Enlarged Heart Left Side Ventricle

[Deadpool99]

Absolutely… I agree

This is one of those topics where you know what the 100% right thing to do is. It’s just a decision you have to make with yourself what you’re willing to sacrifice. I appreciate everyone’s opinions and advice on the subject.

Yep, I understand I'm gonna get some LVH with my tren/HGH but I try to mitigate it and take the necessary breaks, however a little LVH is expected.

Hopefully after I stop blasting and years down the line, it's not that big of an issue.

 

[47Ronin]

~95% of clinically significant LVH in AAS users happens in the context of elevated BP, high cholesterol. or stenosis (stiff valves, a process accelerated by high BP and cholesterol).

If you get your BP and lipids into ideal range, unless you have significant stenosis, you can almost certainly reverse most if not all the LVH (how many mm?) within a year or two and still use AAS with a very low risk of it returning.

We know what it takes to get those markers into ideal range, but an aggressive preventive cardiologist will be useful for accessing those meds and monitoring your progress. You'll also have to ditch the outdated "Pharmaphobia" that many still suffer from as a result of previous generation meds that had many "off target" effects and unknown outcomes.

The good news is this is likely a "near miss" that will get you to be serious about BP and lipids, and head those risk factors off earlier in life than most of your same age peers, preventing, and reversing damage that could ultimately safe you from a heart attack or stroke down the road.

Then you'll be the "I knew a guy who did tons of drugs, Coke, opioid, even steroids, and that fucker is 100 years old, so they must not be bad for you."

We will have to see where this road takes me. I’m definitely going to be working on the BP and cholesterol levels. Likely I will do a cycle again but I am going to pump the brakes for a little bit while I work on getting this under control. Just TRT for now.

 

[Ghoul]

We will have to see where this road takes me. I’m definitely going to be working on the BP and cholesterol levels. Likely I will do a cycle again but I am going to pump the brakes for a little bit while I work on getting this under control. Just TRT for now.

I wouldn't use anything other than TRT (real TRT, physiological levels) until you regress the LVH that's developed. I'm just saying that once it does regress, if BP and lipids are ideal, and you confirm no stenosis, it's extremely unlikely you'd develop LVH again regardless of AAS use. LVH requires excess pressure to develop. AAS makes it more likely in the presence of that excess pressure.

 

[Flatriggerman]

look into Telmisartan. It reduces LVH. Ask around for a Dr. that sees bodybuilders. My PA sees this all the time.

 

[Elektrobot]

If not, he’s gonna start me on some meds.

Maybe look into Carvedilol

I just started that today to try and improve my lv ejection fraction.

When reading about it I noticed it is also used to reduce LV hypertrophy

 

[hexagonal]

If you've got LVH I don't know why you would wait half a year to see if some minor lifestyle improvements regress it when we have medications with excellent safety profiles that we know can help regress it. It sounds like even your resting BP is above normal at 125/80, so if it's going above that the rest of the day when you're not resting, that's a problem.

An ARB like telmisartan will help with the BP and regression.

I'd be looking for a new cardiologist, personally. There are a lot of tools in the toolbox like telmisartan, cilnidipine, eplerenone, empagliflozin, etc.

I wouldn't be taking a "wait and see" approach to a known issue with my heart, personally.

 

[tomvet93]

I struggle to understand how lipids will impact LVH. I 100% understand the mechanism of lipids impacting plaque and then coronary perfusion. Struggle to then see how this causes LVH which as you've stated is typically caused by elevated systemic pressure or stenosis.

Also I struggle to understand how stenosis can be linked to AAS. Typically its caused by valve pathology or congenital size of the outflow tract?

 

[Almoister]

~95% of clinically significant LVH in AAS users happens in the context of elevated BP, high cholesterol. or stenosis (stiff valves, a process accelerated by high BP and cholesterol).

If you get your BP and lipids into ideal range, unless you have significant stenosis, you can almost certainly reverse most if not all the LVH (how many mm?) within a year or two and still use AAS with a very low risk of it returning.

We know what it takes to get those markers into ideal range, but an aggressive preventive cardiologist will be useful for accessing those meds and monitoring your progress. You'll also have to ditch the outdated "Pharmaphobia" that many still suffer from as a result of previous generation meds that had many "off target" effects and unknown outcomes.

The good news is this is likely a "near miss" that will get you to be serious about BP and lipids, and head those risk factors off earlier in life than most of your same age peers, preventing, and reversing damage that could ultimately safe you from a heart attack or stroke down the road.

Then you'll be the "I knew a guy who did tons of drugs, Coke, opioid, even steroids, and that fucker is 100 years old, so they must not be bad for you."

Damn didnt know its like 95% from this lvh is huge scare for me for some reason Thanks for this

 

[Ghoul]

Damn didnt know its like 95% from this lvh is huge scare for me for some reason Thanks for this

It's likely 100% IMO, with those who developed it in the absence of any of those resistance increasing factors just happening to be off cycle when it was discovered, and had normal blood pressure at the time.

Without the heart having to regularly pump against resistance, hypertrophy inducing irreversible LVH is as likely as it is to happen in the heart as any other muscle not subject to resistance. That is, low,

But the sad fact is, on or off cycle, most don't monitor their BP outside a doctor's office.