I don't think anybody here actually thinks BZD's can be non habit forming but kudos for stressing this out. The aas community's proclivity for searching quick solutions to a problem in a fast (direct) acting pill form, does indeed make everybody that much more likely to self prescribe gaba agonists or neuroleptics like gabapentinoids. I (as other members) have stressed the issue of using pregabalin for anxiety numerous time on the pages of meso. Anxiety and aas use have a strong positive correlation, which nobody really wants to acknowledge. The only viable long term solution is going to be something that regulates the HPA axis, calms down the amygdala, restores hippocampal volume, and it should lower excitatory neurotransmission, which is chronically elevated with aas use. SSRI's are excellent at this particular job, however unfortunately the reduction in executive functions control is for some quite noticeable. But, those are some sacrifices one should consider to make if muscle size is such a big life priority. Using something that modulates the glutamate elevations seen in aas use like lamotrigine is also an option, as besides regulating excitatory neurotransmission it to elevates BDNF. But newer AP's like cariprazine, which is a partial D3 and then D2 agonist is also on paper an intriguing idea. But for those who are afraid to touch psych drugs, going after inflammation might be the best option. Here I usually recommend inducing NRF2 via a good sulforaphane supp (Prostaphane - BROX) as the benefits are actually easily observable.
The shorter HL ones, specifically in the absorption phase, do have a higher addiction profile and longer HL BZD's are used to wean patients off of "shorter" ones, so again great you stressed this point too.
Fellas, anxiolytics are not the answer. If you've been cycling for a long time, chances are you've already induced a lot of morphological changes to your HPA axis and your primary goal should be to restore it's normal function. Dealing with anxiety directly while your HPA axis is dysregulated makes little to no sense as all the stressors which would normally provoke a low stress response are now heightened. Not to mention the detrimental effect of a hyperactive amygdala on the prefrontal cortex! And for the love of god, if you have crippling anxiety, which is defining your day to day, go to a true TRT dose for some time, regulate your HPA axis (you'll know when this happens, your state of mind/being will be 180 from your normal delusional, hyperexcitable state) and then slowly re-think your approach to aas.