I wouldn't use T3 if you're off cycle. Especially not during pct or right after.
MESO-Rx
Anabolic Steroids
Prevent Fat Rebound After T3 Cycle
- Thread starter Equinob
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I wouldn't use T3 if you're off cycle. Especially not during pct or right after.
Not exactly. The only reason I"m taking Test is to prevent the loss of LBM while I take the T3 since it is reportedly indiscriminate as to what it catabolizes, not to bulk (It's a poor man's cutting cycle as I couldn't get my hands on Tren, Mast or Var). I had planned on stopping AAS at the same time I stopped T3, and then go on PCT of Nolva and Clomid for 4 weeks following. As I said I've been taking an AI and hcg the entire time, so I should have a fairly easy recovery with little loss of natural test.
The concern is what happens when I stop the T3 cold turkey per the advice given here. So if its going to take 2 weeks to a month (I can't find a clear answer on the web, but 2 weeks seems to be the standard) for my thyroid to get back to normal, the question becomes whether I should continue AAS for another week or two past stopping the T3 before I go on PCT in order to offset the recovery of my thyroid, without dropping calories I need to support my LBM, in order to prevent fat rebound.
Or maybe it's simpler. Maybe I go off T3 & AAS, and up my intake of caffeine, or ephedrine, or other metabolism increasing supplements for the two weeks my thyroid recovers?
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They did a study on patients who took thyroid meds for up to 20yrs and when they came off natural thyroid function came back within 2wks. If you're worried just cease T3 a week or two before your last pin.
Everyone is different. Most seem to have a couple of sluggish days when coming off the t3 and then back to normal. I wouldn't over think it... Just come off of both, start pct, watch your diet, and see how you feel.
I assume this is a 12 week cutting cycle? Where in that cycle do you drop T3?
Janoy crevesa
New Member
How effective is t3 for you guys? I've used it in 50-100mcgs on some oral cycles and 2000 calories and I flattened out and lost tons of strength (and probably muscle). Do you guys take t3 and lose weight on like 3k calories?
I will wait for the drugs to kick in usually 2wks
So on a 12wk cut, weeks 3-7 I will add t3 at 50mcg daily split dosage and just stop cold turkey.
You know if just occurs to me that if I'm speeding up my metabolism to burn more calories, it's probably speeding up the rate at which I metabolize the test too?
Don't overthink this
Haha, that's likely good advice.
I'm still curious about how it works though. I did realize that if the T3 did speed up the rate at which my Test was metabolized, it would also slow down when I dropped the T3.
I was originally going to do just one pin a week since I wasn't trying to gain, but I felt that the drop in hormone levels over the week was leading to muscle catabolism by mid-week (possibly due to the increased metabolism), so I split my dose to twice a week, and that seems to have helped.
For the general question, I'm thinking just stepping up my cardio during the two weeks my thyroid ramps up again should prevent any fat gains as well.
The enzymes that metbaoliZe testosterone and other drugs are in the liver I believe. They have nothing to do with the thyroid.
Haunch
New Member
Good read guys, thanks for the info. My main concern with T3 was about doing permanent damage to my thyroid but according to the study doc mentioned, 20 years of T3 treatment and peoples thyroids snapped back after discontinuing treatment.
I was referring to the Androgenic action of testosterone in the nucleus of the cell, which is regulated by the mitochondria, which is affected by the T3. I think the smaller estrogenic action by way of the amortization of testosterone to estradiol is the only aspect of testosterone metabolized directly by the liver. If I effectively deactivate the aromatase produced by the liver, then this metabolization of the hormone would be minimal during my cycle. The bulk of the conversion of testosterone seems to be regulated by enzymes produced by the adrenal gland, kindneys, cytoplasm, etc., regardless of the ultimate disposition of filtering by the liver. If my understanding of this is lacking, by all means set me straight. But if the primary bonding of testosterone happens in the target cells, and as such would be affected by cell metabolism, then theoretically shouldn't this process be accelerated on T3? Now oral testosterone is metabolized completely differently, but we're talking IM applications here.
