With sleep being an issue, you'd be wise to consider dropping down to an actual physiological dose. Sleep and supraphysiological androgen dosages don't mix. And this doesn't only stand for dosages north of 2000 ng/dl - you might have issues at 1000 ng/dl, so considering an actual trt regime of 500 - 700 ng/dl might be a prudent idea. Taking in to account shbg and the free androgen index ofc.
I have to say that you are exhibiting a rather anxious phenotype and I would reconsider messing with aas to any extent whatsoever. If you think you have issues now, given time, your HPA axis will deteriorate and with it, so will your sleep, anxiety, prefrontal cognitive control, etc.
Another note on susceptibility to androgens related upregulation of "catecholaminergic" signaling. Any psychiatrist who deals with adhd will tell you, that there is a huge discrepancy on intraindividual sensitivity to catecholamines, ie. some users respond to 5 mg's of a stimulant with the same intensity as another person does to 40 mg's, or even more. This is a fairly normal and a common occurrence. Seeing as androgens also potentiate excitatory neurotransmission, one should take into account this same discrepancy, as can clearly be seen from all the varying responses to aas from different users. Somebody might get a huge motivational upbeat from 250 mg's of test and somebody else might not notice anything. Blasting and cruising is not for everybody in fact I'd say it's actually for a really small subset of users. Most should actually stay at a true trt dose and then potentially only sporadically cycle.
If sleep is a issue, hyperexcitability/anxiety is most probably the culprit, as it's the main factor which lowers sleep pressure (the thing that keeps you sedated and prevents you from waking up).
The state of your neurobiology is not static. Specifically the HPA axis does deteriorate, amygdala becomes hypersensitive and then you find yourself fiddling, micro controlling every little minuta while in essence what you should do is focus on HPA axis normalisation and only then, start messing with something as detrimental to the HPA axis as androgens. The brain is a very fluid environment, it undergoes heavy changes all the time and you only have a finite reservoir of stress management before mechanics undergo morphological changes - once it goes empty, you must repair the damage, otherwise you're just driving around with a faulty crankshaft with misaligned timings and all the while you're foolishly experimenting with different engine oils, trying to find a micro environment where things run just barely good enough in order for you to be able to function somewhat adequate.
Anxiety is a big determining factor of sensitivity to catecholamines (which includes aas). And everytime I see somebody caught up in such micromanagement, anxiety is the first thing that comes to mind. You think you have sleep issues? No, you have hyperexcitability issues, which are reflected in poor sleep. This is most notable in sleep maintenance issues. If you continue to push the dosages, it will get only worse with time and you'll need to employ more drastic measures to calm down the HPA axis (like a 3 to 6 month ssri regime for example, which actually might be something worth considering now).
