"Ask Chest Rockwell" column - Open thread for member questions

My question for @ChestRockwell would be, "what are the long term potential cardiovascular effects of trenbolone usage, and how can users prevent or mitagate those effects?"

I mean a good example of the latter are questions posed about AAS cardio-toxicity ---- have these "members" even looked at Doc Scally's thread on the subject ---- from the looks of things ---- few have
 
Was running potg switched to elite. I don't use AI at all during this time. Also I get bloods about every 2-3 months. And free Test is much higher while on mast and so is total t.

It's great that you did bloods while on test and mast because I have a question:
I read everywhere that DHT derivatives block E2 at the receptor site but don't deactivate aromatase and T conversion to E2.
Can you attest that Mast didn't lower your E2 value but rather masked E2 symptoms by blocking their activity at the receptor?
 
Not sure if it lowered it or masked it as I have never had high e2 symptoms other than water retention.

It's great that you did bloods while on test and mast because I have a question:
I read everywhere that DHT derivatives block E2 at the receptor site but don't deactivate aromatase and T conversion to E2.
Can you attest that Mast didn't lower your E2 value but rather masked E2 symptoms by blocking their activity at the receptor?[/QUO
 
Not sure if it lowered it or masked it as I have never had high e2 symptoms other than water retention.

Hence my question: what did your blood test show for E2? Lower than prior to Masteron? Unchanged?
 
I've read your article on the fat loss protocol using GH @ChestRockwell and was curious if your opinion on whether clen is the optimal additional add-on with T3/T4 or if albuterol or similar would be as effective or perhaps doesn't work well at all. Looking forward to your next article.
 
I've read your article on the fat loss protocol using GH @ChestRockwell and was curious if your opinion on whether clen is the optimal additional add-on with t3/T4 or if albuterol or similar would be as effective or perhaps doesn't work well at all. Looking forward to your next article.

Clenbuterol will be optimal in the sense that it will produce an environment of significantly greater fat mobilization potential, as compared to something like albuterol.

However, there are many folks who tend to have a hard time handling clenbuterol and therefore albuterol is a reasonable alternative should one fit into that category.
 
Clenbuterol will be optimal in the sense that it will produce an environment of significantly greater fat mobilization potential, as compared to something like albuterol.

However, there are many folks who tend to have a hard time handling clenbuterol and therefore albuterol is a reasonable alternative should one fit into that category.

Thanks for taking the time to answer. I figured you included the clen due to suggesting an optimal protocol.

I was curious about the albuterol as some "experts" lately have suggested it is not useful for fat loss. That has not been my personal experience but I wanted to see how you felt about it. Thanks again.
 
I was curious about the albuterol as some "experts" lately have suggested it is not useful for fat loss.

"Useful" is a relative term, of course. Yes, It is significantly less potent than clenbuterol. However, if I informed someone they could get a 2-5% additive rate upon lipolysis some would see that as pointless while others would see that as amazing. So, it just depends...
 
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