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This topic has been discussed AND REFUTED on many Meso threads. I challenge ANYONE to locate and post any 19-NOR/PROLACTIN association in evidence based literature, bc it simply does NOT exist!
One simple factoid; Although Nandrolone has been a legally sold since the 1970s, I could not locate a single case report on MedLine/PubMed which even remotely suggested this 19-nor AAS had ANY effect on serum prolactin levels, NOT ONE CASE!
However in spite of this 19-nor AAS being used for some FIFTY YEARS not one case of prolactin related GCM has been reported in evidence based literature. Yet for some reason "bro-science" prevails and supersedes conventional wisdom and scientific evidence to the contrary. The latter speaks volumes about the amount of misinformation that permeates most PED forums fellas.
Moreover have any of these "believers" investigated the clinical features of patients with Prolactinomas as a baseline comparison, do they understand how much the physiological effects of prolactin DIFFER from that of E-2 on breast tissue, have they EVER performed pre and intra-cycling 19-nor "bloods" to uncover the TRUTH for themselves, is their fund of knowledge so limited as to believe "stimulation of SKM progesterone receptor" can NOT result in enhanced progesterone secretion bc SKM is not a part of the human endocrine system, duh!
Finally ask those on "other forums" to post their evidence and I promise it will be "bloods" that someone else conducted, yet are not available for reproduction. In essence all the "reports" of a 19-NOR / Prolactin association are either anecdotes or a cadre of parrots echoing words of broism, that has now become bro-lore!
Why would that matter as both are 19-nor compounds.
Heck don't believe me, perform your own pre and intra-cycle Prolactin levels while running ANY 19-nor AAS.
(How prophetic is it that not a single person has returned to post values that prove 19--nor raise prolactin levels, NOT A ONE, and I've been waiting 4-5 years!)
Or follow bro-science that has revealed NOTHING to the contrary, except anecdotes whom blindly implicate prolactin as the basis for GCM, when the facts are unequivocal GCM, is an E-2 mediated process.