How much Anavar for big deficit?

one5onecc

Member
Been running anavar for 1 week now at 40mg + 200mg test trying to get into single digits by the new year before I bulk.

40mg is definitely stronger than just being on a cruise, at a 1000-1250 deficit everyday my strength has maintained, I feel fuller than when I was in a surplus with no var and the energy and work capacity is nuts. I can easily lift 1 hour then do another 1 hour of cardio with more walks throughout the day despite being on a big deficit where usually I feel deathly.

Would it make sense to drop my dose to 20 or 30mg and just run it for 30 days at 20mg or 20 days at 30mg days? (my current stash runs out in 15 days at 40mg per day)

Would a lower dose for longer provide more results than the larger dose for a shorter time seeing as it gives me more time in a deficit with gear?


Im currently hovering at 190 roughly 11% bodyfat probably not much more than 4-5lb max to lose before I hit my goal. So not sure whether I should just stick to the high dose and try shift the last 4ishlb in two weeks or give myself a runway and lower the dose to cut longer.
 
That's more than enough. I run 50mg max during contest prep

Just focus on your diet and training. Anavar will help preserve muscle, it doesn't burn fat. At your weight 25mg is totally sufficient to do its job
 
Var is highly underrated. It's great for strength and it's a good selection along with 200 mg test for your cut.

I run Var at 25 mg and think that there is not a significant difference between 25 & 50 mg (very slight). I think the difference between 20 & 30 mg will be even less significant. Doses of 10 - 15 mg are what you usually see in studies.
 
Var is highly underrated. It's great for strength and it's a good selection along with 200 mg test for your cut.

I run Var at 25 mg and think that there is not a significant difference between 25 & 50 mg (very slight). I think the difference between 20 & 30 mg will be even less significant. Doses of 10 - 15 mg are what you usually see in studies.

Hello,

not a significant difference in a cut context on in general ?

For someone training for the purpose of gaining strength and
who has run oxandrolone @25mg per day alongside testosterone in a previous cycle, what would lead to the better strength increase in the following cycle phase:

going up to 50mg/day ?
stay @25mg/day of oxandrolone and add another oral ?
 
Hello,

not a significant difference in a cut context on in general ?

For someone training for the purpose of gaining strength and
who has run oxandrolone @25mg per day alongside testosterone in a previous cycle, what would lead to the better strength increase in the following cycle phase:

going up to 50mg/day ?
stay @25mg/day of oxandrolone and add another oral ?
Within the context of cutting. Less is more during a cut (save your gear). You're leveraging the anticatabolic effects rather than attempting to maximize new muscle gain. A mere 200 mg test enanthate is sufficient in even a severe (>55%) energy deficit to actually build some new muscle.

Test attenuates proteolytic and inflammatory responses by reducing resting skeletal muscle expression of fibroblast growth factor-inducible 14 (Fn14), IL-6 [inflammatory responses to exercise], and muscle ring finger protein 1 (MuRF1) [proteolysis].

Duchaine in USHII suggests a dose of 0.125 mg/lb (0.275 mg/kg) of Anavar for maximizing muscle gains. In a deficit, again, 20 mg is plenty.

If you're going to add any new compound at least use something that is anticatabolic by a different mechanism to Test (e.g., Tren decreases GR number; rhGH increasing circulating FFAs promotes lipid oxidation and reduced gluconeogenesis and is therefore protein sparing; decreases leucine oxidation during exercise, etc.)
 
Within the context of cutting. Less is more during a cut (save your gear). You're leveraging the anticatabolic effects rather than attempting to maximize new muscle gain. A mere 200 mg test enanthate is sufficient in even a severe (>55%) energy deficit to actually build some new muscle.

Test attenuates proteolytic and inflammatory responses by reducing resting skeletal muscle expression of fibroblast growth factor-inducible 14 (Fn14), IL-6 [inflammatory responses to exercise], and muscle ring finger protein 1 (MuRF1) [proteolysis].

Duchaine in USHII suggests a dose of 0.125 mg/lb (0.275 mg/kg) of Anavar for maximizing muscle gains. In a deficit, again, 20 mg is plenty.

If you're going to add any new compound at least use something that is anticatabolic by a different mechanism to Test (e.g., Tren decreases GR number; rhGH increasing circulating FFAs promotes lipid oxidation and reduced gluconeogenesis and is therefore protein sparing; decreases leucine oxidation during exercise, etc.)
20mg really for a harsh cut?

Everyone keeps parroting that over 40mg for men while cutting and some folks even running 75mg+
 
20mg really for a harsh cut?

Everyone keeps parroting that over 40mg for men while cutting and some folks even running 75mg+
75 mg is wasteful and far more impactful to lipids. There's little difference between 40 mg and 20 mg (far less than 2X efficacy).

Evidence of losing fat & building muscle (recomp) with 200 mg weekly TE (testosterone enanthate), 1.6g/kg protein, 30% macros from fat, severe caloric restriction (55% energy deficit) for 28 days:
Test-200mg-caloric-restriction-builds-muscle-Table.MesoRx.png
 
Var is highly underrated. It's great for strength and it's a good selection along with 200 mg test for your cut.

I run Var at 25 mg and think that there is not a significant difference between 25 & 50 mg (very slight). I think the difference between 20 & 30 mg will be even less significant. Doses of 10 - 15 mg are what you usually see in studies.
What specifically makes it great for strength? Always wondered why it compared to the other minimally water retentive drugs.
 
What specifically makes it great for strength? Always wondered why it compared to the other minimally water retentive drugs.
There's no clear structure/activity relationship between 2-oxasteroids and the rapid nongenomic strength increase, but I suspect that it is due to increased neural drive & central motor command.

Nongenomic androgen effects on strength encompass (at least in part) voltage-dependent K⁺ currents, Ca²⁺ channels, neurotransmitters, etc.. Anavar likely potently (> T) influences the nervous components of the neuromuscular system (e.g., motor behavior, neuronal activity, intracellular signaling), ↑cortical & spinal cellular excitability by ↓neuronal excitation threshold.
 
75 mg is wasteful and far more impactful to lipids. There's little difference between 40 mg and 20 mg (far less than 2X efficacy).

Evidence of losing fat & building muscle (recomp) with 200 mg weekly TE (testosterone enanthate), 1.6g/kg protein, 30% macros from fat, severe caloric restriction (55% energy deficit) for 28 days:
View attachment 176056
What's the two columns in this table?
Test = the group with 200mg test
PLA = group without steroids ?
Is the conclusion that they lost 4.7 vs 4.1kg fat, while gained 3.8 vs lost 0.9kg muscle ??
 
What's the two columns in this table?
Test = the group with 200mg test
PLA = group without steroids ?
Is the conclusion that they lost 4.7 vs 4.1kg fat, while gained 3.8 vs lost 0.9kg muscle ??
Yes.

Both groups were in a 55% energy deficit with high volumes of exercise including resistance training. These were active military.
 
There's no clear structure/activity relationship between 2-oxasteroids and the rapid nongenomic strength increase, but I suspect that it is due to increased neural drive & central motor command.

Nongenomic androgen effects on strength encompass (at least in part) voltage-dependent K⁺ currents, Ca²⁺ channels, neurotransmitters, etc.. Anavar likely potently (> T) influences the nervous components of the neuromuscular system (e.g., motor behavior, neuronal activity, intracellular signaling), ↑cortical & spinal cellular excitability by ↓neuronal excitation threshold.
I don't suppose there is any documented information that can give an idea of how effective the common androgens are at that particular effect? Mostly curious about potential additions in future cycles to minimize superfluous water weight and bias strength development (not tren though lol).

If you're going to add any new compound at least use something that is anticatabolic by a different mechanism to Test (e.g., Tren decreases GR number; rhGH increasing circulating FFAs promotes lipid oxidation and reduced gluconeogenesis and is therefore protein sparing; decreases leucine oxidation during exercise, etc.)
Do any of the other androgens typically referred to as cutting drugs, have anticatabolic effects different than those that test, tren and anavar exploit? Or just more of the same mechanisms?
 
There's no clear structure/activity relationship between 2-oxasteroids and the rapid nongenomic strength increase, but I suspect that it is due to increased neural drive & central motor command.

Nongenomic androgen effects on strength encompass (at least in part) voltage-dependent K⁺ currents, Ca²⁺ channels, neurotransmitters, etc.. Anavar likely potently (> T) influences the nervous components of the neuromuscular system (e.g., motor behavior, neuronal activity, intracellular signaling), ↑cortical & spinal cellular excitability by ↓neuronal excitation threshold.
Not to derail the thread, but does Anadrol have any evidence of strength increase? It's the one oral that actually gives me a boost at 100mg Preworkout, might be from the mental effects but I actually feel that one oral.
 
Not to derail the thread, but does Anadrol have any evidence of strength increase? It's the one oral that actually gives me a boost at 100mg Preworkout, might be from the mental effects but I actually feel that one oral.
Only anecdotal. My n=1 is that Anadrol is my single favorite compound for increasing strength, not to diminish any other compound. I love the shit.
 
I don't suppose there is any documented information that can give an idea of how effective the common androgens are at that particular effect? Mostly curious about potential additions in future cycles to minimize superfluous water weight and bias strength development (not tren though lol).


Do any of the other androgens typically referred to as cutting drugs, have anticatabolic effects different than those that test, tren and anavar exploit? Or just more of the same mechanisms?
On both accounts, potency to increase strength by neural mechanisms, & anticatabolic pathways by androgen, the data is just not where I wish it was. We have insight so far into Test & Tren with respect to anticatabolism, and to Test for neural mechanisms that increase strength & partly to its rapid nongenomic effects generally. I'm always scouring for this stuff, but the best we have right now is part conjecture.
 
Deca, but it's not for everybody (mostly because of effects on dopamine metabolism).
Can you open up in this further? I get how significantly different individual responses it.

Typically, medically prescribed Caber for example seems to be in 1mg tablets. But I also get that they’re not designed for people using 19’s. UGL’s make them in 0.25mg tablets.

I don’t think I’ve ever gotten related sided from Tren, definately mental sides and I won’t use it again. But I’m looking into trying some deca late into the spring of next year.
 
What is your consensus on tren for the purposes or what I am doing.

Would a small dose of tren A 10-20mg per day be more effective/equally safe at maintaining muscle on a harsh cut or even gaining muscle than a moderate dose of anavar 30-40mg?
 
Can you open up in this further? I get how significantly different individual responses it.

Typically, medically prescribed Caber for example seems to be in 1mg tablets. But I also get that they’re not designed for people using 19’s. UGL’s make them in 0.25mg tablets.

I don’t think I’ve ever gotten related sided from Tren, definately mental sides and I won’t use it again. But I’m looking into trying some deca late into the spring of next year.
Sure, Deca increases serum homovanillic acid, a marker for dopamine metabolism (i.e., breakdown). Deca, even at 100 mg/w, significantly increases the breakdown of dopamine, and this is almost certainly associated with reduced dopamine receptor number in brain & CNS tissues. That is the likeliest explanation for depression and mood alterations caused by nandrolone.

Tren is not even of the same drug class as nandrolone, despite broscience that classifies these as "19-nortestosterone class" drugs. I wouldn't expect Tren to have the same effect on dopamine; in fact, there's reason to suspect opposite effects, increased dopaminergic activity.

I don't understand the reference to cabergoline. It's used clinically for prolactin-secreting adenomas (nothing at all like the androgen-induced increase to estrogens that increases, in some individuals, prolactin). Cabergoline is never recommended for AAS users.
 
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