receptersites

chrisfam5

New Member
does any one know how long it takes to empty out your receptersites, i was told if i didnt stay off for so long that my next cycle would not work?
 
someone just shot you a line of shit....tell this person they are stupid and never to give you any advice again ever..
 
i suggest you go to ARTICLES on the upper left of this page there are some good articles on receptors...used to be one the myth of receptor downregulation...maybe still there i just didnt feellike lookin for it
 
Feeling a little angry today Chris??

What if he's talking about beta II receptors While using Clenbuterol?
 
chrisfam5 said:
does any one know how long it takes to empty out your receptersites, i was told if i didnt stay off for so long that my next cycle would not work?

What CG is saying is that all of the following is a myth:

1. anabolic receptors "burn-out".
2. anabolic receptors need to "empty-out".
3. anabolic receptors down-regulate and need time off to up-regulate.

All of these fallacies were dreamed up in the dark ages of steroid knowledge.

Although they've been scientifically disproven, as you can see from all the misinformation posted on this board; research, understanding and truth are often strangers to wannabe bodybuilders.

MaxRep
 
Phreez, my friend, good to see you! I hope all is well.

Your post is too funny because without seeing it, I specifically addressed anabolic receptors because some receptors, such as the beta-2 receptors you mention, do down-regulate and require time off.

I hope you're having a great weekend!
MaxRep
 
I'm doing well brother, and so is my weekend, a bit too quickly on the later, but still better than sitting in the office.

I too hope that you are having a great weekend! :D


oh and Chris, I was just f*ck'n with ya a little... I'm fairly certain that the original poster was referring to anabolics and not clen... I just couldn't resist ;)
 
MaxRep said:
Phreez, my friend, good to see you! I hope all is well.

Your post is too funny because without seeing it, I specifically addressed anabolic receptors because some receptors, such as the beta-2 receptors you mention, do down-regulate and require time off.

I hope you're having a great weekend!
MaxRep
thanks guys for the input im going to go take a shot right now!!!!!!!!! :D
 
chrisfam5 said:
thanks guys for the input im going to go take a shot right now!!!!!!!!! :D

I didn't mean what I said as an endorsement to not have a sufficient period of "off" time, for other reasons. If you haven't taken off for roughly as long as you were last on, including PCT, then you're using the juice as a crutch and potentially causing yourself problems down the road with your HPTA.

MaxRep
 
Receptor site "emptying" isn't why you take a break between cycles. You want to recover proper HPTA function. Receptors are actually upregulated in response to supraphysiological levels of AAS. Over time (over the course of a longer cycle), they may become downregulated, but I don't think anyone's really shown that. gains will slow down, but that is more likely a response to things like upregulation of cortisol receptors and other indirect effects of AAS use.
 
Phreezer said:
Feeling a little angry today Chris??

What if he's talking about beta II receptors While using Clenbuterol?

you know..i assumed he meant androgen receptors...however in the case he was referring to the beta 2s he could use ketotifen to prevent downregulation...oh are there any beta 2 antagonists
 
wait i fucked up in my last post....what i meant to say was to the hip , hop the hippy to the hippy the hip hip hoppin you dont stop rockin to the bang bang boogy said up jump the boogy to the rythym of the boogity beat
 
Trekkie

chris gordon said:
you know..i assumed he meant androgen receptors...however in the case he was referring to the beta 2s he could use ketotifen to prevent downregulation...oh are there any beta 2 antagonists
HEY CHRIS G,HOW ABOUT CLAMBUTEROL,IS THAT A BETA 2,..OR A DUMB 4.LMK :D


finafreak
 
chris gordon said:
clambutteroil hmmmi think its um...i dont know, let me get help from ultraman
Clambutteroil ??? :D Chris you and the girlfriend getting kinky again??? Peace......Vegas
 
chris gordon said:
you know..i assumed he meant androgen receptors...however in the case he was referring to the beta 2s he could use ketotifen to prevent downregulation...oh are there any beta 2 antagonists


Actually all beta antagonists (such as toprol, lowpresser.etc..) will block beta two recepters.. At small dosages (like less than 25mg).. some beta antaganists can be selective and only block beta 1 recepters in the heart...but once you get over that very small threshold..they all become non-selective beta ataganists...and can lead to bronchial constriction in some people (especially if you have any underlying asthmatic symptoms)

Now drugs, like Albuterol, clenbuterol..etc (beta agonists) are designed to stimulate the beta 2 receptors in your lungs.. (that's how the help fight asthma) however, they will also stimulate the beta 1 recepters in your heart.. That is why beta agonists raise your rhr.. Why it's necessary to be careful while using clen (and clen's long half life and it's effect on heart rate are part of the reason it was actually banned by the FDA)

I've never heard of any diffinitive evidence that ketotifen would keep the beta 2 receptors in the lungs from ever down regulating.... Could you post that study for me?
 
0.02 cents

Phreezer said:
Actually all beta antagonists (such as toprol, lowpresser.etc..) will block beta two recepters.. At small dosages (like less than 25mg).. some beta antaganists can be selective and only block beta 1 recepters in the heart...but once you get over that very small threshold..they all become non-selective beta ataganists...and can lead to bronchial constriction in some people (especially if you have any underlying asthmatic symptoms)

Now drugs, like Albuterol, clenbuterol..etc (beta agonists) are designed to stimulate the beta 2 receptors in your lungs.. (that's how the help fight asthma) however, they will also stimulate the beta 1 recepters in your heart.. That is why beta agonists raise your rhr.. Why it's necessary to be careful while using clen (and clen's long half life and it's effect on heart rate are part of the reason it was actually banned by the FDA)

I've never heard of any diffinitive evidence that ketotifen would keep the beta 2 receptors in the lungs from ever down regulating.... Could you post that study for me?
Damm Phreezer your one smart mofo,.Good info post. I know when you have an allergic rash,& knowing your beta receptors are a big key into solving the problem.


finafreak
 
Last edited:
Phreezer said:
Actually all beta antagonists (such as toprol, lowpresser.etc..) will block beta two recepters.. At small dosages (like less than 25mg).. some beta antaganists can be selective and only block beta 1 recepters in the heart...but once you get over that very small threshold..they all become non-selective beta ataganists...and can lead to bronchial constriction in some people (especially if you have any underlying asthmatic symptoms)

Now drugs, like Albuterol, clenbuterol..etc (beta agonists) are designed to stimulate the beta 2 receptors in your lungs.. (that's how the help fight asthma) however, they will also stimulate the beta 1 recepters in your heart.. That is why beta agonists raise your rhr.. Why it's necessary to be careful while using clen (and clen's long half life and it's effect on heart rate are part of the reason it was actually banned by the FDA)

I've never heard of any diffinitive evidence that ketotifen would keep the beta 2 receptors in the lungs from ever down regulating.... Could you post that study for me?

Sorry it took me so long to get back to you.. I don't watch the anabolic forum that closely anymore...
 
Yes and no... It's actually the histamines that your body produces during the allergic reaction that cause the rash.. and sometimes bronchial constriction.. so by stimulating beta 2 recepters that can help open up bronchial tubes..but the prefered method of treatment in a case like that would have to be benedryl, cortisone (or other antihestimines) or a prefered bronchial dialater like epinephrine etc..
 
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