Seeking Hypersexuality is unwise. Just like amphetamins, it may feel great at first but it ends up 'downregulating' and one chases an unachievable state ... certainly nonsustainable.
At the core of hypersexuality is an impulse control/'addiction' situation. Seeking to be ever stimulated is also seeking to be never satisfied.
It should be noted that dopamine agonists can equally induce compulsive gambling as it can hypersexuality. Does one want to get in dept, loosing their money, home, spouse, and job due to compulsive gambling?
The studies put forth are primarily for treating sexual disorders. Not for pushing ones limits. One can argue, 'but I have a sexual disorder now due to AAS' - then the answer relies primarily with AAS. Also, one cannot have such a disorder even though caused by purely 'physical' reasons without being further compounded by developing comorbid psychological issues.
Now is it possible to improve an AAS induced problem with other meds? Probably. But it will be elusive, complicated and just as likely to compound the issues(s). This forum is full of post of 'instant' results or the expecting of such. Reality is that hormones and neurotransmitters are in a complex balance and changes take a long time to fully stabilize. They may very well stabilize in a state one does not wish to live in as sexual dysfunction well illustrates.
High prolactin -> Low testosterone = TRT in subjects...
Cabergoline in the Treatment of Male Orgasmic Disorder—A Retrospective Pilot Analysis
https://www.sciencedirect.com/science/article/pii/S2050116116000076
Nice study. The responders vs non-responders actually had very close T level increases (150 vs 129) but the responders had significantly greater drop in prolactin. It was stated that "
Of the 87 men undergoing concomitant testosterone therapy during cabergoline treatment, 51 were responders and 36 were non-responders (P ¼ .02) to cabergoline, accounting for 58.6% of all responders and 81.8% of all non-responders."
It is worth noting that cabergoline has many different binding sites. See Binding Profile in
Cabergoline - Wikipedia
Another thing lacking is: what was the magnitude of improvement for the responders? Of course, any improvement is welcome with such a frustrating problem. But it is incorrect the assume all these people were completely 'cured'.
To make things interesting, here is a study indicating that
low prolactin can contribute to sexual dysfunction.
Low prolactin is associated with sexual dysfunction and psychological or metabolic disturbances in middle-aged and elderly men: the European Male A... - PubMed - NCBI
