Sure....doesn't everybody know that??
There's a difference between recognizing the effects of something (any dipshit, even you, is capable of this) and fully understanding the reasons for it. Most recognize tren's potent recomping effects (increasing fat-free mass and concomitantly reducing adipose tissue stores). Instead of rolling your eyes at me, you could learn something. But my posts aren't for everybody, I know.
And just the androgenic nature of the compound should give you a more masculine look, which involves burning and/or redistributing fat. People talk about it's "nutrient partitioning" properties but I'm not educated on that.
That likely goes to the latter mechanism mentioned, androgenic commitment of pluripotent mesenchymal cells to a non-adipogenic lineage (i.e., less fat; more "chiseled" look).
Your recent posts herein regarding the metabolic costs of muscle tissue deposition cannot by itself account for the substantial potency of androgens, including T, in increasing FFM while decreasing adipose tissue deposition. Bhasin, et al. hypothesized that, in addition to direct effects on skeletal muscle, T may promote the commitment of pluripotent precursor cells into the myogenic lineage + inhibit their differentiation into the adipogenic lineage. Since this hypothesis was tested, several additional lines of data have borne this out. It's fair to say it's now part of the model of androgen action.
The sweating due to tren is IMO not likely due to increased RMR/REE, as would be implicit given your proposed mechanism. I say this because of the absence of any evidence (at least that I've seen, I think I would have seen it though) that tren increases RMR/REE.
Tren sweats are very real: apparently not a rapid nongenomic effect, but perhaps you are correct that this hypothetical effect is mediated by the AR (i.e., possibly tren IS so potent that your hypothesis is true, that there is increased REE due to increased metabolic processes in building muscle additive to the commitment of precursor cells to a non-adipogenic lineage; I can certainly accept this, but it's theoretical as I have not seen any published data to suggest that tren actually increases RMR/REE).
Perhaps toxicity? Though tren apparently fares better under ADME-Tox standards than Primo (metenolone enanthate), which fails under Lilly Med Chem Rules, the GSK 4/400 Rule, and Pfizer 3/75 Rule, hence its only being commercially available in select few regions. It is, however, noteworthy, that Primo causes night sweats for many users as well & trenbolone acetate is only commercially available for veterinary use.
It's very likely tren potently antagonizes the MR: perhaps this causes
some quantity of dehydration via sweating.
Perhaps this effect is due to activity at the sigma opioid receptor? Very hypothetical this one.
Any thoughts,
@PeterBond on "Tren sweats?"