A theory about Lipostabil and effectiveness

[Neodavid]

Lipostabil kills fat cells by reacting with the fat inside them.

Fat cells do not divide, you have a fixed number of them after a certain age (unless you go insane and stimulate division by eating past their capacity... which is like HUGE).

Therefore...

The more weight you can lose BEFORE you lipo, the more effective the lipo will be in the long run, BECAUSE, each drop of lipo will kill MANY more tiny fat cells, than it will hugely filled up blimp fat cells.

We are talking a considerable percentage here, up to 300% more effective, if you lose weight first.

What I'm saying is, don't do this on a bulking cycle, but AFTER a cutting cycle. The results will be much greater.

That's my theory, and why I'm waiting another week for lipo, to finnish up my summer cutting cycle. I'll kill 3x the fat cells with the same amount of lipo, by being pretty cut. AND, I'll be able to find those pockets of fat that linger with more accuracy.

 

[GRANVILLE]

Lipostabil kills fat cells by reacting with the fat inside them.

Fat cells do not divide, you have a fixed number of them after a certain age (unless you go insane and stimulate division by eating past their capacity... which is like HUGE).

Therefore...

The more weight you can lose BEFORE you lipo, the more effective the lipo will be in the long run, BECAUSE, each drop of lipo will kill MANY more tiny fat cells, than it will hugely filled up blimp fat cells.

We are talking a considerable percentage here, up to 300% more effective, if you lose weight first.

What I'm saying is, don't do this on a bulking cycle, but AFTER a cutting cycle. The results will be much greater.

That's my theory, and why I'm waiting another week for lipo, to finnish up my summer cutting cycle. I'll kill 3x the fat cells with the same amount of lipo, by being pretty cut. AND, I'll be able to find those pockets of fat that linger with more accuracy.

That would seem an excellent conclusion, the precise mechanism by which lipostabil works is still not fully understood and there are several theories currently floating about. All I can say is that in the lab lipostabil seems to have a biological chemical reaction on the fat in the cell, the reaction takes place (cell expands, hence body swelling) when the reaction is complete the "dissolved" fat leaves the cell quite quickly. It would definitely be beneficial to remove the fat when it is at its most vulnerable, i.e. after a good cutting cycle, I'm sure the reaction would be alot stronger then as well

 

[Neodavid]

Am I mistaken in believing lipostabil also kills the fat cell? I thought for sure it did... TIA

That would seem an excellent conclusion, the precise mechanism by which lipostabil works is still not fully understood and there are several theories currently floating about. All I can say is that in the lab lipostabil seems to have a biological chemical reaction on the fat in the cell, the reaction takes place (cell expands, hence body swelling) when the reaction is complete the "dissolved" fat leaves the cell quite quickly. It would definitely be beneficial to remove the fat when it is at its most vulnerable, i.e. after a good cutting cycle, I'm sure the reaction would be alot stronger then as well

 

[GRANVILLE]

Am I mistaken in believing lipostabil also kills the fat cell? I thought for sure it did... TIA

In the lab the lipostabil doesn't touch cellular material its action is purely as a fat "solvent" via a biochemical action, so the cell remains, otherwise it could also dissolve other organs which it certainly does not.....................................sorry you'll still have to watch your diet lol, still its not likely those cells will fill up with fat as they appear to be left collapsed!!!!

 

[Neodavid]

Oh, okay, right... yeah, that makes sense.

Hey, rendered inert is good enough for me.

If you ever find out for sure, let us know man. That's some pretty cool stuff.

In the lab the lipostabil doesn't touch cellular material its action is purely as a fat "solvent" via a biochemical action, so the cell remains, otherwise it could also dissolve other organs which it certainly does not.....................................sorry you'll still have to watch your diet lol, still its not likely those cells will fill up with fat as they appear to be left collapsed!!!!

 

[einstein1905]

Fat cells have recently been found to be able to secrete a number of different factors.....they are no longer thought of as simply storage cells. Two secreted factors that are most likely relevant here are IL-6 and TNF alpha, both of which are inflammatory mediators. Clearly inflammation occurs when using lipostabil, and it's localized to adipose, since lipostabil doesn't migrate from the injection site.

Now, it has been shown that in individuals with increased levels of insulin resistance that they have much higher levels of IL-6 and TNF-a....both of these factors have an inverse relationship with adiponectin.

Individuals with insulin resistance have higher levels of TNF-a and IL-6 on a systemic level. However, if lipostabil is inducing adipocytes to secrete TNF-a and IL-6 in a more localized fashion, there is potential (and I say potential) for localized insulin resistance, specific to adipose around the areas of lipostabil use......this would be great, since the areas treated are usually the areas most difficult to rid of fat. Increased localized insulin resu=istance would mean that it would be much more difficult to regain fat in these areas......something to think about anyway.

 

[GRANVILLE]

Fat cells have recently been found to be able to secrete a number of different factors.....they are no longer thought of as simply storage cells. Two secreted factors that are most likely relevant here are IL-6 and TNF alpha, both of which are inflammatory mediators. Clearly inflammation occurs when using lipostabil, and it's localized to adipose, since lipostabil doesn't migrate from the injection site.

Now, it has been shown that in individuals with increased levels of insulin resistance that they have much higher levels of IL-6 and TNF-a....both of these factors have an inverse relationship with adiponectin.

Individuals with insulin resistance have higher levels of TNF-a and IL-6 on a systemic level. However, if lipostabil is inducing adipocytes to secrete TNF-a and IL-6 in a more localized fashion, there is potential (and I say potential) for localized insulin resistance, specific to adipose around the areas of lipostabil use......this would be great, since the areas treated are usually the areas most difficult to rid of fat. Increased localized insulin resu=istance would mean that it would be much more difficult to regain fat in these areas......something to think about anyway.

That would certainly be a bonus einstein, and certainly seems more than probable, best keep it quiet or the suppliers will be puting the price up.

 

[Tank01]

That would certainly be a bonus einstein, and certainly seems more than probable, best keep it quiet or the suppliers will be puting the price up.

you know what sucks....

there is some stupid regulation against sending money via WU to Brazil from CA... and they wouldnt give me a reason why... I wanted to get some Procaine and Tiractol ... lame damnit... lame...

 

[Neodavid]

Have no friends in other states?

you know what sucks....

there is some stupid regulation against sending money via WU to Brazil from CA... and they wouldnt give me a reason why... I wanted to get some Procaine and Tiractol ... lame damnit... lame...