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Interesting Soy Study...
- Thread starter SPE
- Start date
In the first place, Asian diets are not "high in soy". That is a myth. And most of the soy they do eat has been fermented, whihc is different than eating the soy protein which is being pushed on us.
Soy acts as estrogen in the body. I do not recommend any male ever take soy protein.
Soy acts as estrogen in the body. I do not recommend any male ever take soy protein.
ok
I took it off because the further I was reading into other studies, the more negatives I saw and just said screw it. It made me think that soy isoflavones could act similar to DIM?? Anyways, here ya go:
Cancer Res. 2000 Mar 1;60(5):1299-305. Related Articles, Links
Increased urinary excretion of 2-hydroxyestrone but not 16alpha-hydroxyestrone in premenopausal women during a soya diet containing isoflavones.
Lu LJ, Cree M, Josyula S, Nagamani M, Grady JJ, Anderson KE.
Department of Preventive Medicine and Community Health, The University of Texas Medical Branch, Galveston 77555-1110, USA. LLu@UTMB.EDU
Asian diets high in soy are associated with lower risk for breast cancer compared with Western diets. Moreover, higher levels of two putative carcinogenic metabolites of 17beta-estradiol, 4- and 16alpha-hydroxyestrogen, and lower amounts of anticarcinogenic metabolites, 2-hydroxyestrogens, have been associated with greater breast cancer risk. In this study, we tested the hypothesis that consumption of a soya diet containing the weakly estrogenic isoflavones genistein and daidzein may alter the metabolism of 17beta-estradiol to 2- and 16alpha-hydroxylated products. Eight pre-menopausal women were placed on a soya-containing, constant diet in a metabolic unit. The diet provided 400 kilocalories from soymilk and 113-202 mg/day (158 +/- 26 mg/day, mean +/- SD) isoflavones daily for a complete menstrual cycle. After a washout period of 4 months, the subjects consumed the same diet, but with soymilk that contained <4.5 mg/day isoflavones ("isoflavone-free"). Urine samples were collected for 24 h daily for the entire cycle during each soya diet period for the analysis of daidzein, genistein, and 2- and 16alpha-hydroxyestrone. Subjects excreted measurable amounts of daidzein (11.6-39.2 mg/day) and genistein (2.9-18.2 mg/day) during the isoflavone-rich soya diet but not during the isoflavone-free soya diet. The diet rich in isoflavones increased the cycle mean daily urinary excretion of 2-hydroxyestrone (averaged over the entire cycle) from 11.6 +/- 2.06 to 17.0 +/- 2.96 nmol/12-h (P = 0.03), a 47% increase. However, the mean daily excretion of 16alpha-hydroxyestrone did not change (7.0 +/- 1.14 nmol/12-h during the isoflavone-free and 7.7 +/- 1.25 nmol/12-h during the isoflavone-rich diet; P = 0.36). The ratio of 2-hydroxyestrone to 16alpha-hydroxyestrone was higher during the isoflavone-rich soya diet (2.6 +/- 0.34) than during the isoflavone-free diet (2.0 +/- 0.32; P = 0.01), a 27% increase. These results suggest that soya isoflavones increase the metabolism of endogenous estrogens to the protective 2-hydroxylated estrogens in women, and this may play an important role in lowering 17beta-estradiol levels and the long-term risk for breast cancer.
I took it off because the further I was reading into other studies, the more negatives I saw and just said screw it. It made me think that soy isoflavones could act similar to DIM?? Anyways, here ya go:
Cancer Res. 2000 Mar 1;60(5):1299-305. Related Articles, Links
Increased urinary excretion of 2-hydroxyestrone but not 16alpha-hydroxyestrone in premenopausal women during a soya diet containing isoflavones.
Lu LJ, Cree M, Josyula S, Nagamani M, Grady JJ, Anderson KE.
Department of Preventive Medicine and Community Health, The University of Texas Medical Branch, Galveston 77555-1110, USA. LLu@UTMB.EDU
Asian diets high in soy are associated with lower risk for breast cancer compared with Western diets. Moreover, higher levels of two putative carcinogenic metabolites of 17beta-estradiol, 4- and 16alpha-hydroxyestrogen, and lower amounts of anticarcinogenic metabolites, 2-hydroxyestrogens, have been associated with greater breast cancer risk. In this study, we tested the hypothesis that consumption of a soya diet containing the weakly estrogenic isoflavones genistein and daidzein may alter the metabolism of 17beta-estradiol to 2- and 16alpha-hydroxylated products. Eight pre-menopausal women were placed on a soya-containing, constant diet in a metabolic unit. The diet provided 400 kilocalories from soymilk and 113-202 mg/day (158 +/- 26 mg/day, mean +/- SD) isoflavones daily for a complete menstrual cycle. After a washout period of 4 months, the subjects consumed the same diet, but with soymilk that contained <4.5 mg/day isoflavones ("isoflavone-free"). Urine samples were collected for 24 h daily for the entire cycle during each soya diet period for the analysis of daidzein, genistein, and 2- and 16alpha-hydroxyestrone. Subjects excreted measurable amounts of daidzein (11.6-39.2 mg/day) and genistein (2.9-18.2 mg/day) during the isoflavone-rich soya diet but not during the isoflavone-free soya diet. The diet rich in isoflavones increased the cycle mean daily urinary excretion of 2-hydroxyestrone (averaged over the entire cycle) from 11.6 +/- 2.06 to 17.0 +/- 2.96 nmol/12-h (P = 0.03), a 47% increase. However, the mean daily excretion of 16alpha-hydroxyestrone did not change (7.0 +/- 1.14 nmol/12-h during the isoflavone-free and 7.7 +/- 1.25 nmol/12-h during the isoflavone-rich diet; P = 0.36). The ratio of 2-hydroxyestrone to 16alpha-hydroxyestrone was higher during the isoflavone-rich soya diet (2.6 +/- 0.34) than during the isoflavone-free diet (2.0 +/- 0.32; P = 0.01), a 27% increase. These results suggest that soya isoflavones increase the metabolism of endogenous estrogens to the protective 2-hydroxylated estrogens in women, and this may play an important role in lowering 17beta-estradiol levels and the long-term risk for breast cancer.
Last edited:
Another...
Cancer Res. 2000 Aug 1;60(15):4112-21. Related Articles, Links
Decreased ovarian hormones during a soya diet: implications for breast cancer prevention.
Lu LJ, Anderson KE, Grady JJ, Kohen F, Nagamani M.
Department of Preventive Medicine, The University of Texas Medical Branch, Galveston 77555, USA. LLu@utmb.edu
Ovarian hormones are biomarkers for breast cancer risk. Soybean consumption may be responsible in part for lower levels of ovarian hormones and decreased rates of breast cancer in women in Asia compared with Western populations. Soybeans contain a significant amount of the isoflavones daidzein and genistein, which are weak estrogens. The purpose of this study was to determine whether soya feeding decreases circulating levels of ovarian hormones and gonadotropins. Ten healthy, regularly cycling women consumed a constant soya-containing diet on a metabolic unit, starting on day 2 of a menstrual cycle until day 2 of the next cycle. Blood and urine samples were obtained daily for one menstrual cycle before and during soy feeding. The diet was calculated to maintain constant body weight, included 400 kilocalories from a 36-ounce portion of soymilk, and provided 113-207 mg/day (154.0+/-8.4 mg/day, mean +/- SE) of total isoflavones. For the group, the soya diet provided more carbohydrate and less protein than the home diets. Daily consumption of the soya diet reduced circulating levels of 17beta-estradiol by 25% (P<0.01, Wilcoxon signed rank test, two-tailed) and of progesterone by 45% (P<0.0001) compared with levels during the home diet period but had no effect on luteinizing hormone or follicle-stimulating hormone. Mean menstrual cycle length did not change during the soya diet; a slight decrease in mean luteal cycle length was marginally statistically significant (P = 0.06). Urinary excretion of isoflavones was 33.8+/-5.3 mg/day (mean +/- SE) and when expressed as percentage of intake, varied substantially (21.9+/-3.3% of intake; range, 9.1-36.7%) among the subjects. Mean daily serum levels of daidzein and genistein (free and conjugated forms) 15 h after soymilk were 2.89+/-0.53 microg/ml and 0.85+/-0.22 microg/ml, respectively, indicating systemic bioavailability of these substances. Secondary analyses by multiple regression showed that decreases in follicular and luteal phase 17beta-estradiol levels were positively associated with urinary isoflavone excretion, an association affected by age, and were inversely associated with decreases in protein intake. Decreases in progesterone levels during the soya diet were inversely associated with increases in intakes of genistein and were affected by the interaction of the intakes of daidzein with energy or with fiber. Consumption of an isoflavone-containing soya diet reduced levels of ovarian steroids in normal women over the entire menstrual cycle without affecting gonadotropins. This suggests that at least under the conditions of this study, soya-induced reductions of circulating ovarian steroids are not mediated by gonadotropins. Decreases in ovarian hormones are related to isoflavones contained in soy and also to energy intake and other components such as protein and fiber but not fat. Our results may explain decreased ovarian hormone levels and decreased risk of breast cancer in populations consuming soya diets and have implications for reducing breast cancer risk by dietary intervention.
Cancer Res. 2000 Aug 1;60(15):4112-21. Related Articles, Links
Decreased ovarian hormones during a soya diet: implications for breast cancer prevention.
Lu LJ, Anderson KE, Grady JJ, Kohen F, Nagamani M.
Department of Preventive Medicine, The University of Texas Medical Branch, Galveston 77555, USA. LLu@utmb.edu
Ovarian hormones are biomarkers for breast cancer risk. Soybean consumption may be responsible in part for lower levels of ovarian hormones and decreased rates of breast cancer in women in Asia compared with Western populations. Soybeans contain a significant amount of the isoflavones daidzein and genistein, which are weak estrogens. The purpose of this study was to determine whether soya feeding decreases circulating levels of ovarian hormones and gonadotropins. Ten healthy, regularly cycling women consumed a constant soya-containing diet on a metabolic unit, starting on day 2 of a menstrual cycle until day 2 of the next cycle. Blood and urine samples were obtained daily for one menstrual cycle before and during soy feeding. The diet was calculated to maintain constant body weight, included 400 kilocalories from a 36-ounce portion of soymilk, and provided 113-207 mg/day (154.0+/-8.4 mg/day, mean +/- SE) of total isoflavones. For the group, the soya diet provided more carbohydrate and less protein than the home diets. Daily consumption of the soya diet reduced circulating levels of 17beta-estradiol by 25% (P<0.01, Wilcoxon signed rank test, two-tailed) and of progesterone by 45% (P<0.0001) compared with levels during the home diet period but had no effect on luteinizing hormone or follicle-stimulating hormone. Mean menstrual cycle length did not change during the soya diet; a slight decrease in mean luteal cycle length was marginally statistically significant (P = 0.06). Urinary excretion of isoflavones was 33.8+/-5.3 mg/day (mean +/- SE) and when expressed as percentage of intake, varied substantially (21.9+/-3.3% of intake; range, 9.1-36.7%) among the subjects. Mean daily serum levels of daidzein and genistein (free and conjugated forms) 15 h after soymilk were 2.89+/-0.53 microg/ml and 0.85+/-0.22 microg/ml, respectively, indicating systemic bioavailability of these substances. Secondary analyses by multiple regression showed that decreases in follicular and luteal phase 17beta-estradiol levels were positively associated with urinary isoflavone excretion, an association affected by age, and were inversely associated with decreases in protein intake. Decreases in progesterone levels during the soya diet were inversely associated with increases in intakes of genistein and were affected by the interaction of the intakes of daidzein with energy or with fiber. Consumption of an isoflavone-containing soya diet reduced levels of ovarian steroids in normal women over the entire menstrual cycle without affecting gonadotropins. This suggests that at least under the conditions of this study, soya-induced reductions of circulating ovarian steroids are not mediated by gonadotropins. Decreases in ovarian hormones are related to isoflavones contained in soy and also to energy intake and other components such as protein and fiber but not fat. Our results may explain decreased ovarian hormone levels and decreased risk of breast cancer in populations consuming soya diets and have implications for reducing breast cancer risk by dietary intervention.
Urinary isoflavonoid phytoestrogen and lignan excretion after consumption of fermented and unfermented soy products.
Hutchins AM, Slavin JL, Lampe JW.
Department of Food Science and Nutrition, University of Minnesota, St. Paul, USA.
OBJECTIVE: To compare the effects of consumption of fermented and unfermented soy products on excretion of urinary isoflavonoid phytoestrogens and lignans in healthy men. DESIGN: A randomized, crossover trial consisting of two 9-day feeding periods following 5 days of baseline data collection. SUBJECTS: Healthy men, aged 20 to 40 years, were recruited from the University of Minnesota Twin Cities community. Of the 22 subjects who began the study, 17 completed all feeding periods. INTERVENTIONS: Fermented soy product (112 g tempeh) or unfermented soy (125 g soybean pieces) was consumed during each controlled feeding period. MAIN OUTCOME MEASURE: Urine samples collected while subjects consumed their habitual diets and on the last 3 days of each feeding period were analyzed for isoflavonoid and lignan content by isotope dilution gas chromatography-mass spectrometry. STATISTICAL ANALYSIS PERFORMED: Comparisons of isoflavonoid and lignan excretion were analyzed using the general linear model procedure. Orthogonal contrasts were used to determine treatment differences of interest. RESULTS: Urinary excretion of isoflavonoids (equol, O-desmethylangolensin [O-DMA], daidzein, genistein) was higher and excretion of lignans (enterodiol, enterolactone) was lower when subjects consumed soy-supplemented diets than when they consumed their habitual diets (P < .05). Urinary isoflavonoid excretion and lignan excretion were similar when subjects consumed tempeh and soybean pieces diets; however, recovery of daidzein and genistein was significantly higher when subjects consumed the tempeh diet than when they consumed the soybean pieces diet (P < .002). When fed soy, 5 of 17 subjects excreted high amounts of equol. These five subjects tended to excrete less O-DMA and daidzein than the 12 subjects who excreted low amounts of equol (P < .06). CONCLUSIONS: Fermentation of soy decreased the isoflavone content of the product fed but increased the urinary isoflavonoid recovery. This finding suggests that fermentation increases availability of isoflavones in soy.
That last part kind of goes against what you were saying SWALE
Hutchins AM, Slavin JL, Lampe JW.
Department of Food Science and Nutrition, University of Minnesota, St. Paul, USA.
OBJECTIVE: To compare the effects of consumption of fermented and unfermented soy products on excretion of urinary isoflavonoid phytoestrogens and lignans in healthy men. DESIGN: A randomized, crossover trial consisting of two 9-day feeding periods following 5 days of baseline data collection. SUBJECTS: Healthy men, aged 20 to 40 years, were recruited from the University of Minnesota Twin Cities community. Of the 22 subjects who began the study, 17 completed all feeding periods. INTERVENTIONS: Fermented soy product (112 g tempeh) or unfermented soy (125 g soybean pieces) was consumed during each controlled feeding period. MAIN OUTCOME MEASURE: Urine samples collected while subjects consumed their habitual diets and on the last 3 days of each feeding period were analyzed for isoflavonoid and lignan content by isotope dilution gas chromatography-mass spectrometry. STATISTICAL ANALYSIS PERFORMED: Comparisons of isoflavonoid and lignan excretion were analyzed using the general linear model procedure. Orthogonal contrasts were used to determine treatment differences of interest. RESULTS: Urinary excretion of isoflavonoids (equol, O-desmethylangolensin [O-DMA], daidzein, genistein) was higher and excretion of lignans (enterodiol, enterolactone) was lower when subjects consumed soy-supplemented diets than when they consumed their habitual diets (P < .05). Urinary isoflavonoid excretion and lignan excretion were similar when subjects consumed tempeh and soybean pieces diets; however, recovery of daidzein and genistein was significantly higher when subjects consumed the tempeh diet than when they consumed the soybean pieces diet (P < .002). When fed soy, 5 of 17 subjects excreted high amounts of equol. These five subjects tended to excrete less O-DMA and daidzein than the 12 subjects who excreted low amounts of equol (P < .06). CONCLUSIONS: Fermentation of soy decreased the isoflavone content of the product fed but increased the urinary isoflavonoid recovery. This finding suggests that fermentation increases availability of isoflavones in soy.
That last part kind of goes against what you were saying SWALE
If anyone knows of a study to back up this statement, then I would love to see the study.SWALE said:Soy acts as estrogen in the body.
RE: Soy
Links to some interesting soy articles.... Personally I find this site (the Weston Proce Foundation) to have a good degree of morals and ethical integrity. And they aren't selling any supplemental products (so there's not that concern about "how much of this BS can I trust)... and they cite a lot of clinical research that isn't "bought and paid for" by any "vested interets"....
http://www.westonaprice.org/soy/soy_controversy.html
http://www.westonaprice.org/soy/tragedy.html
http://www.westonaprice.org/soy/soyandbrain.html
http://www.westonaprice.org/soy/advertising.html
http://www.westonaprice.org/soy/soy_toxicity.html
http://www.westonaprice.org/soy/notmilk.html
http://www.westonaprice.org/soy/darkside.html
http://www.westonaprice.org/soy/ploy.html
http://www.westonaprice.org/soy/soyconquest.html
really good one:
http://www.westonaprice.org/soy/isoflavones.html
Their home page:
http://www.westonaprice.org/splash_2.htm
(List of article Topics are down the left hand side)
Dr. Weston Price wrote a book (I believe it was back in the 1930s) on nutrition and natural diets that is still today simply an excellent read....
"Nutrition and Physical Degeneration"
[ame="http://www.amazon.com/exec/obidos/tg/detail/-/0879838167/qid=1122768480/sr=8-1/ref=pd_bbs_sbs_1/102-8973314-4700135?v=glance&s=books&n=507846"]Amazon.com: Nutrition and Physical Degeneration (9780879838164): Weston Andrew Price: Books[/ame]
Larry
Links to some interesting soy articles.... Personally I find this site (the Weston Proce Foundation) to have a good degree of morals and ethical integrity. And they aren't selling any supplemental products (so there's not that concern about "how much of this BS can I trust)... and they cite a lot of clinical research that isn't "bought and paid for" by any "vested interets"....
http://www.westonaprice.org/soy/soy_controversy.html
http://www.westonaprice.org/soy/tragedy.html
http://www.westonaprice.org/soy/soyandbrain.html
http://www.westonaprice.org/soy/advertising.html
http://www.westonaprice.org/soy/soy_toxicity.html
http://www.westonaprice.org/soy/notmilk.html
http://www.westonaprice.org/soy/darkside.html
http://www.westonaprice.org/soy/ploy.html
http://www.westonaprice.org/soy/soyconquest.html
really good one:
http://www.westonaprice.org/soy/isoflavones.html
Their home page:
http://www.westonaprice.org/splash_2.htm
(List of article Topics are down the left hand side)
Dr. Weston Price wrote a book (I believe it was back in the 1930s) on nutrition and natural diets that is still today simply an excellent read....
"Nutrition and Physical Degeneration"
[ame="http://www.amazon.com/exec/obidos/tg/detail/-/0879838167/qid=1122768480/sr=8-1/ref=pd_bbs_sbs_1/102-8973314-4700135?v=glance&s=books&n=507846"]Amazon.com: Nutrition and Physical Degeneration (9780879838164): Weston Andrew Price: Books[/ame]
Larry
Good One...
Nutr Cancer. 2000;36(1):14-8. Related Articles, Links
Inverse association of soy product intake with serum androgen and estrogen concentrations in Japanese men.
Nagata C, Inaba S, Kawakami N, Kakizoe T, Shimizu H.
Department of Public Health, Gifu University School of Medicine, Japan.
The cross-sectional relationships of soy product intake and serum testosterone, estrone, estradiol, sex hormone-binding globulin, and dihydrotestosterone were examined in 69 Japanese men. Soy product intake was estimated from a semiquantitative food frequency questionnaire. Serum estradiol concentration was significantly inversely correlated with soy product intake (r = -0.32, p = 0.009), and serum estrone concentration was nonsignificantly inversely correlated with soy product intake (r = -0.24, p = 0.05) after controlling for age, body mass index, smoking status, and ethanol intake. Total and free testosterone concentrations were inversely correlated with soy product intake after controlling for the covariates, but these correlations were of border line significance (r = -0.25, p = 0.05 and r = -0.25, p = 0.06, respectively). Similar correlations were observed for these hormones with isoflavone intake from soy products. The data suggest that soy product intake may be associated with the endogenous hormone levels in Japanese men.
PMID: 10798211 [PubMed - indexed for MEDLINE]
Nutr Cancer. 2000;36(1):14-8. Related Articles, Links
Inverse association of soy product intake with serum androgen and estrogen concentrations in Japanese men.
Nagata C, Inaba S, Kawakami N, Kakizoe T, Shimizu H.
Department of Public Health, Gifu University School of Medicine, Japan.
The cross-sectional relationships of soy product intake and serum testosterone, estrone, estradiol, sex hormone-binding globulin, and dihydrotestosterone were examined in 69 Japanese men. Soy product intake was estimated from a semiquantitative food frequency questionnaire. Serum estradiol concentration was significantly inversely correlated with soy product intake (r = -0.32, p = 0.009), and serum estrone concentration was nonsignificantly inversely correlated with soy product intake (r = -0.24, p = 0.05) after controlling for age, body mass index, smoking status, and ethanol intake. Total and free testosterone concentrations were inversely correlated with soy product intake after controlling for the covariates, but these correlations were of border line significance (r = -0.25, p = 0.05 and r = -0.25, p = 0.06, respectively). Similar correlations were observed for these hormones with isoflavone intake from soy products. The data suggest that soy product intake may be associated with the endogenous hormone levels in Japanese men.
PMID: 10798211 [PubMed - indexed for MEDLINE]
SPE said:Nutr Cancer. 2000;36(1):14-8. Related Articles, Links
Inverse association of soy product intake with serum androgen and estrogen concentrations in Japanese men.
Nagata C, Inaba S, Kawakami N, Kakizoe T, Shimizu H.
Department of Public Health, Gifu University School of Medicine, Japan.
The cross-sectional relationships of soy product intake and serum testosterone, estrone, estradiol, sex hormone-binding globulin, and dihydrotestosterone were examined in 69 Japanese men. Soy product intake was estimated from a semiquantitative food frequency questionnaire. Serum estradiol concentration was significantly inversely correlated with soy product intake (r = -0.32, p = 0.009), and serum estrone concentration was nonsignificantly inversely correlated with soy product intake (r = -0.24, p = 0.05) after controlling for age, body mass index, smoking status, and ethanol intake. Total and free testosterone concentrations were inversely correlated with soy product intake after controlling for the covariates, but these correlations were of border line significance (r = -0.25, p = 0.05 and r = -0.25, p = 0.06, respectively). Similar correlations were observed for these hormones with isoflavone intake from soy products. The data suggest that soy product intake may be associated with the endogenous hormone levels in Japanese men.
PMID: 10798211 [PubMed - indexed for MEDLINE]
I imagine that may have been the article referenced on the Weston Price web page - http://www.westonaprice.org/soy/soy_update2001.html
QUOTE:
An article published in Nutrition and Cancer 2000 which found lower testosterone levels and higher estrogen levels in Japanese men who consumed higher levels of soy foods.
END QUOTE
Also:
http://life-enthusiast.net/enzyme/wong_soy.htm
QUOTE:
Instead of helping prevent the bad effects of environmental or natural estrogen dominance soy isoflavones are now known to increase the bad effect of estradiol and estrone the two major bad guys of the estrogen family. (1,2,3). Kills testicular tissue. In men it permanently reduces testicular function and lowers Lutinizing Hormone production. LH is what signals your testicles to work. This increases the probability of estrogen dominance in men with its hair loss, swollen and cancerous prostates. (4,5).
References:
1. Casanova, M., et al.; Developmental effects of dietary phytoestrogens in Sprague -Dawley rats and interactions of genistein and daidzein with rat estrogen receptors alpha and beta in vitro. Toxicol Sci 1999, Oct.; 51 (2): 236-44.
2. Santell, L., et al.: Dietary genistein exerts estrogenic effects upon the uterus, mammary gland and the hypothalamic / pituitary axis in rats. J. Nutr 1997 Feb.;127 (2): 263-9.
3. Harrison, R.M., et al.; Effect of genistein on steroid hormone production in the pregnant rhesus monkey. Proc Soc Exp Biol Med 1999 Oct.; 222(1): 78-84.
4. Nagata, C., et al.; Inverse association of soy product intake with serum androgen and estrogen in Japanese men. Nut Cancer 2000; 36(1): 14-8.
5. Zhong, et al.; Effects of dietary supplement of soy protein isolate and low fat diet on prostate cancer. FASEB J 2000; 14(4): a531.11.
END QUOTE
Also noted this guy sure didn't pull any punches:
QUOTE:
As Americas largest cash crop soy is being touted as having a myriad of health benefits. Far from! Soy is poison, period! All paid for opinion to the contrary... Any opinions to contradict the facts noted above have been paid for by the Agribusiness giants Monsanto and Archer Daniels Midland. Once public knowledge of their manipulation of public opinion and of the FDA becomes widely known, expect monster class action lawsuits against these folks.
END QUOTE
Larry
OK, this is weird.
I am getting websites that take opposing positions on this study. Some of the websites (see above - plus many others*) claim that this study proves that soy is not a beneficial product to be consumming, while other websites turn around and make the opposite claim!
For example:
http://www.lef.org/magazine/mag2003/oct2003_report_andropause_03.htm
QUOTE:
In one study of 97 men, 49-to-72 years of age, the researchers showed that men who had high levels of soy intake had lower levels of estradiol (a form of estrogen) when compared to men with lower levels of soy intake.[21]
Reference: 21. Nagata C, et al. Inverse association of soy product intake with serum androgen and estrogen concentrations in Japanese men. Nutr Cancer 2000; 36(1): 14-18.
END QUOTE
What I found most "interesting" about this LEF article is that they appear to be citing a completely different source or study.
Note that they refer to a "...study of 97 men, 49-to-72 years of age..." and credit that to Reference number 21 (Nagata C, et al. Inverse association of soy product intake with serum androgen and estrogen concentrations in Japanese men) - and yet the actual study itself refers to "relationships of soy product intake and serum testosterone, estrone, estradiol, sex hormone-binding globulin, and dihydrotestosterone were examined in 69 Japanese men..."
Seems to me like LEF "blew it" on that one and cited the wrong study (or whatever).
Larry
(*)
http://www.roex.com/Articles/dangersofisoflavinsarticle.htm
http://home.xtra.co.nz/hosts/soyonlineservice/Male Health.htm
http://home.xtra.co.nz/hosts/soyonlineservice/files/Men1.htm
http://home.xtra.co.nz/hosts/soyonlineservice/Male Health.htm
http://home.xtra.co.nz/hosts/soyonlineservice/Site_Map.htm
http://home.xtra.co.nz/hosts/soyonlineservice/BigBull/Big Ugly Bull.htm
I am getting websites that take opposing positions on this study. Some of the websites (see above - plus many others*) claim that this study proves that soy is not a beneficial product to be consumming, while other websites turn around and make the opposite claim!
For example:
http://www.lef.org/magazine/mag2003/oct2003_report_andropause_03.htm
QUOTE:
In one study of 97 men, 49-to-72 years of age, the researchers showed that men who had high levels of soy intake had lower levels of estradiol (a form of estrogen) when compared to men with lower levels of soy intake.[21]
Reference: 21. Nagata C, et al. Inverse association of soy product intake with serum androgen and estrogen concentrations in Japanese men. Nutr Cancer 2000; 36(1): 14-18.
END QUOTE
What I found most "interesting" about this LEF article is that they appear to be citing a completely different source or study.
Note that they refer to a "...study of 97 men, 49-to-72 years of age..." and credit that to Reference number 21 (Nagata C, et al. Inverse association of soy product intake with serum androgen and estrogen concentrations in Japanese men) - and yet the actual study itself refers to "relationships of soy product intake and serum testosterone, estrone, estradiol, sex hormone-binding globulin, and dihydrotestosterone were examined in 69 Japanese men..."
Seems to me like LEF "blew it" on that one and cited the wrong study (or whatever).
Larry
(*)
http://www.roex.com/Articles/dangersofisoflavinsarticle.htm
http://home.xtra.co.nz/hosts/soyonlineservice/Male Health.htm
http://home.xtra.co.nz/hosts/soyonlineservice/files/Men1.htm
http://home.xtra.co.nz/hosts/soyonlineservice/Male Health.htm
http://home.xtra.co.nz/hosts/soyonlineservice/Site_Map.htm
http://home.xtra.co.nz/hosts/soyonlineservice/BigBull/Big Ugly Bull.htm
Larry...
That is actually what I got from the study as well. The title mentions "Inverse association of soy product intake with serum androgen and estrogen concentrations in Japanese men." SO, if soy intake goes up, then estradiol goes down. Unfortunately same thing for T and DHT. So, at this point I'm at a crossroads leaning towards soy is bad.
That is actually what I got from the study as well. The title mentions "Inverse association of soy product intake with serum androgen and estrogen concentrations in Japanese men." SO, if soy intake goes up, then estradiol goes down. Unfortunately same thing for T and DHT. So, at this point I'm at a crossroads leaning towards soy is bad.
Last edited:
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