Sure, Deca increases serum homovanillic acid, a marker for dopamine metabolism (i.e., breakdown). Deca, even at 100 mg/w, significantly increases the breakdown of dopamine, and this is almost certainly associated with reduced dopamine receptor number in brain & CNS tissues. That is the likeliest explanation for depression and mood alterations caused by nandrolone.
Tren is not even of the same drug class as nandrolone, despite broscience that classifies these as "19-nortestosterone class" drugs. I wouldn't expect Tren to have the same effect on dopamine; in fact, there's reason to suspect opposite effects, increased dopaminergic activity.
I don't understand the reference to cabergoline. It's used clinically for prolactin-secreting adenomas (nothing at all like the androgen-induced increase to estrogens that increases, in some individuals, prolactin). Cabergoline is never recommended for AAS users.