low-mid estrone levels on boldenone cyp

[Photon]

So with MENT added on top of this you also have the undetectable methylated E2. Does having your E2 number help you dial things in nonetheless? Like if E2 were 2, you might be able to conclude that the methylated E2 is also very low. If E2 is 37 then you know its not... Do you just have to go by feel.

Methyl e2 doesn't show up, it's completely feels. You might be able to better dial it in by completing crashing your e2 then running MENT. That way you don't need to worry about e2 from aromatization.

 

[LGOP528]

Methyl e2 doesn't show up, it's completely feels. You might be able to better dial it in by completing crashing your e2 then running MENT. That way you don't need to worry about e2 from aromatization.

Completely crushing natural E2 wouldn’t be a good idea. Methyl e2 is ERa selective and you need ERb receptors to be activated to calm your system.

 

[Photon]

Completely crushing natural E2 wouldn’t be a good idea. Methyl e2 is ERa selective and you need ERb receptors to be activated to calm your system.

How'd this work for those who don't run T?

 

[LGOP528]

How'd this work for those who don't run T?

I think we have had this conversation before. There are reports of people having problems without running a test base. There would also not be any DHT without a test base.

 

[LGOP528]

How'd this work for those who don't run T?

The guy from our conversation before was supplying E2. Then the link to the report of ment only cycle is in there too.

Post in thread 'Test/EQ/Dbol'

Sep 16, 2025

HUH?????

Post #73

It’s one guy but I knew I wasn’t crazy.

• LGOP528

• 

 

[Photon]

I think we have had this conversation before. There are reports of people having problems without running a test base. There would also not be any DHT without a test base.

I believe we did! lol

 

[level3]

Alright, I flip flopped back and forth in my head a few times but I decided to drop the hundo and get both the EIA and LC/MS Estrone tests from Labcorp. Bloods on Monday!

 

[SuperJew]

I got back my ~4.5 weeks bloods on Test C 250/Bold C 400/5iu HGH. I was also gobbling HCG and dbol at the time of the test to fight my crashing E2. As you can see: TT: ~1500, Estrone (LC/MS) 18, E2 sensitive 12.7

I wasn't sure whether to add this one my other thread or start a new one. https://thinksteroids.com/community...st-boldenone-hgh-cycle-high-hs-crp.134434822/ But this one has some more information about the cycle.

I will say that leading up to this, my estrogen management was chaotic since it's my first time with bold so I didn't know how it would affect my E2. My impression though was that raising E1 happened to pretty much anyone though.

Based on this thread: https://thinksteroids.com/community/threads/nerd-insight-abt-eq-boldenone.134434099/post-3666080 I am seeing evidence here and elsewhere that LCMS test gets a crashed E1 but the other essay labcorp offers gets a high estrone.

Any reason for me to get another estrone test later after a few weeks of stable hormones or is it simple as that? Those whole time I was expecting a high score.

View attachment 373558
View attachment 373559

How'd you feel with that E2? That's about where I landed when using EQ recently and I felt like I was going insane.

 

[level3]

It came back quick! Blood drawn on February 2, results today on Feb 6. Previous estrone test was on January 7.

Here we are gents:

E2 sensitive:


E1 serum:


E1 LC/MS:


There you have it folks. EIA test gives high estrone, LC/MS test gives low estrone.

Now we just need more folks to do this to confirm I'm not some weird outlier.

For reference, here's a summary of everything estrogen-related on my cycle so far. I did also take dbol here and there to help with low E2 symptoms, but since that is not gonna show up on the tests, I'm leaving it out. If someone can say why it might be relevant though, I'll dig out the data (I log every dose of almost everything I take - the only real exceptions being electrolytes and taurine, which I just do by feel).

December 8-December 30: 300mg Test C/300mg Bold C per week, dosed EOD. 100mg Test U dosed once per week. I was previously on 200mg Test U every week for the previous ~year.
December 8-January 7: Varying HCG doses to try to keep up with crashing E2. Would be annoying to type them all out but again if someone can convince me its worthwhile, I'll do it. I haven't taken any HCG since starting on E2 cyp.
January 1-January 17: 250 Test C/400mg Bold C per week, dosed EOD.
January 19-Present: 125 Test C/125 Test P/400 Bold C per week, dosed ED.
January 7: 1mg E2 Cyp.
January 9-Present: 0.3mg E2 cyp dosed EOD (total of 1.05mg/wk).

One small issue is that there is one dose of E2 on January 25 that I think there's a 50/50 shot was 4x the dose I meant to take (so it would be 1.2mg). This is because I made a new vial that was 4x diluted for more precise dosing, and I just don't remember if I dosed the right one that day. Given that my E2 has come out to ~20, seems unlikely, but its a possibility. I don't think it would majorly affect these numbers, but I want to be precise.

@Type-IIx since you have written probably more technical information about estrogen and boldenone on this forum than anyone, and I've found it very helpful, I wanted to draw your attention here in particular to see if you have any comments.

 

[Photon]

It came back quick! Blood drawn on February 2, results today on Feb 6. Previous estrone test was on January 7.

Here we are gents:

E2 sensitive:
View attachment 377903

E1 serum:
View attachment 377904

E1 LC/MS:
View attachment 377905

There you have it folks. EIA test gives high estrone, LC/MS test gives low estrone.

@Type-IIx since you have written probably more technical information about estrogen and boldenone on this forum than anyone, and I've found it very helpful, I wanted to draw your attention here in particular to see if you have any comments.

Thank you for posting.
This is probably the 4th confirmation we have so far that EQ and its derivatives do not convert to E1.

@dinfar1337
@bigdaddyandfriends
@Type-IIx

Post in thread 'Boldenone (EQ) Misconceptions and Comparison with Metenolone (Primo) [Author: Type-IIx]'

Oct 20, 2025

1. Misconception that boldenone is nonaromatizable, or that its use (in sufficiently high doses) permit abstaining from AI/SERM use in users that are not resilient against aromatizing effects (e.g., fluid retention/edema, growth of breast tissue, etc.)

Reality: Boldenone aromatizes (to estrone [E1] & estradiol [E2]) at 58% the rate of testosterone. This is significant. However, since Estrone (E1) is a weak estrogen, because lacks the 17β-OH group, it possesses only 2% of E2's potency to transactivate ERα (associated with aromatizing effects, many of...

• Photon

• 

So far it seems like those who report an increase, are doing e2 sensitive, e1 non-sensitive.

This article as well.
E2 sensitive, but E1 non-sensitive! Why?!
It's not even the same comparison!

Primobolan / Equipoise Crashed my E2 – Help!

Understand the complex interaction of Primobolan and Equipoise with estrogen levels, symptoms of low estrogens, and practical decision-making strategies for managing these effects in AAS users.

thinksteroids.com

 

[dinfar1337]

Thank you for posting.
This is probably the 4th confirmation we have so far that EQ and its derivatives do not convert to E1.

@dinfar1337
@bigdaddyandfriends
@Type-IIx

Post in thread 'Boldenone (EQ) Misconceptions and Comparison with Metenolone (Primo) [Author: Type-IIx]'

Oct 20, 2025

1. Misconception that boldenone is nonaromatizable, or that its use (in sufficiently high doses) permit abstaining from AI/SERM use in users that are not resilient against aromatizing effects (e.g., fluid retention/edema, growth of breast tissue, etc.)

Reality: Boldenone aromatizes (to estrone [E1] & estradiol [E2]) at 58% the rate of testosterone. This is significant. However, since Estrone (E1) is a weak estrogen, because lacks the 17β-OH group, it possesses only 2% of E2's potency to transactivate ERα (associated with aromatizing effects, many of...

• Photon

• 

So far it seems like those who report an increase, are doing e2 sensitive, e1 non-sensitive.

This article as well.
E2 sensitive, but E1 non-sensitive! Why?!
It's not even the same comparison!

Primobolan / Equipoise Crashed my E2 – Help!

Understand the complex interaction of Primobolan and Equipoise with estrogen levels, symptoms of low estrogens, and practical decision-making strategies for managing these effects in AAS users.

thinksteroids.com

btw i called type2 out for being debunked by more plates more dates and some reddit freaks half year ago and he changed subjects and didnt respond after that. sorry for my dead responses but the bloodwork just confirm what i already looked into xd

we have NOTHING confirming eq aromatizes into estrone besides a vitro rat study with the wrong compound misquoted from a book 20 years after ww2 which everyone has taken for the absolute truth because they cant read eq's chemical name

but the concept of boldeone having 50% of aromatese to testosterone is simply wrong i believe mpmd did a wonderful deep dive into some hidden research on reddit or whatever for it. and the book which stated it mixed it up with another unknown steroid which shares a common name but isnt commonly used

the research we have on boldeone that most guys know is fake info taken by a book with a study for fetus and child growth and boldeone isnt mentioned in that study lmao.

Derek's wrong, Llewellyn's right. Is that surprising?

“Boldenone is considered a mildly estrogenic steroid. Aromatization studies suggest that its rate of conversion to estrad iol is roughly half that of testosterone.1” (Llewellyn, 2017, p. 297)

“4-androstene-3,17-dione” (Gual et al., 1962, p. 923) androstenedione; Adione; ASD shares the same aromatization rate as testosterone, same moiety analogous to 1,4-androstadien-3,17-dione (boldione) vis-a-vis boldenone

This permits us to say that boldenone (EQ) aromatizes at ~ 58% the rate of T (0.35 / 0.60) to estrone and estradiol

you just get a bunch of idiots comparing apples to orangutans online. Joke.

Not being able to understand something so as to not be able to meaningfully critique it in any concrete way, but making a video about it anyway, doesn't mean facts don't exist and that you're not wrong. In fact, they do and he is

but the catch on boldione(1,4-androstadiene-3,17-dione) in vitro and not BOLDEONE(1,4-androstadien-17β-ol-3-one) is right.

although we could compare androstenedione to testosterone and get a reasonable comparison.

i just wanted to higlight these 2 points from my original post, stating what i got from derek he got right, the 50% figure likely isnt accurate. it was in 1962 and he did his best he could but its a hypothesis which isnt right and it seems way more individual than a set number at 50%.

and eq can inhibit estrogen production to almost 0. but its different from person to person.

all other shit he said is bs.

just wanted to highlight the little moment of shine he stole from that redditor from around 6-10 minutes in the video

i also mean't the 50% is pulled out of lewyns a$$.

obviously you're comparing eq without hydroxyl group to testosterone and doing it right.

which is right biochemically, and i see that is what is right on the paper but i just dont believe its right in real life, and the methods is just not 100% fool proof, which is the only thing derek said right in that video partially right he stole from the redditor.