How much Anavar for big deficit?

What is your consensus on tren for the purposes or what I am doing.

Would a small dose of tren A 10-20mg per day be more effective/equally safe at maintaining muscle on a harsh cut or even gaining muscle than a moderate dose of anavar 30-40mg?
Absolutely, not least of all because 140 mg weekly trenbolone acetate is incredibly powerful (with respect to AR potency alone that's equivalent to 619 mg testosterone weekly; more if accounting for relative molecular weight of acetate vs. enanthate/cypionate). The standard recommendation of 350 mg tren acetate weekly is absurdly high (a big mistake by Bill Roberts to have promulgated IMO) and absolutely spills over into myriad off-target systems throughout the body at such concentrations. You could use tren enanthate (2X weekly bolus) or hexyloxyphenylpropionate at 20 mg/wk (1X weekly bolus) and see highly favorable anticatabolic effects.
 
Sure, Deca increases serum homovanillic acid, a marker for dopamine metabolism (i.e., breakdown). Deca, even at 100 mg/w, significantly increases the breakdown of dopamine, and this is almost certainly associated with reduced dopamine receptor number in brain & CNS tissues. That is the likeliest explanation for depression and mood alterations caused by nandrolone.

Tren is not even of the same drug class as nandrolone, despite broscience that classifies these as "19-nortestosterone class" drugs. I wouldn't expect Tren to have the same effect on dopamine; in fact, there's reason to suspect opposite effects, increased dopaminergic activity.

I don't understand the reference to cabergoline. It's used clinically for prolactin-secreting adenomas (nothing at all like the androgen-induced increase to estrogens that increases, in some individuals, prolactin). Cabergoline is never recommended for AAS users.
I was always told to have Cabergoline on hand if you ever run Tren or nandrolone?

Pardon my ignorance, I’m still learning.
 
Absolutely, not least of all because 140 mg weekly trenbolone acetate is incredibly powerful (with respect to AR potency alone that's equivalent to 619 mg testosterone weekly; more if accounting for relative molecular weight of acetate vs. enanthate/cypionate). The standard recommendation of 350 mg tren acetate weekly is absurdly high (a big mistake by Bill Roberts to have promulgated IMO) and absolutely spills over into myriad off-target systems throughout the body at such concentrations. You could use tren enanthate (2X weekly bolus) or hexyloxyphenylpropionate at 20 mg/wk (1X weekly bolus) and see highly favorable anticatabolic effects.
On the subject of tren's spillover, what have you read that explains tren's effect on breathing? More importantly, any potential way to minimize that side effect? Obviously don't use too much lol. That's the one side effect that I just cannot deal with.
 
On the subject of tren's spillover, what have you read that explains tren's effect on breathing? More importantly, any potential way to minimize that side effect? Obviously don't use too much lol. That's the one side effect that I just cannot deal with.
Tren's apparent effect (it's very real I've experienced it) on, I believe VO₂ (basically cellular uptake of O₂ by muscle), must have something to do with Tren's effects on heart rate, stroke volume, and the difference in O₂ content between arterial & venous blood. I suspect it alters all of these factors; but it's not clear to me the relationship or directions of causation. Anyway, it results in apparent increased cardiorespiratory quotient (approaching hyperventilation). The increase to HCT/Hb can result in these symptoms, but on a more chronic time-frame. Everyone's using too much Tren, a great drug, but probably should be capped around 150 mg/wk. More drugs to treat these multiple effects is not the solution, reduced dose is.
 
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I was always told to have Cabergoline on hand if you ever run Tren or nandrolone?

Pardon my ignorance, I’m still learning.
Don't apologize for being confused bro, I am always confused by the claims about caber and where its use make sense (supposedly). I've seen its use with Tren being suggested because of prolactin and/or progesterone, but Tren doesn't increase prolactin (it reduces it actually); and it activates the PR (progesterone receptor), which has nothing to do with caber. I've seen it, most confusingly, claimed that Tren induced prostate growth calls for caber because ??? It's a total non-sequitur, Tren, prostate growth/BPH, and caber. Tren if anything reduces prostate size. I think, quite honestly, caber just gets thrown at every problem because it enhances sex by decreasing the male refractory period and is dopaminergic. It's effectively a recreational drug used by bodybuilders. It's dangerous stuff actually; bad for the cardiovascular system in particular.
 
Tren's apparent effect (it's very real I've experienced it) on,
Oh it most assuredly is real. The exact causes I've never really known and only read some plausible ideas. But when I am literally gassed from walking up 5 flights of stairs with a 10 pound back pack, something is horribly wrong.
I believe VO₂ (basically cellular uptake of O₂ by muscle), must have something to do with Tren's effects on heart rate, stroke volume, and the difference in O₂ content between arterial & venous blood. I suspect it alters all of these factors; but it's not clear to me the relationship or directions of causation. Anyway, it results in apparent increased cardiorespiratory quotient (approaching hyperventilation). The increase to HCT/Hb can result in these symptoms, but on a more chronic time-frame. Everyone's using too much Tren, a great drug, but probably should be capped around 150 mg/wk. More drugs to treat these multiple effects is not the solution, reduced dose is.
I swore it off years ago but I may give 100mg/wk a try. Thanks for sharing your insight.
 
Absolutely, not least of all because 140 mg weekly trenbolone acetate is incredibly powerful (with respect to AR potency alone that's equivalent to 619 mg testosterone weekly; more if accounting for relative molecular weight of acetate vs. enanthate/cypionate). The standard recommendation of 350 mg tren acetate weekly is absurdly high (a big mistake by Bill Roberts to have promulgated IMO) and absolutely spills over into myriad off-target systems throughout the body at such concentrations. You could use tren enanthate (2X weekly bolus) or hexyloxyphenylpropionate at 20 mg/wk (1X weekly bolus) and see highly favorable anticatabolic effects.
not to be argumentative. would you really suggest tren of that low dose for a cut? Have you used it?


would you say that is better than anavar or vice versa?
 
Yes. Yes. Yes.
I was thinking of using anavar vs mast vs a low dose tren for a cut...

mast just because I like it. but I keep playing with the thought of a low dose tren. was contemplating 100mg a week of tren hex but if you have done it on even a lower dose.

I like to cut on a cruise... I wanted to do a low dose AAS cruise of 300-400 max per week of AAS(depending on the type of combination.)


Have you had any sides or is it all benefit for you?
 
I am probably too much of a pussy and risk some mental sides. I will probably stick with daily anavar lol
I'm with you. I've read enough posts from members talking about the various sides they got, and I was like no way will I touch the stuff with what all I read. It's tempting to try a low dose on a cut...but nah.
 
I was thinking of using anavar vs mast vs a low dose tren for a cut...

mast just because I like it. but I keep playing with the thought of a low dose tren. was contemplating 100mg a week of tren hex but if you have done it on even a lower dose.

I like to cut on a cruise... I wanted to do a low dose AAS cruise of 300-400 max per week of AAS(depending on the type of combination.)


Have you had any sides or is it all benefit for you?
Var is great too for cutting, but Tren is just superior in a direct comparison. Var we know tends to shed ab fat via a hepatic ketogenic mechanism (triaglycerol lipase). Tren is potently antiadipogenic & insulin sensitizing (e.g., reduces PPARγ expression), reducing lipid accumulation, inducing lipolysis, and committing fat cell precursors to a myogenic rather than adipogenic lineage. Not to mention its potent anticatabolism (important during cutting). I certainly do get sides at Tren 350+ mg. I won't run it again unless a high quality lab comes out with tren hex on par with Balkan, and it'll be at <= 150 mg. I like Mast but I don't think it offers any unique fat loss benefits besides that which is AR-mediated (at a fairly low AR potency, unlike Tren). Mast I like too, but it doesn't fare well in a direct comparison with respect to fat loss benefits versus Var or Tren (it does very well at tissue-level estrogen modulation).
 
Probably all the tren I took it was fake as what everyone is writing about it I don't get it. And all steroids also...
Or the other options everything is very exaggerated
 
Probably all the tren I took it was fake as what everyone is writing about it I don't get it. And all steroids also...
Or the other options everything is very exaggerated
I just have a job where I have to be empathetic. I am afraid to lose that. thats my biggest concern with taking tren. I was contemplating starting this on a week I was off of work to see how I would do.
 
fuck it. i'll do tren hex(or E) at 50-75mg a week.
It can't be that bad.

ill do a cutting cruise of test U 200mg, 50mg of tren hex. Maybe low mast? Idk
 
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fuck it. i'll do tren hex(or E) at 50mg a week.
It can't be that bad.

ill do a cutting cruise of test U 200mg, 50mg of tren hex. Maybe low mast? Idk
I don't believe that you even notice 50mg of tren.
The low amount that I took was 200 mg and it was like I didn't took any. Didn't notice anything. Even if I was on test only or test and 200 mg tren was the same
 
fuck it. i'll do tren hex(or E) at 50-75mg a week.
It can't be that bad.

ill do a cutting cruise of test U 200mg, 50mg of tren hex. Maybe low mast? Idk
It wasn't for me. My mood/demeanor was the same as without, only heartburn after 6-7 weeks. Didn't affect my appetite either...
 
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