Starting first cycle with pubertal gyno. Start ai from first pin?

[CensoredBoardsSuck]

If i wasn't busy being productive elsewhere id happily dig up that thread on Steroid.com where you were schooled over and over again until Brian banned you forever.

Ask and ye shall receive. I always laugh at the part where Sworder can't figure out how he got semen in his rectum. LMFAO:


http://forums.steroid.com/hormone-replacement-therapy-low-testosterone-treatment-anti-aging/519006-trenbolone-hrt-better-than-testosterone.html (http://forums.steroid.com/hormone-r...-trenbolone-hrt-better-than-testosterone.html)

Examples of Sworder's extensive knowledge of AAS:

Originally Posted by Sworder
Why can AAS cause your penis to stop working like it should? Hard-ons aren't as hard as they used to be. No more pointing to the sky and sometimes it goes limp during intercourse. viagra and cialis increase the bloodflow to the penis but this is not repairing what caused the ED in the first place. It is simply bypassing the problem. I am not talking about possible low testosterone (hormonal imbalance) being the problem, it is occurring through a different pathway.

Any ideas are welcome!


This was one of his posts on 06-18-2012, 08:02 PM

Originally Posted by Sworder
Ok, first off sorry to soil your mind with the imagery I am projecting.


So, past 3 days after I have been taking a shit I have noticed some what I thought looked like mucus in the toilet. Didn't think about it that much, just thought I had spit in the toilet or something earlier. Found it again 2 days ago, didn't really pay attention. Then yesterday as I am taking a shit, I notice ejaculatory fluids coming out thick as I am squeezing out the turds.

Is this because my prostate is enlarged right now or what is my excrement pushing against to make the fluid discharge(about 5ml, thick yellowish cloudy) out of my phallus ?


p.s I am on a lot of gear right now..

Originally Posted by Sworder

Do obese men require a higher TRT dose to be effective?

Why?


10/7/12

Originally Posted by Sworder
I have so called "puffy nipples" from puberty and I have never really noticed that they were puffy. I just thought it was normal. I have yet to do a cycle and my question is as follows: does me having puffy nipples mean I am more prone to gyno than others? If I do get gyno would it just keep building on to that small glad that is already there from my pubescence?


Would be great to hear from somebody that has the same "problem"..


on the 5/29/11

Originally Posted by Sworder
Hey guys, both me and my brother are on cycles and we both noticed our left nipples moving down and to the left(towards the armpit). I have had itchy/sore areolas but my brother has had no problems. Is the relocation of the nipple just a result of more muscle mass? Is this common?

 

[Sworder]

Apart from all the attacks going on, 0.25 eod second day from first pin till mid cycle bloods with adjustments if the gyno starts swelling before the mid cycle bloods, otherwise adjust after mid cycle bloods. Sounds good?

Yes, that's a minor dose. Don't touch your nipples all the time and if you do get a flare-up, DON'T WORRY. It's easily reversed, just up your AI dose a little bit. I would personally do 1 mg that day and then keep going 0.5 eod after a flare-up. Just don't freak out; tits don't grow overnight. Also, if you notice joint pain, that your "BF" is losing drastically, or depression cut the AI out as that is a result of your e2 crashing.

 

[Big_paul]

No, he is probably referring to a small glandular node behind the areola. The % of people having pre-pubertal/pubertal gyno is very high. Most people don't even notice it.

"Tits" don't grow overnight. Even if OP gets a flare up it's just a swelling of the tissue, it goes down really fast and there is no reason to worry.
Don't get butthurt BP.

I would pay the devil to put you in front of me.

 

[Sworder]

I would pay the devil to put you in front of me.

You wish.
We can exchange info if you want! PM me.

 

[cdoubleu]

Doesn't matter, you do not need an AI in the start...
I have gyno too from puberty, do not listen to everybody that is saying use THIS and THAT amount. They have no idea about your e2 levels so DO NOT LISTEN to them, it will make you tank your e2 because you are paranoid and these guys are telling you to use an AI from the get go.. Get labs if needed to see your e2.
E2 is great for many reasons, GH production, ligament/tendons, and many other factors. Don't crash your e2. Use AI as needed, get labs if needed but there is no standard dose you should start with..
Also, with PCT it may improve, nolva during the cycle isn't beneficial..

Take this for what it is...some good and bad advice and decide for yourself, which it looks like you have. The blood test is very good advice, but may be hard to accomplish early in the cycle depending on your circumstances. If you start your cycle and wait a while to get some bloods like he is suggesting a large amount of aromatization could have occurred already.

Do not take for granted how quickly this process actually begins. You may think that since it takes 5ish weeks to reach optimal testosterone levels then your estrogen wont get all that elevated either.

That being said what is being said about crashing e2 is very real as well so use caution. If i were to do it over again i would have started a serm a few days or so before my first pin to allow levels to build up a bit. Then use adex accordingly.

 

[Dwizzle]

Its ok to start at .25mg EOD especially if you know you are prone to gyno already. If your E2 crashes it won't be so bad at .25mg EOD you feel like you have arthritis in your joints.

Listen to your body, or until you can get some mid 4wk or mid cycle bloods done. Be smart and safe; adjust doses accordingly! Good luck brother!

 

[Northern Nutrition]

Apart from all the attacks going on, 0.25 eod second day from first pin till mid cycle bloods with adjustments if the gyno starts swelling before the mid cycle bloods, otherwise adjust after mid cycle bloods. Sounds good?

Sounds perfect. Keep us posted, Ludwig.

 

[Northern Nutrition]

Ask and ye shall receive. I always laugh at the part where Sworder can't figure out how he got semen in his rectum. LMFAO:


http://forums.steroid.com/hormone-replacement-therapy-low-testosterone-treatment-anti-aging/519006-trenbolone-hrt-better-than-testosterone.html (http://forums.steroid.com/hormone-r...-trenbolone-hrt-better-than-testosterone.html)

Examples of Sworder's extensive knowledge of AAS:

Originally Posted by Sworder
Why can AAS cause your penis to stop working like it should? Hard-ons aren't as hard as they used to be. No more pointing to the sky and sometimes it goes limp during intercourse. viagra and cialis increase the bloodflow to the penis but this is not repairing what caused the ED in the first place. It is simply bypassing the problem. I am not talking about possible low testosterone (hormonal imbalance) being the problem, it is occurring through a different pathway.

Any ideas are welcome!


This was one of his posts on 06-18-2012, 08:02 PM

Originally Posted by Sworder
Ok, first off sorry to soil your mind with the imagery I am projecting.


So, past 3 days after I have been taking a shit I have noticed some what I thought looked like mucus in the toilet. Didn't think about it that much, just thought I had spit in the toilet or something earlier. Found it again 2 days ago, didn't really pay attention. Then yesterday as I am taking a shit, I notice ejaculatory fluids coming out thick as I am squeezing out the turds.

Is this because my prostate is enlarged right now or what is my excrement pushing against to make the fluid discharge(about 5ml, thick yellowish cloudy) out of my phallus ?


p.s I am on a lot of gear right now..

Originally Posted by Sworder

Do obese men require a higher TRT dose to be effective?

Why?


10/7/12

Originally Posted by Sworder
I have so called "puffy nipples" from puberty and I have never really noticed that they were puffy. I just thought it was normal. I have yet to do a cycle and my question is as follows: does me having puffy nipples mean I am more prone to gyno than others? If I do get gyno would it just keep building on to that small glad that is already there from my pubescence?


Would be great to hear from somebody that has the same "problem"..


on the 5/29/11

Originally Posted by Sworder
Hey guys, both me and my brother are on cycles and we both noticed our left nipples moving down and to the left(towards the armpit). I have had itchy/sore areolas but my brother has had no problems. Is the relocation of the nipple just a result of more muscle mass? Is this common?

LMFAO!!

 

[Sworder]

Ask and ye shall receive. I always laugh at the part where Sworder can't figure out how he got semen in his rectum. LMFAO

BPH, put your reading glasses on there was no semen in the rectum. Don't let your imagination float off.

 

[CensoredBoardsSuck]

BPH, put your reading glasses on there was no semen in the rectum.

"Technically," you're right. It wasn't still in their when you made the post. It had been pushed out during your bowel movement and that's when you noticed it in the toilet.

 

[Sworder]

No, "technically" it wasn't still in their when you made the post. It had been pushed out during your bowel movement and that's when you noticed it in the toilet.

BPH

 

[CensoredBoardsSuck]

BPH

Backdoor Penis Hustler. Duh!

What's your point?

 

[Dwizzle]

Originally Posted by Sworder
Ok, first off sorry to soil your mind with the imagery I am projecting.


So, past 3 days after I have been taking a shit I have noticed some what I thought looked like mucus in the toilet. Didn't think about it that much, just thought I had spit in the toilet or something earlier. Found it again 2 days ago, didn't really pay attention. Then yesterday as I am taking a shit, I notice ejaculatory fluids coming out thick as I am squeezing out the turds.

Is this because my prostate is enlarged right now or what is my excrement pushing against to make the fluid discharge(about 5ml, thick yellowish cloudy) out of my phallus ?


p.s I am on a lot of gear right now..

Originally Posted by Sworder


I am not even trying to start shit right now but I just read this, LMFAAAOOOOOOOOOOOOOOOOOOOOOOOOO

Omg this shit is so funny ejaculatory fluids???? bahahahahaha Why the fuck is it so descriptive Its like a graphic fucking novel

 

[CensoredBoardsSuck]

Omg this shit is so funny ejaculatory fluids???? bahahahahaha Why the fuck is it so descriptive Its like a graphic fucking novel

It's because Sworder gets to relive the magical moment the ejaculatory fluid was deposited in his rectum all over again. Sworder probably had to type that post one-handed.

 

[Sworder]

It's because Sworder gets to relive the magical moment the ejaculatory fluid was deposited in his rectum all over again. Sworder probably had to type that post one-handed.

Benign prostatic hyperplasia was the cause. Didn't think I had to spell it out for you.

 

[Big_paul]

No, he is probably referring to a small glandular node behind the areola. The % of people having pre-pubertal/pubertal gyno is very high. Most people don't even notice it.

"Tits" don't grow overnight. Even if OP gets a flare up it's just a swelling of the tissue, it goes down really fast and there is no reason to worry.
Don't get butthurt BP.

I'm not butt hurt sworder, but how do you think guys get gyno. Do they run a cycle and watch tits grow? If op uses an ai and or a serm he's covered.

 

[Sworder]

I'm not butt hurt sworder, but how do you think guys get gyno. Do they run a cycle and watch tits grow? If op uses an ai and or a serm he's covered.

Yup, he is covered!
IDK how people get gyno that are on the boards, typically people on the boards are pretty good at posting. Maybe they neglect or are too overweight to notice gyno symptoms?

There are a couple guys that have gyno surgery ask them instead. I have had tons of flare ups but it always gets taken care of.

OP has no larger chance than anybody else to develop bitch tits, I wouldn't recommend starting an AI with a long ester from the get go. And too much paranoia from the board will make him take too much AI. The SERM is better however that inhibits GH production as well as some other things. Where do you draw the line of being cautious vs scared so it hurts your gains?

Tamoxifen attenuates pulsatile growth hormone secretion: mediation in part by somatostatin
http://press.endocrine.org/doi/abs/10.1210/endo.130.6.1350760
Abstract
Tamoxifen, a partial competitive antagonist to the estrogen receptor, is a potent inhibitor of the proliferation of experimental mammary carcinoma in the rat and is widely used clinically in the treatment of breast cancer. Blockade of estrogen receptors present on neoplastic cells represents the classic mechanism of action of tamoxifen, but the drug has a variety of other actions that may contribute to its antiproliferative properties. While it is recognized that estrogens play an important role in modulating pulsatile GH release, the effect of antagonists to sex steroid receptors on GH secretory dynamics has not previously been described. In the present study we examined the effect of tamoxifen on pulsatile GH secretion in free-moving adult male and female rats. The drug, when administered in a manner previously shown to be associated with antineoplastic activity, caused a marked suppression of the amplitude of spontaneous GH secretory bursts and significantly reduced mean 6-h plasma GH levels in both sexes compared to those in their respective peanut oil-injected controls. Inhibition of spontaneous GH pulses persisted for up to 7 weeks after tamoxifen administration in both sexes. Immunoneutralization of endogenous somatostatin in tamoxifen-treated male rats completely restored both GH pulse amplitude (121.6 +/- 9.5 vs. 62.5 +/- 13.5 ng/ml in tamoxifen-treated rats given normal sheep serum; P less than 0.02) and mean 6-h plasma GH levels (53.3 +/- 6.6 vs. 17.9 +/- 3.6 ng/ml in normal sheep serum-treated rats; P less than 0.01) to levels observed in our peanut oil-injected controls. These results demonstrate that 1) tamoxifen has potent inhibitory effects on pulsatile GH secretion; and 2) the blunting of GH pulse amplitude by tamoxifen is mediated at least in part by increased release of endogenous somatostatin. These findings motivate further investigation of the clinical significance of tamoxifen-induced suppression of GH secretion in relation to the antineoplastic activity of this commonly used drug.

 

[CensoredBoardsSuck]

Yup, he is covered!
IDK how people get gyno that are on the boards, typically people on the boards are pretty good at posting. Maybe they neglect or are too overweight to notice gyno symptoms?

There are a couple guys that have gyno surgery ask them instead. I have had tons of flare ups but it always gets taken care of.

OP has no larger chance than anybody else to develop bitch tits, I wouldn't recommend starting an AI with a long ester from the get go. And too much paranoia from the board will make him take too much AI. The SERM is better however that inhibits GH production as well as some other things. Where do you draw the line of being cautious vs scared so it hurts your gains?

Tamoxifen attenuates pulsatile growth hormone secretion: mediation in part by somatostatin
http://press.endocrine.org/doi/abs/10.1210/endo.130.6.1350760
Abstract
Tamoxifen, a partial competitive antagonist to the estrogen receptor, is a potent inhibitor of the proliferation of experimental mammary carcinoma in the rat and is widely used clinically in the treatment of breast cancer. Blockade of estrogen receptors present on neoplastic cells represents the classic mechanism of action of tamoxifen, but the drug has a variety of other actions that may contribute to its antiproliferative properties. While it is recognized that estrogens play an important role in modulating pulsatile GH release, the effect of antagonists to sex steroid receptors on GH secretory dynamics has not previously been described. In the present study we examined the effect of tamoxifen on pulsatile GH secretion in free-moving adult male and female rats. The drug, when administered in a manner previously shown to be associated with antineoplastic activity, caused a marked suppression of the amplitude of spontaneous GH secretory bursts and significantly reduced mean 6-h plasma GH levels in both sexes compared to those in their respective peanut oil-injected controls. Inhibition of spontaneous GH pulses persisted for up to 7 weeks after tamoxifen administration in both sexes. Immunoneutralization of endogenous somatostatin in tamoxifen-treated male rats completely restored both GH pulse amplitude (121.6 +/- 9.5 vs. 62.5 +/- 13.5 ng/ml in tamoxifen-treated rats given normal sheep serum; P less than 0.02) and mean 6-h plasma GH levels (53.3 +/- 6.6 vs. 17.9 +/- 3.6 ng/ml in normal sheep serum-treated rats; P less than 0.01) to levels observed in our peanut oil-injected controls. These results demonstrate that 1) tamoxifen has potent inhibitory effects on pulsatile GH secretion; and 2) the blunting of GH pulse amplitude by tamoxifen is mediated at least in part by increased release of endogenous somatostatin. These findings motivate further investigation of the clinical significance of tamoxifen-induced suppression of GH secretion in relation to the antineoplastic activity of this commonly used drug.

That's rat data, Sworder. LMAO

In case you don't know, rats are not human. More worthless bullshit! Try again.

 

[Sworder]

That's rat data, Sworder. LMAO

In case you don't know, rats are not human. More worthless bullshit! Try again.

http://press.endocrine.org/doi/abs/10.1210/jcem.79.2.8045971

Boys! Google extrapolate or find your own studies

 

[CensoredBoardsSuck]

http://press.endocrine.org/doi/abs/10.1210/jcem.79.2.8045971

Boys! Google extrapolate or find your own studies

No, you made the claim, you back it up. You don't get to pawn the responsibility for supporting your claims off on others.

You've got the right species now, but it's a SINGLE study that was conducted over FOUR days and only had 10 subjects. No conclusions can be drawn from that weak evidence. Try again.