BouleauBoulot
New Member
I've read a bit about it, from what I understand it's nothing but HCG and a SERM/AI.
Why not just take HCG during the cure to prevent testicular atrophy due to lack of LH, then stop the cure + HCG and possibly take a real suicidal AI (not a serm like clomid) like exemestan in case the estrogens would be too high at the end of treatment and would slow down the return of LH ?
Clomid does absolutely nothing except artificially increase gnRH production by antagonizing estrogen receptors in the pituitary gland. The increase in LH and therefore testosterone therefore stops once you stop clomid, and it doesn't really matter anyway because the goal is simply for the pituitary to restart normal production of gnRH and the testicles respond well enough to regain normal testosterone levels.
The only answers I got were average low IQ bodybuilders who arrogantly contradicted me without any biochemical explanation. I just have the impression that everyone follows out of habit, without really knowing why, a delirium of stupid bodybuilders who don't understand biochemistry.
I spoke to an endocrinologist recently, if a patient has testicular atrophy after many years of trt or steroids, all he does is add something like 500ui of HCG eod, and the testicles gradually come back to normal normal functions, while on TRT. Obviously LH is still at 0 but the pituitary extremely quickly resumes normal production of gnRH in the event of cessation of testosterone signaling or downstream hormones.
Why not just take HCG during the cure to prevent testicular atrophy due to lack of LH, then stop the cure + HCG and possibly take a real suicidal AI (not a serm like clomid) like exemestan in case the estrogens would be too high at the end of treatment and would slow down the return of LH ?
Clomid does absolutely nothing except artificially increase gnRH production by antagonizing estrogen receptors in the pituitary gland. The increase in LH and therefore testosterone therefore stops once you stop clomid, and it doesn't really matter anyway because the goal is simply for the pituitary to restart normal production of gnRH and the testicles respond well enough to regain normal testosterone levels.
The only answers I got were average low IQ bodybuilders who arrogantly contradicted me without any biochemical explanation. I just have the impression that everyone follows out of habit, without really knowing why, a delirium of stupid bodybuilders who don't understand biochemistry.
I spoke to an endocrinologist recently, if a patient has testicular atrophy after many years of trt or steroids, all he does is add something like 500ui of HCG eod, and the testicles gradually come back to normal normal functions, while on TRT. Obviously LH is still at 0 but the pituitary extremely quickly resumes normal production of gnRH in the event of cessation of testosterone signaling or downstream hormones.