M1T is indeed c17 methylated (oral) DHB
It was a popular OTC ‘prohormone’ back in the day. It worked but of course was significantly liver toxic, and became illegal.
It’s been said that DHB is liver toxic which is not expected from its chemical structure - it is in fact primobolan without the c1 methyl group which attenuates its potency and was added for partial oral bioavailability as primobolan acetate. Removing that methyl group should make it less toxic, not more.
I’ve wondered if old M1T raws were reprocessed and brewed to be sold as injectable ‘DHB’ - which would explain the liver toxicity.
This exact situation happened not too long ago with a source that used methyl-stenbolone raws to brew ‘stenbolone acetate’ - and it was sold as such before someone sent it off for testing and word got out. I have got four vials of it. and occasionally use it as an injectable preworkout. And it’s a good one!
Stenbolone is c2-methylated DHB, M-sten adds C17-methylation. So basically dimethyl-DHB.
Despite never being produced by pharma, DHB is actually the parent compound of several AAS including M1T, stenbolone, M-Sten, and primobolan. And DHB is of course 5α-reduced boldenone, but it’s not clear if boldenone is a candidate for human 5α-reductase. If it is, then it again brings into question why exactly DHB is liver toxic, because even high doses of boldenone are not…
There should be another limb of the steroid family tree, consisting of Δ1-testosterone and its various derivatives. Primobolan belongs in that group, unlike masteron.