Testosterone acetate most powerful testosterone?

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HEY I HAVE BEEN LOOKING INTO WHICH TEST IS BEST FOR GAINS / SIDES AND MOST POWERFUL PER MG OTHER THEN JUST TESTOSTERONE SUSPENSION / TEST ACE LOOKS LIKE THE WINNER, MOST POWERFUL AND BEST ...WHY DO UG LABS NOT MAKE THIS STUFF??:confused:

CONTRARY TO WHAT PEOPLE SAY OR BELIEVE ALL TEST IS NOT EQUAL

EXAMPLE -
100MG TEST ACE = 83MG {SO 500MG TEST ACE IS REALLY 415MG}
100MG TEST D = 62MG {SO 500MG TEST D IS REALLY 310MG}

TESTOSTERONE ACETATE HAS MORE MG OF ACTUAL TEST THEN ANY OTHER TEST OUT THERE WITH A ESTER ATTACHED TO IT
- WHY DONT PEOPLE MAKE THIS STUFF AND YOU SEE IT ON THE MARKET?? EOD INJECTIONS WOULD BE GREAT, WOULD SHOW BETTER RESULTS AND THIS TEST WOULD NOT HAVE THE SAME PROBLEMS AS YOU SEE WITH TEST P
This is the same for Testosterone, Nandrolone, Trenbolone etc. ESTER WEIGHTS

Base: 100mg
Acetate: 83mg:drooling:
Propionate: 80mg
Isocaproate: 72mg
Enanthate: 70mg
Cypionate: 69mg
Phenylpropionate: 66mg
Decanoate: 62mg
Undecanoate: 61mg
Undecylenate: 60mg
Laurate: 56mg
 
who cares about mg of test per ml?? it is irrelevant really

I see some labs put out crazy concentrations like 400mg/ml and some fools get all excited that it is so powerful, give me a weaker concentration that stays in solution well and doesn't hurt as much in situ and that is what matters

and to answer your question, I think the problem with -acetates is that they are not as soluble, so that you can't get a very high concentration with them anyways, or they will crash, probably limited to around 100mg/ml I am guessing before you would start to see problems
 
Why not just up the dosage of a longer ester?

The only time I'd ever use a short ester is to finish up a cycle. Otherwise it's a hassle and literally a pain in the ass to use them.
 
Once the ester is removed Test is Test..

It's all timing.. Blood levels..

Why do some say Sust gives less water,
Then say Prop gives less water?

Answer: Cause people are retarded

TREN BABY!

O no, Mast P now
Just put in order, for 5 grams..
Should last me 3 weeks :D

I have used every type of test,suspension,prop,sustanon,enathate,and cypionate and the only way there was a difference was with dose.Higher dose gave greater effects.
 
It matters very little if an ester, or a suspension is chosen the average T peak is 36 hours.

However the reason T-s users begin to feel the effects sooner and with greater intensity (literally immediately post injection), is the absence of an attached ester.

This allows the suspension to "dissociate" into it's separate components (solute and solvent) thereby promoting prompt movement of testosterone into the blood stream.

Finally once the "suspension testosterone" enters the vascular system there's no waiting around for an esterase enzyme to cleave the ester, since only then can testosterone enter the cell.

jim
 
It matters very little if an ester, or a suspension is chosen the average T peak is 36 hours.

However the reason T-s users begin to feel the effects sooner and with greater intensity (literally immediately post injection), is the absence of an attached ester.

This allows the suspension to "dissociate" into it's separate components (solute and solvent) thereby promoting prompt movement of testosterone into the blood stream.

Finally once the "suspension testosterone" enters the vascular system there's no waiting around for an esterase enzyme to cleave the ester, since only then can testosterone enter the cell.

jim

So, when you say the T peak occurs 36 hrs post inject.

If the ester was enanthate, that would still be T with the ester attached and therefore unusable by the body?

So the estrified horomone enters the bloodstream via the IM depot, where the ester is cleaved AFTER entering the bloodstream? If so, would blood tests show elevated free T, or total T during this period? Even tho the user was feeling little to zero effect?

Also, does T without an ester always have the same half-life? Meaning we wait different amounts of time for the ester to be cleaved, then once this occurs the T always lives for 24-36 additional hours "post-cleavage";) lol.
 
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So, when you say the T peak occurs 36 hrs post inject.

If the ester was enanthate, that would still be T with the ester attached and therefore unusable by the body?

So the estrified horomone enters the bloodstream via the IM depot, where the ester is cleaved AFTER entering the bloodstream? If so, would blood tests show elevated free T, or total T during this period? Even tho the user was feeling little to zero effect?

Also, does T without an ester always have the same half-life? Meaning we wait different amounts of time for the ester to be cleaved, then once this occurs the T always lives for 24-36 additional hours "post-cleavage";) lol.

no... the ester is cleaved at the injection site, at 36 hours the peak he is talking about is free testosterone
 
Correct FY!

The 36 hour measurement can be either the Total testosterone or the Free testosterone since the peak averages 36 hours (+ or - 12 hours depending upon the ester)

Furthermore any esterified testosterone which escapes the depot or the immediately surrounding area, is rapidly cleaved by serum "esterase enzyme".

This enzyme is ubiquitous throughout the blood stream, since esters are a component of FATS and "de-esterification" is required for their metabolism.

Consequently "esterified testosterone" is NOT a part of either the free or total testosterone level.

The answer to your question Stretch is has already been answered by MW, once the ester is separated from testosterone "testosterone is testosterone" as is the half life.

jim
 
There are several reasons a "peak" value is reached at 36 hours. But suffice it to say "free testosterone" has a very short half life in the serum, because unless it attaches itself to a circulating protein it will enter the cell.

The type of ester determines how long a particular AAS remains in the depot, which is the primary determinant of the AAS half life because once exposed to esterase the anabolic won't be around much longer, perhaps a few hours at best.

jim
 
I've read related discussions in medicine which are much less germane once steady state levels are achieved, and drug toxicity is not problematic (such as AAS) IMO.

However when using particularly "toxic" medications, such as chemotherapy drugs which have a narrow therapeutic window, the differences are very important, AND settled with serial blood levels since that is the ONLY means of determining the operational kinetics of ANY drug

jim
 
I've read related discussions in medicine which are much less germane once steady state levels are achieved, and drug toxicity is not problematic (such as AAS) IMO.



jim

I'm sorry for being obtuse, but what are you saying here?

You've read related discussions in medicine which are less relevant once steady state levels are achieved?

I'm lost.
 
I will mention one factor (which was discussed in that debate) which has a marked impact on any IM drug but especially those which are oil based.

That is, the muscle chosen as the injection site may be more important then the ester itself.

Several studies have shown AAS deltoid injections double and may even triple an anabolics half life (compared to the gluteal region) ESPECIALLY if that muscle IS NOT exercised (deltoid in particular)

Consequently most studies use the gluteal muscle as the reference standard because the glutes are inherently "exercised" thru walking alone.

jim
 
I'm a thigh guy myself...just easier to do. I always wondered about how injecting and then exercising a specific muscle effected absorption.
 
I'm not familiar with any studies which looked specifically at "exercise" as a solitary variable, but I've no doubt any activity which enhance blood flow will increase absorption and shorten the half life.

What are you doing up so early mate? Are u in the office today?
:)
 
I found 100mg of testerone suspension ED very dramatic since there is no ester at all.You would have to do a very high dose for for a few weeks to get the same effects as 100mg of suspension,probably in excess of 1000 mg a week.....
 
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