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einstein1905 said:Just because someone has the money to have their ideas published, doesn't make them correct. I have the book. There is lots of good info in their. However, there is also lots of antiquated dogma in there that most of us have grown to learn isn't the best way to go about things.
einstein1905 said:The body does not convert to a catabolic state during longer cycles. The ability of AAS to effectively block corticosteroid receptors greatly reduces the effects of cortisol-mediated catabolism. As for your lipid profile, proper use of anti e's can keep your lipid profile well within range.
Not to sound too arrogant, but look at some of the things I said regarding longer cycles and means to counteract the plateau that comes with them. Far too long have I heard people preaching against ending cycles with higher androgenic compounds.....the opposite makes more sense to maintain levels of bioavailable AAS and counteract mounting SHBG and increased estrogen to androgen ratios. Another thing, longer cycles will not be nearly as effective when NOT using an AI. As the cycle progresses, estrogen levels will mount, which greatly increases SHBG levels. The "keep anti e's on hand" stuff is also a product of the days of shorter cycles....it's no longer applicable for optimal growth, since cycles have evolved into 10-15 week cycles on average.jimanold22 said:Hey bro's this is a very interesting post.Keep it going.I'm very interested because on longer cycle's I stop growing pretty much after the 9th week.Also like Seand95 with joint's start aching sometimes hurting after I'm of cycle for around 5-6 weeks.I thought it was caused by growing to fast or I thought maybe I was trining to heavy.You guys have cleared these issues up a little for me.keep it up.
jimanold22 said:Hey bro,Your not being arrogant.I did read the post about counteracting long cycle plateau's.I was not sure exactly about the use of anti-e's.Correct me if im wrong.In longer cycle's you want estrogen level's to be high to keep SHBG level's up?Im also not sure about this statement.
However, it's been shown in vitro (but not yet in vivo) that ARs do downregulate over time, when exposed to constant high levels of AAS (test). Androgens lower SHBG levels, whereas estrogens increase SHBG levels, so utilizing sufficient anti e's throughout a cycle can have advantages from many different standpoints.
Maybe I'm not catching what your saying exactly.
