Type-II'x Podcast: Gear, Growth, and Gains - Episode I: DHEA for crashed E2, LR3 IGF-I FACTS, allopregnanolone and pregnenolone (P5) & Epistane!

Type-IIx

Well-known Member

Enjoy!
Thank for the podcast, I enjoyed listening.
 
Interesting. Just bought dhea for this reason . Aromasin is a whole nother beast from compared to other AIs never again w that shit
 
Interesting. Just bought dhea for this reason . Aromasin is a whole nother beast from compared to other AIs never again w that shit
Depends on your exemestane dose with respect to AAS compounds and doses. There's nothing inherently bad about it as an AI compound selection. On the contrary, actually! Anastrozole (Arimidex) is by far the worst, for suppressing IGF-I, bone catabolism, and hair loss. Old drug, 1st gen. AI. Exemestane is a modern AI.
 
Depends on your exemestane dose with respect to AAS compounds and doses. There's nothing inherently bad about it as an AI compound selection. On the contrary, actually! Anastrozole (Arimidex) is by far the worst, for suppressing IGF-I, bone catabolism, and hair loss. Old drug, 1st gen. AI. Exemestane is a modern AI.
You recommend Exemestane over Arimidex? How’s the dosing schedule? EOD @ 12.5?
 
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Enjoy!


DHEA applied transdermally will mostly convert into androgens, namely androstenedione and DHT, and will further oppose estrogens instead of helping rise them.
 
Depends on your exemestane dose with respect to AAS compounds and doses. There's nothing inherently bad about it as an AI compound selection. On the contrary, actually! Anastrozole (Arimidex) is by far the worst, for suppressing IGF-I, bone catabolism, and hair loss. Old drug, 1st gen. AI. Exemestane is a modern AI.

Exemestane is definitely the best as it structurally an androgen, but will still bring about the deleterious effects low estrogens have.

Addtionnally, it has the potential to permanently desensitize e2 receptors through prolonged use as many users and myself found out.
 
I've heard that igf-lr3 can be used for lagging body parts. Essentially, pin the lagging muscle pwo. What's your opinion on this actually working like that?
 
DHEA applied transdermally will mostly convert into androgens, namely androstenedione and DHT, and will further oppose estrogens instead of helping rise them.
If you say so
Exemestane is definitely the best as it structurally an androgen, but will still bring about the deleterious effects low estrogens have.

Addtionnally, it has the potential to permanently desensitize e2 receptors through prolonged use as many users and myself found out.
If you say so
 
If you say so

Couple of studies in this thread for your culture :


I also have used transdermal DHEA for a year with 6 different bloodworks to assess so ;)

If you say so

Yes I say so, as well as at least 6 other forum members.
Would you be so obtuse as to refute anecdotal empirical evidence ?

If a study hasn't been conducted on a certain topic, you just put your head in the sand ? Hahahahahahaha
 
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