus

Member
Post your lab work. I dont want to jump to conclusions, but if you don't post it it just seems like you're one of the many who claim "low t" in hopes of not getting flamed while searching for advice on aas or how to work around just doing them and still get some benefits. No reason to be embarrassed, theyre just numbers. And don't forget to edit out your personal info if you post them. Seen many make the mistake of not hiding it lately.
I do not mean to sound rude in any way, but what is the point of posting my lab values if anyone can just screenshot them from google or any other place? Genuine question. Nonetheless here are my lab results. Doctors told me I'm in the normal range for everything which I highly disagree with because I am always tired, have no motivation, and no libido. I've had these symptoms before I even knew what low testosterone was. Please help if you could.

[EDIT: Potentially identifying information removed at request of OP.]
 

Nicolaus

Member
Not arimidex. I'd recommend Aromasin (chemical name exemestane). I tried to stack exemestane with my Clomid but my blood work indicates my exemestane is bunk stuff so I'm in the process of sourcing it from elsewhere now. Ideally I'd be running Clomid + exemestane.

Exemestane does not cause estrogen rebound effect when you stop taking it. It also is shown to lower SHBG levels. Furthermore it has a metabolite that binds to androgen receptors and seems to elicit androgenic effects. In a study where women were given exemestane for 2 years, the women gained an average of 4.4kg of lean mass while losing nearly 1kg of fat verse the control group if I recall correctly. It's theorized this was caused by the androgenic metabolite. These women were NOT weight training nor doing specific dieting.

Clomid and nolvadex are similar but not the same. They have different preferential binding targets for estrogen receptors and different agonist/antagonist signalling effects. But studies show that both do increase testosterone levels substantially. Some ppl get emotional side effects and/or blurred vision from Clomid, if that happens to you I'd switch over to nolvadex (tamoxifen). Also there's at least 1 study out there that claims Clomid causes a desensitization to LHRH and its effects could dwindle, but there are doctors using Clomid for TRT in patients for years not encountering that, so... *shrug* but supposedly this won't happen with nolvadex. However Clomid seems to cause higher t levels than tamoxifen from what I could tell reading literature.

Personally I'm on Clomid right now but have tamoxifen on hand and may swap over just as an experiment.

Appreciate the response and studies. So, is it a definitive fact that Clomid causes esteogen rebound effects? I don't think you said that directly but assuming you implied it. Also, why not take both nolvadex and clomid? And why not Arimidex.

I believe one of the members posted a source above us, called Pharmacy and they seem to sell Exemestane on their thread called European Pharmaceuticals.
 

guyzcool

Member
Appreciate the response man. What is a pharmaceutical ancillary? Like, serms and AIs? Is this guy legit. You're right man, I'd like to try out clomid and other things before I try AAS. Do you happen to be on AAS? Also, what were your experiences with SERMs like clomid?

Thank you
Yes, in on test e, 500mg/wk to boost athletic performance, and @pharmacist is the most legit thing you'll find on here. Real pharmacuticals from a pharmacy. I have some of his stuff on order, and will be taking it soon. I can comment on it then.
 
Appreciate the response and studies. So, is it a definitive fact that Clomid causes esteogen rebound effects? I don't think you said that directly but assuming you implied it. Also, why not take both nolvadex and clomid? And why not Arimidex.

I believe one of the members posted a source above us, called Pharmacy and they seem to sell Exemestane on their thread called European Pharmaceuticals.


Your confused. Clomid and nolvadex are SERMs Selective Estrogen Receptor Modulators. It means they bind to estrogen receptors and modulate their activity. Essentially they are synthetic estrogens.

AIs are aromatase enzyme inhibitors. A certain % of your testosterone will convert into estrogen thanks to the aromatase enzyme. When you increase your testosterone you'll also end up with more estrogens thanks to the aromatase enzyme. There are two types of AIs one type called reversible binds TEMPORARILY with the aromatase enzyme and prevents it from doing its job. The body will respond by trying to make more aromatase enzyme, this isn't an issue so long as you keep taking the AI but once you stop all that once inactive enzyme becomes active again only now you have way more if it In Your body because your body went and made unnaturally high levels of it. This causes really high levels of estrogen temporarily and is what I mean when I say estrogen rebound. Arimidex (anastrozole) and Femara (letrozole) are this reversible type of AI.

Aromasin (exemestane) is an irreversible aromatase inhibitor. It binds with the enzyme and permanently deactivates it, forever. Once you stop the AI none of the former enzyme re activates, instead the body MUST make new aromatase enzyme. The advantage of this is if you were to miss a dose or something you won't run the risk of getting an estrogen rebound effect. It takes I believe 2-3 days for the body to restore the aromatase enzyme levels. Also as I said exemestane appears to lower SHBG and has a metabolite called 17-hydroxyexemestane that has androgenic effects and may help with your physique goals.


The Pharmacist seems good to go so you will be in good hands with him. I've personally got an order of Clomid coming from him at the moment and I've not seen anyone say a bad thing about him.
 
I should also note to you. There should be a "stacking" effect on t levels by using both a SERM and an AI.

The SERM will cause more LH which will cause more testosterone production. The AI will stop a % of that testosterone from being converted into estrogen.

Let's say the SERM made your T go from 100 to 200, but your aromatase converts 50% to estrogens. You'd really only have 100 T from that 200. But then you add an AI and stop the 50% conversion, now you'd have T at 200. Random numbers, but to illustrate the effect.
 

Nicolaus

Member
I should also note to you. There should be a "stacking" effect on t levels by using both a SERM and an AI.

The SERM will cause more LH which will cause more testosterone production. The AI will stop a % of that testosterone from being converted into estrogen.

Let's say the SERM made your T go from 100 to 200, but your aromatase converts 50% to estrogens. You'd really only have 100 T from that 200. But then you add an AI and stop the 50% conversion, now you'd have T at 200. Random numbers, but to illustrate the effect.

Okay wow, interesting. Ok so, Clomid stimulates FSH and LH levels because this drug is a competitive inhibitor of estradiol and blocks estrogen from binding to the hypothalamus. Since I have low test, fsh, and LH, I can only conclude that I may have secondary hypogonadism. That's what I need, an increase in FSH and LH which will ultimately increase testosterone levels. And like you said, stacking clomid with Exemestane will only help due to a possible sustained or decreased SGBH levels, which will help with more bioavailabile testosterone. And you are not taking nolvadex because clomid is good enough, correct?
Would you happen to know if clomid and nolvadex are very similar in action/chemical structure, thereby rendering the usage of both unncessary?

Also, would you happen to know the proper dosage and duration of clomid/AIs and if discontinued, I would go back to my normal test levels? I have read in some articles that the benefits are permanent, due to a "jumpstart" kind of effect from clomid on the hypothalamus-pituitary axis.


Thank you so much for the help
 

Nicolaus

Member
@Megadick3000

Forgot to add.

Why do not people just take the irreversible AI instead of reversible AI such as arimidex? Are there worse side effects on the aromasin?

I read that aromasin is steroidal and arimidex is non-steroidal. Not sure the significance of this statement.
 

guyzcool

Member
@Megadick3000

Forgot to add.

Why do not people just take the irreversible AI instead of reversible AI such as arimidex? Are there worse side effects on the aromasin?

I read that aromasin is steroidal and arimidex is non-steroidal. Not sure the significance of this statement.
I take aromisin, I like it a lot, if I start getting a lump under my nipples I up my dosage and it goes away almost next day
 
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