peculiarfind
New Member
Let's assume that someone has never done a cycle and their test levels are normal, say 800ng...
If that person were to take an AI, E2 would be lowered. When the person stopped taking the AI, E2 would increase. With that said, wouldn't it be impossible to experience E2 rebound because test levels are in normal range? Thinking that aromatization would return to normal TT:E2 levels.
On the other hand, if the same person were to take nolva and then stop, rebound would be possible because E2 was being synthesized, but not used, and so E2 has built up?
Is this accurate or am I missing something?
This could apply to someone (me) who is several months past PCT, but experiencing high E2 with normal TT. I was thinking of AI followed up by a SERM, but worried about rebound again. Then I thought just to take the AI to get E2 levels down and then come off of that...
If that person were to take an AI, E2 would be lowered. When the person stopped taking the AI, E2 would increase. With that said, wouldn't it be impossible to experience E2 rebound because test levels are in normal range? Thinking that aromatization would return to normal TT:E2 levels.
On the other hand, if the same person were to take nolva and then stop, rebound would be possible because E2 was being synthesized, but not used, and so E2 has built up?
Is this accurate or am I missing something?
This could apply to someone (me) who is several months past PCT, but experiencing high E2 with normal TT. I was thinking of AI followed up by a SERM, but worried about rebound again. Then I thought just to take the AI to get E2 levels down and then come off of that...