Trying not to do the nube shuffle.

[The ole man]

Well 1st injection had pip for 4 days.
2nd injection pip for 1 day and it was barely anything.
3rd injection pip is about to kill me..

So its fairly easy to assume that PIP is bad technique....
My 2nd time was much slower and I let it set once all was injected for 10 secs, before pulling it out..
I felt an increase in strength in the gym today...
and a few extra reps too...

All and all so far doing pretty good.
need to get better and meal prep and count my micros.. Im depending on 3 protein shakes to put me over.. I need to do better at getting my macros from food.

 

[HIGHRISK]

Well 1st injection had pip for 4 days.
2nd injection pip for 1 day and it was barely anything.
3rd injection pip is about to kill me..

So its fairly easy to assume that PIP is bad technique....
My 2nd time was much slower and I let it set once all was injected for 10 secs, before pulling it out..
I felt an increase in strength in the gym today...
and a few extra reps too...

All and all so far doing pretty good.
need to get better and meal prep and count my micros.. Im depending on 3 protein shakes to put me over.. I need to do better at getting my macros from food.

It could very well be technique. I get pip sometimes and sometimes I don't. I rotate from each Glute to each delt to each quad. Once u rotate spots and pin your virgin muscles it will get better and easier. Delts are my favorite and easiest for me.

 

[The ole man]

It could very well be technique. I get pip sometimes and sometimes I don't. I rotate from each Glute to each delt to each quad. Once u rotate spots and pin your virgin muscles it will get better and easier. Delts are my favorite and easiest for me.

So I did yesterday morning's pin in right quad. It went real smooth. I didnt rush it, giving several seconds from start to finish. I also made sure not to let my muscle move at all thru out.. Almost seems like a wiggle of the toe will cause some muscle fibers to move.. Anyway, I don't even feel it today. Best pin yet.. I think I got it now, at least in the right quad, which is the easiest spot for me. Delts seem a lil cray cray with 1 ml of t300 E for me.. I figured that was prob a better spot for Test P..
Im up 7 pounds with slightly better bfi..
Im almost to my 4th week of epistane, and starting 3rd week of T 300mg/ml E

 

[The ole man]

So I noticed today, that my nipples were hard and very sensitive, and my belly had a full feel to it. Being new, Im still getting to know the signs of high estro. I decided to take 1/4 of a arimidex.
Im 17 days into my cycle at 600 mg Test E a week.
Along with 40 mg of epistane
and 12.5 mg of osta

How long does the first dose take to see effects?
Is there a way to know how often I should take it?

I figure 13 more days and then get bloods taken?

 

[JackSmooth]

So I noticed today, that my nipples were hard and very sensitive, and my belly had a full feel to it. Being new, Im still getting to know the signs of high estro. I decided to take 1/4 of a arimidex.
Im 17 days into my cycle at 600 mg Test E a week.
Along with 40 mg of epistane
and 12.5 mg of osta

How long does the first dose take to see effects?
Is there a way to know how often I should take it?

I figure 13 more days and then get bloods taken?

Stay consistent with the ai so something like .25mg eod and as long as youre not showing high estro sides keep it at that and wait till about the 6th week to get bloods done and adjust the ai dose accordingly.

 

[The ole man]

Stay consistent with the ai so something like .25mg eod and as long as youre not showing high estro sides keep it at that and wait till about the 6th week to get bloods done and adjust the ai dose accordingly.

Appreciate it bro!

 

[The ole man]

So last 2 pins have been no pip at all... I guess you could say my virgin muscles are reg hoes now. So im up about 10 pounds and my strength has increased. I also moved up to 1/2 mg eod of dex and I still seem to get itchy nips, and sensitive/hard nips. I continue to monitor. Down to my last 2 days of epistane. Test and Mk2866 the rest of the way...

 

[The ole man]

Started 6th week today, no pip anymore.. Blood tests at end of week...
So far been smooth, pumps in the gym are off the chain...
need to eat more!!! im up 10 pounds, but my abs are slightly more visible... Its so easy to eat a bunch of food that isnt good for you, but freaking expensive, and more difficult to eat proper...
Dealing with sciatica in the middle of my cycle sucks but trying to power thru it.. stretching is freaking harder than working out..

So im thinking Jan, my next cycle is gonna be test and tren. Im gonna go test P at 400 mg/week and tren A at 300 mg/week..
Run that 8 weeks...
From what i read, tren is a tricky aas, as some ppl handle it and some ppl dont, and with the tren A if I experience sides that are too much I can discontinue the tren and it will be out of my system in days, compared to a long ester tren which might take me weeks to overcome....
The more you learn about this, the more intriguing it becomes...
Any other advice about trn or tren/test stack, im up for recommendations and comments..

 

[The ole man]

I got a question for you guys.
So ordered
1 vial test e 300
2 vials of tren e 200
2 vials of mast e 200
And cabaser .

So customs snagged there first attempt at delivery...
They said they would redeliver asap..
After a week i mess them and they said sorry we havent sent it and they had to wait on some inventory..
Since my address was burnt up on the first try, and i had to provide them with another, the only available thing i had was my very old parents house..
So my anxiety was very unsettling up until i received the order..
So i got the order and 1 vial of mast is missing, 1 vial of tren is missing.
As well as the cabuser .
Im bugged out on even letting them try to reship the rest..
So how would you use this combination?
10 ml of test e 300 mg
10 ml of tren e 200 mg
10 ml of mast e 200 mg
Before i only received half an order my plans were
Test 150 mg a week
Tren 350 mg a week
Mast 400 mg a week
But now that i only have half order..
And im not ordering out of country for awhile.
So how can i utilize what i have...
My thought were
Plan 1
Run test e at 150 mg a week
400 mg ten e from week 1thru 5
400 mg mast e. Week 6 week 10
Or
300 mg test e
200mg tren e
200mg mast
10 weeks

 

[FourOneDeuxFitt]

I got a question for you guys.
So ordered
1 vial test e 300
2 vials of tren e 200
2 vials of mast e 200
And cabaser .

So customs snagged there first attempt at delivery...
They said they would redeliver asap..
After a week i mess them and they said sorry we havent sent it and they had to wait on some inventory..
Since my address was burnt up on the first try, and i had to provide them with another, the only available thing i had was my very old parents house..
So my anxiety was very unsettling up until i received the order..
So i got the order and 1 vial of mast is missing, 1 vial of tren is missing.
As well as the cabuser .
Im bugged out on even letting them try to reship the rest..
So how would you use this combination?
10 ml of test e 300 mg
10 ml of tren e 200 mg
10 ml of mast e 200 mg
Before i only received half an order my plans were
Test 150 mg a week
Tren 350 mg a week
Mast 400 mg a week
But now that i only have half order..
And im not ordering out of country for awhile.
So how can i utilize what i have...
My thought were
Plan 1
Run test e at 150 mg a week
400 mg ten e from week 1thru 5
400 mg mast e. Week 6 week 10
Or
300 mg test e
200mg tren e
200mg mast
10 weeks

Well if you don’t feel like waiting for more gear I would go with your last option of 300/200/200 for 10 weeks. That’s just my .02 but in all honesty I would wait to get what you need for your original plan and not jump the gun.

 

[The ole man]

I got a question for you guys.
So ordered
1 vial test e 300
2 vials of tren e 200
2 vials of mast e 200
And cabaser .

So customs snagged there first attempt at delivery...
They said they would redeliver asap..
After a week i mess them and they said sorry we havent sent it and they had to wait on some inventory..
Since my address was burnt up on the first try, and i had to provide them with another, the only available thing i had was my very old parents house..
So my anxiety was very unsettling up until i received the order..
So i got the order and 1 vial of mast is missing, 1 vial of tren is missing.
As well as the cabuser .
Im bugged out on even letting them try to reship the rest..
So how would you use this combination?
10 ml of test e 300 mg
10 ml of tren e 200 mg
10 ml of mast e 200 mg
Before i only received half an order my plans were
Test 150 mg a week
Tren 350 mg a week
Mast 400 mg a week
But now that i only have half order..
And im not ordering out of country for awhile.
So how can i utilize what i have...
My thought were
Plan 1
Run test e at 150 mg a week
400 mg ten e from week 1thru 5
400 mg mast e. Week 6 week 10
Or
300 mg test e
200mg tren e
200mg mast
10 weeks

I meant for second

Well if you don’t feel like waiting for more gear I would go with your last option of 300/200/200 for 10 weeks. That’s just my .02 but in all honesty I would wait to get what you need for your original plan and not jump the gun.

I know, but after 2 months of anxiety im just not into it..
Maybe i just hold it like you said, and try to grab the rest at the end of the year..
Esp since i dont got the cabuser...
Appreciate the reply!
Thanks brother...

 

[The ole man]

Well if you don’t feel like waiting for more gear I would go with your last option of 300/200/200 for 10 weeks. That’s just my .02 but in all honesty I would wait to get what you need for your original plan and not jump the gun.

So it looks like I’m gonna end up with both bottles of tren. But only 1 of the bottles of mast.
I also ended up with some dbol.

So I’m gonna utilize what I have.
I’ve never did dbol but I have ran epistane 2x and the last time was 5 weeks at 20/30/40/50/50
Night sweats got pretty serious the last week.
Got 1 vial of test e 300
2 vials of tren e 200
1 vial of mast e 200
100 dbol tabs

Got prammi, adex, Clomid, tamox already in the cabinet.
I got some hcg that I never used, but I read that it loses its viability pretty quick, but I still got it.

I have been off cycle since last July.
I’ve dropped my bfi from 24 to 13ish
While trying to maintain as much muscle as possible.

My diet has improved.
I eat mainly fish, chicken breasts, tuna, turkey, and beef as my meats.
I kinda follow the Keto diet with a couple modifications.
I do UD 2.0 for 4 weeks on and 4 weeks off.
I also prob fall out of ketosis since I have a dinner salad which is homemade of all the things I hate cooked.
The ingredients are Romaine, cabbage, spinach leaf, mushrooms, broccoli, shredded carrots, shredded radishes, tomatoes, celery, cucumbers, green peppers, and onions.
Thinking about trying all the other leafy greens in my next salad I make. I make enough for the whole week!
Sunday is cooking day lol.
Cook all the salmon, chicken breasts, green beans with oinios and pink himilayan sea salt.
I use a dressing made with mayo(made with olive oil)some no fat sour cream.
I bought a few different dressings tonight.
1 is a plain oil and vinegar by Newman’s Own. Supposed to be Keto friendly.

I also buy beef ribs, trim the meat from the fat and the bone.
I cook up the beef in chuncks, and then scramble eggs up in the grease and mix them. Steak and eggs baby!
But the real reason is I make a bone broth out the beef bones. Simmer for 48 hrs. Oh it’s good!!! I have 8 ounces every day!

Anyway, as you see, I did that cycle, then I spent the rest of this time, working on my diet. I haven’t had soda or takeout but 1 time in 14 months.

I ate steak and shake 1 time, and it rather moved thru me quite quick!!
I didn’t do that again.


I know on cycle I should up my intake, and I usually don’t eat till 2 pm, starting at 7, as I have coffee and mct oil up till then.
On cycle I prob should add a good breakfast in, eggs, maybe some avocados, and maybe I start using the 5 jugs of protein I bough on sale. They been in the cabinet since I realized that salmon and tuna put protein powder to shame. As well as cause digestive issues.


I have no digestive issues, and this is the first time I can ever remember being without digestive issues.

I’m really happy mentally and emotionally.
When you shit good, you live good!

Anyway what’s your thoughts on how I should run my cycle now?
Damn that was long reply lol.

 

[The ole man]

So saw this while researching.
Says for experienced users.
Which having only 1 -12 week cycle of test, and 2 pro hormone cycles under my belt, I realize that isn’t me.

WEEKS
1-4 DANABOL 40MG/DAY ( TAKE 20 MG TWICE A DAY)
1-12 TESTOSTERONE ENANTHATE 500MG/WEEK ( IN 2 SHOTS PER WEEK )
1-12 PRIMOBOLAN 400MG/WEEK ( 2 SHOTS PER WEEK)
7-12 WINSTROL 50MG EVERY DAY


14-17 WEEKS , PCT AS FOLLOW:

DAY 1 – CLOMID 200MG + NOLVADEX 40MG
FOLLOWING 10 DAYS – CLOMID 50MG + TAMOXIFEN 20MG
FOLLOWING 10 DAYS – CLOMID 50MG OR TAMOXIFEN 20MG.


That seems just a lil too much for me.
So how does this look?

WEEKS
1-4 DANABOL 25mg/DAY
1-12 TESTOSTERONE ENANTHATE 200 mg/WEEK ( IN 2 SHOTS PER WEEK )
1-12 PRIMOBOLAN 300mg/WEEK ( 2 SHOTS PER WEEK)
7-12 Mast at 450mg/week (2 shots per week)
1-12 ARIMIDEX .5 eod
I have pranni for the tren, to help with prolactin but unsure of dose, esp adding it beside arimidex.

I also have hcg, but I’ve had it over a year unused??????


14-17 WEEKS , PCT AS FOLLOW:

DAY 1 – CLOMID 200MG + NOLVADEX 40MG
FOLLOWING 10 DAYS – CLOMID 50MG + TAMOXIFEN 20MG
FOLLOWING 10 DAYS – CLOMID 50MG OR TAMOXIFEN 20MG



I could use a little feed back if anyone has some.
Thanks. Been a great journey so far.

Had bloods taken last year.
Everything checked out good except my t3 was down and I think it had to do with my use of clen a couple weeks before.
Being tren I’m figuring bloods are even more important.
How and what blood test should I run before and after?


Thanks before hand for any and all advice. The advice I’ve received here has been detrimental to my progression.

 

[Btcowboy]

Well my two cents, with 1 14 week test only cycle under my belt. 500mg per week in 2 250mg pins per week.

I dont think that's too bad on the injectables side of things. I cant offer opinion on he orals. I know there will be others with alot more advice than I can give.

 

[The ole man]

Well my two cents, with 1 14 week test only cycle under my belt. 500mg per week in 2 250mg pins per week.

I dont think that's too bad on the injectables side of things. I cant offer opinion on he orals. I know there will be others with alot more advice than I can give.

Much appreciated.
I’m kinda wanting some feed back on running the arimidex along side the prammi.
I’m going with 200mg/wk test as I’ve read the tren sides multiple the higher the test.
How ever the dbol will require some test to overcome the shutdown of nat test.
I was gonna run the tren at 350/wk but I decided to back off just a little as I’ve never run tren.
And it’s a long ester so I have to deal with the sides for a period of time if I don’t handle it well, and stop it mid cycle

I ran 600 mg/wk test for 10 weeks and I loved it. Ran a oral, epistane for the first 5. Some night sweats, nothing I couldn’t handle, a lil extra laundry.

 

[The ole man]

Then I read articles that say prolactin concern are bro science and problems that come from unpure tren usually from a ugl.

This is a decent article on it.

This is what it all boils down to concerning Prolactin and how it relates to the anabolic steroid using community. The concern for a very long time (and still continues today) is that various anabolic steroids may or may not promote Prolactinergic effects on the body during use, either through stimulating Prolactin secretion from the pituitary gland or perhaps expressing Prolactin-related activity elsewhere in the body through other mechanisms. What should be noted first before moving on is the importance of having clinical research and data in order to verify claims. Unfortunately due to the current general legal status of anabolic steroids in the Western world (especially in the United States), clinical research and data is limited in the scope of this area. Research on the effects of various anabolic steroids in their use as performance and physique enhancers is very difficult to conduct because of the aforementioned legal status, and furthermore there are other limitations as well, such as: the allocation of funding for said research, the perceived need or requirement for such research, and so on and so forth. At this point in time, there is very little data to go by when the question of Prolactin and anabolic steroids is concerned (specifically in humans, and specifically at bodybuilding doses, and specifically under the conditions that a bodybuilder would use them).

For the time being, the best logical conclusions to make are diligent ones that acknowledge the lack of current research/information, and to understand that until such research is (hopefully) done in the future and data is gathered, theoretical knowledge and what we can discern from anecdotal evidence is the best we can do to form logical conclusions. Anecdotal evidence (as well as evidence in the form of blood test results) from many anabolic steroid users within the bodybuilding community have demonstrated significant PRL increases from those who use two particular anabolic steroids that are Progestins by nature: Trenbolone and Nandrolone (Deca-Durabolin). These hyperprolactinaemic afflicted individuals do also typically experience one or more of the typical Prolactin side effects: anrogasmia, lactation from the nipples, etc. There are also numerous individuals who can utilize these compounds without any hyperprolactinaemic blood results or symptoms. These anabolic steroids are also known and classified as 19-nor compounds (designating that these compounds lack a carbon atom at the 19th position, which is a carbon held by all other anabolic steroids). This is, by definition, what any 19-nor compound is. Further derivatives of Trenbolone or Nandrolone (such as Methyltrienolone) are logically also Progestins. By virtue of this fact, it is logical that individuals experiencing hyperprolactinaemia would come to the conclusion that because Trenbolone and/or Deca-Duabolin are Progestins, they must be causing the problem.

The truth is that as research has shown (referenced in the introduction of this article), Progesterone is in fact an inhibitor of Prolactin in numerous tissues throughout the body, including at the mammary gland. Therefore, the logical conclusion we can make is that the use of Progesterone and/or Progestins should not cause an increase in Prolactin levels, but should in fact have the opposite effect! However, this is only half of the total conclusion. For the other half, we must conclude that this is inconclusive due to a lack of direct evidence concerning Trenbolone and Deca and their effects, especially at bodybuilding dosages, on Prolactin levels.

The important concept to grasp at this point is the fact that Trenbolone and Nandrolone are derivatives of Progesterone, they are modifications of it. And these anabolic steroids can very well have unknown additional effects throughout the body in different tissues (as well as the endocrine system) due to these modifications. This is best compared to a similar situation: Anadrol 50. Anadrol 50 is a derivative of DHT (Dihydroteststerone). As such, Anadrol shares many similar characteristics with its parent hormone, as all derivatives of parent hormones normally do. Therefore, Anadrol 50 does possess the inability to interact with the aromatase enzyme and is therefore unable to aromatize (convert) into Estrogen. However, at the same time it is common knowledge that Anadrol carries with it a reputation for being one of the most notorious anabolic steroids to afflict Estrogen-related side effects on the user (bloating, water retention, gynecomastia, etc.). This doesn’t make sense considering it is unable to aromatize, so why is this the case? It is hypothesized that it is Anadrol itself and/or one or more of its metabolites that binds to the Estrogen receptor in different tissues in the body to exert these effects. Unfortunately this is a standing hypothesis that is yet to be tested and confirmed via clinical study, but considering everything we do currently know about Anadrol, there must be activity ongoing that does not conform to conventional logic here.

This is the same conclusion we must make with Trenbolone and Deca-Durabolin as they relate to Prolactin. They are derivatives of Progesterone and by all means should express the same anti-PRL effects, but their modifications possibly grant them other mysterious characteristics and qualities that might result in seemingly contrary activity to what conventional logic dictates to us (akin to the Anadrol example used). Furthermore, there are plenty of users who have used Trenbolone and Nandrolone with no Prolactin-related issues, and upon resuming use several cycles later, they experienced the Prolactin-related issues. The problem seems to be intermittent across the community, and until further research can be conducted that simulate/mimic the exact conditions under which anabolic steroid users use Progestins and experience Prolactin-related side effects, we will never know for sure.

These are the questions that need to be answered in the future if studies of this nature are to be conducted:

– The possibility of mysterious activity of Progestin anabolic steroids due to their chemical modifications.
– Ruling out the possibility that many anabolic steroid users are utilizing adulterated products (usually underground products) purchased on the black market as a result of prohibition. Many products do not contain the hormone that is advertised on the label, or they may contain additional hormones, or even additives of other sorts that might disrupt the endocrine system of the user in many unexpected ways.
– The role of Estrogens and other hormones that are vital to mammary growth and function in the midst of the use of anabolic steroids such as Trenbolone or Nandrolone.

Control, Elimination, and Prevention Prolactin
Control of Prolactin would ideally be the first and best possible course of action before any other measures are taken. An ounce of prevention is worth a pound of cure, after all, and it is not healthy in any way to completely eliminate Prolactin levels or allow them to rise above the normal range. As evidenced previously in this article, the control of Estrogen seems to be the key factor in maintaining low levels of Prolactin in the body, as Estrogen has a direct stimulatory effect on not only the synthesis of Prolactin at the pituitary gland, but also in facilitating mammary tissue function and development. As a matter of fact, one study conducted on female lambs involved the administration of Trenbolone along with Estradiol (E2) and another group of lambs with Estradiol-only, which resulted in the expected effect of Prolactin increases as a result of Estradiol, but the Trenbolone + E2 group experienced an anti-Estrogen effect from Trenbolone, preventing the mammary stimulus of Estrogen[1]. This is hardly surprising, considering it is common knowledge that androgens can and do decrease the number of Prolactin receptors in the body as well[2](especially strong androgens such as Trenbolone). Maintaining an high androgen:estrogen ratio is a key factor in controlling Prolactin as well, as evidenced by one study in which a subject experienced significant Prolactin increase during Testosterone administration as a result of the aromatization of the administered Testosterone into Estrogen[3].

Therefore, the first and foremost strategy for the anabolic steroid user should be to merely maintain Estrogen levels within the normal range during the use of any and all anabolic steroids, period.

If Prolactin has become a significant concern and prevention is no longer a viable course of action, various medications can be sought after in order to directly tackle the problem. As mentioned in the introduction of this article, dopamine is the body’s natural hormone/neurotransmitter that is responsible for the suppression of Prolactin secretion. As such, various medications known as dopamine agonists have been discovered and developed in the treatment of hyperprolactinaemia. Dopamine agonists (such as Cabergoline, Bromocriptine, and Pramipexole among many others) bind to the same receptors that dopamine itself binds to, and initiates similar effects (to different degrees), resulting in an inhibition of Prolactin secretion[4] [5] [6]. Estrogen control should be sought after simultaneously as well.


Thoughts???

 

[The ole man]

Well received an email from supplier that he is gonna resend the half order I didn’t get.
So now I got (when every thing arrives)
2 vials of test e 300
3 vials of tren e 200
2 vials of mast e 200
100 dbol tabs
50 whinny tabs.

So shit, maybe I should rethink this.

I feel like the mast, whinny should be together, but separate cycle from the dbol.
I’m at 13% bfi but lost a lot of size dropping from 24% bfi to 13%
So maybe should do a 12 week cycle
Without the mast, and save it for later with the whinny.
So test/dbol stack?
To keep my gains from the dbol, should I add some tren, or continue just with test?