Ids-isys Total Igf-i Assay & Diagnosis And Treatment Of Gh Deficiency

Michael Scally MD

Doctor of Medicine
10+ Year Member
Varewijck AJ, Lamberts SWJ, van der Lely AJ, Neggers SJCMM, Hofland LJ, Janssen JAMJL. The Introduction of the IDS-iSYS Total IGF-I Assay may have Far-reaching Consequences for Diagnosis and Treatment of GH Deficiency. The Journal of Clinical Endocrinology & Metabolism. http://press.endocrine.org/doi/abs/10.1210/jc.2014-2558

Context: Insulin-like growth factor-I (IGF-I) measurements are used for screening and monitoring GH deficiency (GHD) and acromegaly.

Objective: To study whether the introduction of the IDS-iSYS IGF-I assay would lead to different clinical interpretations in GHD and acromegaly.

Design: In 106 GHD subjects and in 15 acromegalic subjects visiting our university hospital, total IGF-I levels were measured before and during therapy by using the Immulite (IM) assay and IDS-iSYS (ID) assay. Z-scores were calculated by using assay-specific age-specific normative range values. All treatment decisions were based upon results obtained by the IM assay.

Results: GHD: 1] Absolute IGF-I concentrations differed significantly between both IGF-I assays before treatment (p<0.001), but not during GH treatment (p= 0.32); 2]. Mean Z-scores for IGF-I differed significantly before starting (IM: -2.23 vs. ID: -1.43 (p<0.001) and during GH treatment (IM: -0.60 vs. ID: +0.21 (p<0.001).

Acromegaly: 1] Absolute IGF-I concentrations did not differ between both IGF-I assays before treatment (p=0.18), but were significantly different during treatment (p= 0.009); 2]. Mean Z-scores for IGF-I were not different before starting (IM: 10.93 vs. ID: 10.78 (p=0.41) or during treatment (IM: 3.60 vs. ID: 3.18 (p=0.23).

Conclusions: In GHD subjects mean IGF-I Z-scores significantly differed when measured by the IM assay compared to the ID assay irrespective of treatment. In contrast, in acromegaly mean IGF-I Z-scores did not differ significantly between both assays. Our study suggests that replacement of the IM assay by the ID assay may have far-reaching consequences for the clinical diagnosis and treatment of GHD.
 
Varewijck AJ, Lamberts SWJ, van der Lely AJ, Neggers SJCMM, Hofland LJ, Janssen JAMJL. The Introduction of the IDS-iSYS Total IGF-I Assay may have Far-reaching Consequences for Diagnosis and Treatment of GH Deficiency. The Journal of Clinical Endocrinology & Metabolism. http://press.endocrine.org/doi/abs/10.1210/jc.2014-2558

Context: Insulin-like growth factor-I (IGF-I) measurements are used for screening and monitoring GH deficiency (GHD) and acromegaly.

Objective: To study whether the introduction of the IDS-iSYS IGF-I assay would lead to different clinical interpretations in GHD and acromegaly.

Design: In 106 GHD subjects and in 15 acromegalic subjects visiting our university hospital, total IGF-I levels were measured before and during therapy by using the Immulite (IM) assay and IDS-iSYS (ID) assay. Z-scores were calculated by using assay-specific age-specific normative range values. All treatment decisions were based upon results obtained by the IM assay.

Results: GHD: 1] Absolute IGF-I concentrations differed significantly between both IGF-I assays before treatment (p<0.001), but not during GH treatment (p= 0.32); 2]. Mean Z-scores for IGF-I differed significantly before starting (IM: -2.23 vs. ID: -1.43 (p<0.001) and during GH treatment (IM: -0.60 vs. ID: +0.21 (p<0.001).

Acromegaly: 1] Absolute IGF-I concentrations did not differ between both IGF-I assays before treatment (p=0.18), but were significantly different during treatment (p= 0.009); 2]. Mean Z-scores for IGF-I were not different before starting (IM: 10.93 vs. ID: 10.78 (p=0.41) or during treatment (IM: 3.60 vs. ID: 3.18 (p=0.23).

Conclusions: In GHD subjects mean IGF-I Z-scores significantly differed when measured by the IM assay compared to the ID assay irrespective of treatment. In contrast, in acromegaly mean IGF-I Z-scores did not differ significantly between both assays. Our study suggests that replacement of the IM assay by the ID assay may have far-reaching consequences for the clinical diagnosis and treatment of GHD.

Creatnine fell from around the 1.88 and 2.00 levels to 1.3. And gfr rose from 32 and 35 to 50. This seemed to be a direct result from HGH (ugl) use. Do you know of any studies that have been done on HGH and kidney failure?
 
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