Dbol/primo cycle = No hpta shutdown?

I was researching primobolan- specifically it's effects on the hpta axis when I came across this statement(admittedly along with several others just like it). What does everyone make of this article? *More specifically the parts that are bolded*
Is there any legitimacy to these claims, or just bro science?
.
.
.
.
.
"Some steroids only REDUCE TESTOSTERONE PRODUCTION(to varying degrees), whereas other steroids will SHUTDOWN the HPTA resulting in a complete cessation of androgen production.


*NOT ALL ANDROGENS CAUSE SHUTDOWN*

"Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.

SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Turinabol, Anavar, Halotestin, Wistrol, Equipoise, Dianabol, Masteron, Primobolan)

Very Androgenic/Progestenic/Estrogenic steroids(Trenbolone, Nandrolone, Anadrol, Testosterone) cause a COMPLETE shutdown of endogenous hormone production.

The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier recovery!


The Following steroids will NOT SHUTDOWN THE HPTA:


Turinabol, Anavar, Proviron, Halotestin, Wistrol, Equipoise, Dianabol, Masteron, Primobolan, Clostebol, and 4-ADiol.


Pre-PCT: PRE-PCT allows the HPTA to begin LH/FSH output, while still receiving additional anabolic support. This is the peroid of time where we utilize a NON-inhibitory steroid while the endogenous testosterone level begins to recover. This occurs PRIOR TO FULL PCT, so that by the time we begin full PCT the HPTA has already began recovering.


Active RECOVERY: The HPTA BEGINS to restore endogenous testosterone production once it detects the body's androgen level beginning to decline(end of cycle).

Therefore, HPTA CAN BEGIN TO RECOVER WHILE STILL IN AN ANABOLIC STATE!


The following drugs can be used during Active Recovery:

Anavar/Proviron= 40mgs/25mgs
Anavar/Masteron= 40mgs/300mgs
Primobolan/Masteron= 300mgs/300mgs
Turinabol/Proviron= 40mgs/25mgs
Turinabol/Masteron= 40mgs/300mgs
Winstrol/Masteron= 50mgs/300mgs
Dianabol/Proviron= 15mgs/25mgs
Dianabol/Masteron= 15mgs/300mgs


Examples...


In a SHORT CYCLE:
Weeks 1-4: Testosterone Propionate, 100mgs ED
Weeks 1-4: Dianabol, 50mgs ED
Weeks 1-4: NPP, 400mgs
Weeks 4-8: **PRE-PCT(ACTIVE RECOVERY)**
Weeks 8-?: **POST CYCLE THERAPY**


A Standard Cycle:
Weeks 1-6: Dianabol, 30mgs ED
Weeks 1-10: Testosterone Enanthate, 500mgs
Weeks 8-12: Winstrol, 100mgs ED
Weeks 12-16: **PRE-PCT(ACTIVE RECOVERY) **
Weeks 16-26: **POST CYCLE THERAPY**


DO NOT end your cycle ABRUPTLY! Don't just END your cycle cold-turkey! If you are SHUTDOWN, full restoration can take weeks and even MONTHS. Therefore, one should REMAIN ON minimally-inhibitive STEROIDS(HPTA) in an attempt to
MAINTAIN the gains they made while ON CYCLE, while STILL BEGINNING TO RECOVER TESTOSTERONE PRODUCTION. On top of that, one still continues to progess from the mild additional anabolic support.

NOT only does it mean that you can run a COMPLETE CYCLE with NO SHUTDOWN whatsoever(as long as the right compounds, dosages, and durations are used), it also means that if you ARE SHUTDOWN from your cycle, you do NOT HAVE TO COME RIGHT OFF CYCLE! Actually, it is BETTER TO STAY ON CYCLE WHILE YOUR ENDOGENOUS TESTOSTERONE LEVEL BEGINS TO INCREASE!


You may also run a cycle that COMPLETELY AVOIDS SHUTDOWN:

Weeks 1-6: Dianabol, 40mgs ED
Weeks 1-10: Anavar, 50mgs ED
Weeks 1-10: Masteron, 100mgs EOD

Or

Weeks 1-6: Dianabol, 40mgs ED
Weeks 1-10: Primobolan, 500mgs
Weeks 6-14: Turinabol, 60mgs ED


And Many many more! There are tons of NON-inhibitory cycles that you can devise using my my list above for your guideline. Your days of HPTA suffering are over!



By understanding WHICH steroids cause SHUTDOWN and which steroids do NOT, we can formulate a perfect EXTENDED CYCLE.


The Hypothalamus has Androgen, Estrogen, and Progesterone receptors.

Each and EVERY anabolic steroid affects these receptors DIFFERENTLY.

Some steroids affect ALL receptors, while some only affect ONE type of receptor, while others have very little effect on ANY of these receptors.

UNDERSTANDING WHICH steroids affect which receptors, and to WHAT DEGREE, will FULLY enable the steroid user to COMPLETELY and systematically AVOID HPTA SHUTDOWN!

By understanding WHICH steroids cause SHUTDOWN and which steroids do NOT, we can formulate a perfect EXTENDED CYCLE.

Steroids that cause an OVERSATURATION(too many receptors activated) of these various hormone receptors, WILL CAUSE SHUTDOWN.

Steroids that DO NOT CAUSE an OVERSATURATION of ANY of these various hormone receptors, will NOT cause SHUTDOWN!

The Following drugs either DIRECTLY or INDIRECTLY activate ESTROGEN receptors, to varying degrees:

Testosterone
Methandrostenolone
Mathandriol
Oxymetholone
Nandrolone
Boldenone

The Following drugs either DIRECTLY or INDIRECTLY activate PROGESTERONE receptors, to varying degrees:

Nandrolone
Trenbolone
Oxymetholone

The Following drugs activate Androgen receptors, to varying degrees:

Testosterone
Methandrostenolone
Mathandriol
Oxymetholone
Nandrolone
Boldenone
Trenbolone
Halotestin
Oxandrolone
Stanzolol
Chlorodehydromethltestosterone
Methyltestosterone
Methenolone...
(ALL AAS*)

As we can see, the steroids that cause HPTA SHUTDOWN either OVERSATURATE ONE SPECIFIC receptor, or they activate too many TOTAL receptors(Androgen/Estrogen/Progesterone)

For instance, Trenbolone causes HPTA SHUTDOWN because it OVERSATURATES BOTH, the ANDROGEN and the PROGESTERONE receptors.

Testosterone causes SHUTDOWN because it converts to ESTROGEN and DHT, therefore, it oversaturates the Androgen/Estrogen receptors.

As we can ALSO SEE, the steroids that DO NOT cause SHUTDOWN of the HPTA, do NOT oversaturate ANY of the different hormone receptors, and thus, do NOT cause SHUTDOWN.

Methenolone(Primobolan) does not possess ANY Estrogenic or Progestational ACTIVITY WHATSOEVER. It does, by virtue of being an anabolic steroid, posses a SMALL Androgenic component. Because it lacks ANY ESTROGENIC/PROGESTATIONAL component, and it lacks a strong Androgenic component, it WILL NOT CAUSE SHUTDOWN!

Oxandrolone(Anavar) posseses NO Estrogenic/Progestational component either. AND, it also lacks a strong androgenic component. Thus, Anavar will NOT cause shutdown."


By understanding WHICH steroids cause SHUTDOWN and which steroids do NOT, we can formulate a perfect EXTENDED CYCLE.
My issue with the "titrate down to allow endogenous testosterone to recover" thing is we already know that even a small trt dose of 100mg/week of pharma grade testosterone will eventually shut you down. So there won't be any "recovery" from simply lowering the dosage from your cycle even if you lower the dose significantly. Unless I am missing something?
 
I'm on TRT I blast and cruise also.
What I do is
1. Blast
2. Cruise
3. TRT
4. Lower than TRT
5. Come off completely no more than 6 weeks.

What I've noticed when I lower my dosage from trt and also come off is .
My libido is higher my balls are bigger and fuller and my semen production goes up.
When I lower my trt dosage to the minimum I'm feeling like I produce testosterone and when I come off also , I do feel better also and libido is sky high.

So maybe there's something to it. I don't tell that I start producing testosterone with lower dosage and completely off. But I do feel like it.
 
I'm on TRT I blast and cruise also.
What I do is
1. Blast
2. Cruise
3. TRT
4. Lower than TRT
5. Come off completely no more than 6 weeks.

What I've noticed when I lower my dosage from trt and also come off is .
My libido is higher my balls are bigger and fuller and my semen production goes up.
When I lower my trt dosage to the minimum I'm feeling like I produce testosterone and when I come off also , I do feel better also and libido is sky high.

So maybe there's something to it. I don't tell that I start producing testosterone with lower dosage and completely off. But I do feel like it.
Wondering if you’ve used hcg and if so how did you feel from libido and balls perspective versus this approach?
 
There's a lot of truth in this post. It's a more complicated approach that requires more discipline than most people are willing to have.
 
Back
Top