??? for einstein

[pumppumppuk]

i agree with most of all your post, and the others i dont know enough to agree or disagree...lol.

in the last years ive used 10mg ed during my cycles with proper pct.

im getting things together for my summer cycle (i can list it if you want), but i was thinking of trying .50mg of arimidex ed or eod on this one.

i see you mention nolva a lot, but never a-dex. why? i know how differently they work, and figured i would give it a try because it seemed it might be a better option just to block production instead of blocking the binding, since you would have a build up that would have to be dealed with during pct.

just wanted to pick your brain.

went ahead and posted cycle since i had to do an edit.


1-10 arimidex .5mg ED or EOD
1-4 50mg dbol ed
1-10 250 mg test e. 2Xweek
1-5 400mg deca
5-10 125mg tren e. 2Xweek
5-10 50mg winny eod
5-12 2.5mg bromo IF NEEDED FOR PROLACTIN


pct
days 1 10 hcg 500iu ed
days 1 10 nolva 20mg
day 11 150mg clomid
day 12 - 18 100mg clomid
day 19 34 50mg clomid




PPP

 

[krom]

i agree with most of all your post, and the others i dont know enough to agree or disagree...lol.

in the last years ive used 10mg ed during my cycles with proper pct.

im getting things together for my summer cycle (i can list it if you want), but i was thinking of trying 50mg of arimidex ed or eod on this one.

i see you mention nolva a lot, but never a-dex. why? i know how differently they work, and figured i would give it a try because it seemed it might be a better option just to block production instead of blocking the binding, since you would have a build up that would have to be dealed with during pct.

just wanted to pick your brain.



PPP

I think you meant .50mg ed or eod not 50mg

 

[pumppumppuk]

yes, you are right....thanx.....50mg would be quite costly.


PPP

 

[einstein1905]

I always use a minimum of 0.25mg/day of Adex and 10mg/day of nolva....all the way through pct, with the exception that nolva gets bumped to 20mg/day at that time. My test/wk dosage is never even 700mg/wk, so these doses work well for me. many factors will come into play as far as dosing the AI....age, bf, test dose, and just inherent aromatase activity. Nonetheless, the doses I listed should be the minimum for any cycle IMO, and there isn't really any legit argument against it. With research sites all over the place, price and availability aren't issues, so there's no good excuse. AIs have been shown to increase IGF-1 levels, and it's known that reduced estrogen decreases SHBG, which results in more free AAS. Nolva is the perfect complement for lipid reasons as well as preventing gyno symptoms.

 

[pumppumppuk]

why both a-dex and nolva thru the cycle. ive always read, told, and believed that nolva decreased IGF-1 levels, but always seemed argued to what degree. if a-dex stops production why add nolva or just add nolva for lipid levels.

dont get me wrong im not trying to argue. just trying to get your prospective because you are def very knowledgable on these subjects.


thanx


PPP

 

[Southernjuice]

I know you are'nt looking for feedback on the other parts of your cycle but gotta ask....why EOD on the winny rather than ED.

 

[einstein1905]

why both a-dex and nolva thru the cycle. ive always read, told, and believed that nolva decreased IGF-1 levels, but always seemed argued to what degree. if a-dex stops production why add nolva or just add nolva for lipid levels.

dont get me wrong im not trying to argue. just trying to get your prospective because you are def very knowledgable on these subjects.


thanx


PPP

Nolva's effects on IGF-1 are not clear. there are an equal number of studies that show both increases and decreases in IGF-1 levels with tamoxifen administration. So, IMO, it's a wash. the effects on IGF-1, if any at all, are hepatic IGF-1, and not muscular IGF-1, so it's debatablehow significant the effects would be anyway.

Another common misconception is that AIs prevent the formation of estrogen. they compete as substrate for aromatase and inhibit formation in a dose-dependent manner. Therefore, the dose will determine the level of inhibition. We want estrogen....and lots of it. estrogen has excellent anabolic properties. What we want is as high of levels of estrogen as possible without experiencing estrogenic sides and also controlling SHBG levels.. The doses of AIs we use will only control estrogen levels, they won't come close to suppressing them to or below normal physiological levels, in the doses I suggest. Our goal is to control estrogen to a degree that we can reap its benefits yet avoid its detriments.

 

[pumppumppuk]

thanx for the info E.

Southern Juice,
actually three reasons,
1st i like stick over oral on winny
2nd im a pussy with winny sticks
3rd im mainly using it to help combat some of the sides from the tren and eod works well for me on this. some will argue about the justification on this reason, but i like it.


PPP

 

[Southernjuice]

thanx for the info E.

Southern Juice,
actually three reasons,
1st i like stick over oral on winny
2nd im a pussy with winny sticks
3rd im mainly using it to help combat some of the sides from the tren and eod works well for me on this. some will argue about the justification on this reason, but i like it.


PPP

Well EOD will help with the sides from anadrol use but don't see it working for tren....but maybe I'm lost. Ok bro, just wondering.