First, what was different about the conditions of testing in the first versus the second test? Were you fasted for both?
Certainly, GH has an indirect effect on HbA1c (it raises it) whereas IGF-I has an opposing effect on HbA1c (it lowers it). An increase in HbA1c may fairly be characterized as showing an increased hyperglycemia (GH-induced). HbA1c is a proxy measure of the glycosylated hemoglobin % and RBC turnover. All that matters is your A1C is healthy here.
The meaningfulness of Met's effects on IGFBPs and IGF-I serum levels is far from clear from the literature. Bovine granulosa cells do not function similarly enough to our healthy human cells, primarily skeletal muscle, et cetera; and besides, the interactions between IGF-I and its 6 binding proteins in some instances (i.e., in skeletal muscle) may increase its half-life and bioavailability. To confidently state that Met reduces IGF-I bioavailability (despite clearly augmenting serum IGF-I in response to rhGH treatment) with the current literature on the matter is a sure sign of a conman if you've heard that somewhere.