Loving the injectable superdrol, is stacking injectable orals less harsh on liver and body then stacking orals?
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Probably, but I would be more concerned about the chronic inflammation from the solvents used in injectable oral formulations. Since the drug does not have a long aliphatic tail frequently seen in esterified drugs (which increases their solubility in oils), ethyl oleate and/or guaiacol are typically used as cosolvents, and they have a tendency to increase C-reactive protein.Loving the injectable superdrol, is stacking injectable orals less harsh on liver and body then stacking orals?
Nah. While injectable orals bypass first pass metabolism, the 17AA compounds are marked by high oral (>90%) bioavailability, long half-life (hours), and due to this are still metabolized primarily by the liver. So, hepatic strain is virtually identical via the intramuscular route versus oral.Loving the injectable superdrol, is stacking injectable orals less harsh on liver and body then stacking orals?
