LABS toward end of Torem PCT, help!!

Yah i wonder if it's just the torem that's bad or all the serms and AI's. Makes me wonder I'm sitting on some of there supplements as well. I wonder if there is any way to test these products, like labmax but for serms.


I don't know, to be fair I've had success with their nolva and clomid. I had to run their adex at .75 mg ED which is kind of high just to keep E2 in check.

Mythotik has had good results with their Torem but as you can see in this thread several others had bad experiences with their Torem and Candy's was from a different batch then mine.

All things considered it's certainly not worth the risk in my opinion!!
 
I'll do bloods in 3 weeks and let you know what there is to be known. I'll have to request copies of my bloods from a couple years ago for baseline too. I've got my fingers crossed. I woke up with a towering hard on and I've been feeling good. Weight is stable, but strength is down a bit.

Hey Candy, what batch number are you running? Mine is FA13082601C
 
Actually Toremifene is better than Tamoxifene for that (receptor blocking) if you read the breast cancer studies. It does aid male fertility, but through a different mechanism of action than Clomid because Clomid is essentially synthetic LH.

So would using Torem on cycle at start of sides backed up with an AI make sense.
Then the old Nolva/Clomid for Pct
 
So would using Torem on cycle at start of sides backed up with an AI make sense.
Then the old Nolva/Clomid for Pct

I hesitate to say yes only because I'm now convinced my original torem was at the very least under dosed, and I was using Adex from another source, but my gyno sides were contained until I discontinued the Adex. Now that I've switched Torem sources I am still asymptomatic for gyno, and have not had any Adex since last Thursday. Blood work is going to be the tell tale here.

My batch number is different. I'll look it up again when I get home.
 
Hey candy- jballz and myself were wondering the rationale behind blood donation in between cycles would be... I heard you mentiin it in another thread- mind jacking this one for a moment to spout some knowledge?
 
Actually Toremifene is better than Tamoxifene for that (receptor blocking) if you read the breast cancer studies. It does aid male fertility, but through a different mechanism of action than Clomid because Clomid is essentially synthetic LH.

Lets not get into a pissing contest but where are you coming up with this? Clomid is in no way a synthtic LH / it isnt even a synthetic LH mimetic. It is made up of 2 isomers with estrogenic agonist antagonist activities. The reason it is effective in fertility more so than other serms is that one isomer induces lower amplitude pulses of GnRH which increaases both LH and FSH. The other induces irregular, large amplitude pulse of GnRH which shifts the LH/FSH ratio towards FSH favor..which results in increased spermatogenisis and sperm motility. Clomid works via negative feedback just like every other serm..not by acting as a "synthetic LH".
 
Lets not get into a pissing contest but where are you coming up with this? Clomid is in no way a synthtic LH / it isnt even a synthetic LH mimetic. It is made up of 2 isomers with estrogenic agonist antagonist activities. The reason it is effective in fertility more so than other serms is that one isomer induces lower amplitude pulses of GnRH which increaases both LH and FSH. The other induces irregular, large amplitude pulse of GnRH which shifts the LH/FSH ratio towards FSH favor..which results in increased spermatogenisis and sperm motility. Clomid works via negative feedback just like every other serm..not by acting as a "synthetic LH".

Check this thread out Jimmy.. I'd be curious to hear your thoughts..

https://thinksteroids.com/community/threads/134349589
 
Lets not get into a pissing contest but where are you coming up with this? Clomid is in no way a synthtic LH / it isnt even a synthetic LH mimetic. It is made up of 2 isomers with estrogenic agonist antagonist activities. The reason it is effective in fertility more so than other serms is that one isomer induces lower amplitude pulses of GnRH which increaases both LH and FSH. The other induces irregular, large amplitude pulse of GnRH which shifts the LH/FSH ratio towards FSH favor..which results in increased spermatogenisis and sperm motility. Clomid works via negative feedback just like every other serm..not by acting as a "synthetic LH".

My point was more that Clomid works to incite the body's production of testosterone where as Tamoxifen works to primarily act as a competitor for breast tissue estrogen receptors and that Toremifene shows the potential to do both of those things.

No worries mate, you're right in your explanation. I won't even take my dick out, let alone enter a pissing contest. I never said you weren't smarter than me, I just called you unhelpful. :)
 
My point was more that Clomid works to incite the body's production of testosterone where as Tamoxifen works to primarily act as a competitor for breast tissue estrogen receptors and that Toremifene shows the potential to do both of those things.

No worries mate, you're right in your explanation. I won't even take my dick out, let alone enter a pissing contest. I never said you weren't smarter than me, I just called you unhelpful. :)

Let me say this in the most helpful way possible - .Thats not accurate either.... at all. You are inaccurately overgeneralizing and unintentionally passing along misinformation. Tamoxifen is extremely effective at inducing test production , it also happens to be extremely effective at preventing gyno. To say it doesnt effectively do both is completely inaccurate. (Perhaps if you knew me you wouldn't have questioned the intention of my first post in this thread and then you could be focusing on the things im posting here..and find I can be very helpful) From now on lets not call each other anything and maybe we can both learn something.
 
I used CEM Torem last year and tested at 700ish a few weeks after PCT. After another cycle I did Torem + Nolva and tested at over 1000ng/dl. Maybe the batch the OP got was bad or it was a bad bottle (I'm sure not every bottle gets teh same concentration. Maybe the mixer stop and the liquid settled and then started pouring again. I'm sure it can happen).

Also, if you you say you ran PCT and didn't lose any gains then you are basically a lying cunt or your gear was bunk. And you have no clue that gear causes water and glycogen retention that is released when gear is no longer administered.

BRB, running cycle, gaining 30lbs and keeping it all a few weeks PCT.
 
I used CEM Torem last year and tested at 700ish a few weeks after PCT. After another cycle I did Torem + Nolva and tested at over 1000ng/dl. Maybe the batch the OP got was bad or it was a bad bottle (I'm sure not every bottle gets teh same concentration. Maybe the mixer stop and the liquid settled and then started pouring again. I'm sure it can happen).



Also, if you you say you ran PCT and didn't lose any gains then you are basically a lying cunt or your gear was bunk. And you have no clue that gear causes water and glycogen retention that is released when gear is no longer administered.



BRB, running cycle, gaining 30lbs and keeping it all a few weeks PCT.


I seem to remember 3 different people complaining in this thread about the Torem and all weren't from the same batch.

Water retention hung around that cycle as I was just starting Rips.. Not to mention it was my first cycle in 8 years and I had been close to that weight before, I guess you have no clue about muscle memory.. :rolleyes:

I have no idea who you are and could give a fuck less what your fuck ass thinks.. Good day.. :)
 
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I used CEM Torem last year and tested at 700ish a few weeks after PCT. After another cycle I did Torem + Nolva and tested at over 1000ng/dl. Maybe the batch the OP got was bad or it was a bad bottle (I'm sure not every bottle gets teh same concentration. Maybe the mixer stop and the liquid settled and then started pouring again. I'm sure it can happen).

Also, if you you say you ran PCT and didn't lose any gains then you are basically a lying cunt or your gear was bunk. And you have no clue that gear causes water and glycogen retention that is released when gear is no longer administered.

BRB, running cycle, gaining 30lbs and keeping it all a few weeks PCT.

CEM schill?
 
Actually Toremifene is better than Tamoxifene for that (receptor blocking) if you read the breast cancer studies. It does aid male fertility, but through a different mechanism of action than Clomid because Clomid is essentially synthetic LH.


I'm not sure it's right to label Clomid as synthetic LH. If that was the case, we wouldnt need HCG... we would just be using clomid throughout cycle and PCT. Too bad because that would be awesome!
 
I'm not sure it's right to label Clomid as synthetic LH. If that was the case, we wouldnt need HCG... we would just be using clomid throughout cycle and PCT. Too bad because that would be awesome!

Read the thread jballz posted earlier in this thread.
 
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