connoristiny
New Member
Are they essentially interchangeable? Is it worth spending the extra money for primo?
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What's better as cruise add on or both are not good idea?Mast is just a bit more androgenic, might benefit strength slightly greater vs Primo. They're virtually interchangeable.
I think either one is a fine idea for this (IMO better than upping the test), just avoid the temptation to turn your cruise into a blast.What's better as cruise add on or both are not good idea?
I'm cutting/cruising on 100 test/100 mast because why not.What's better as cruise add on or both are not good idea?
What are your goals?Are they essentially interchangeable? Is it worth spending the extra money for primo?
I think masteron will be more relevant in this matterThe two options would have different effect. you’ll have to try each as see which you prefer because your desired outcomes are probably different to mine.
I want to minimise weight gain while increasing my ability to recover from resistance exercise, so adding 100mg of mast makes more sense than upping my test dose from 200mg to 300.
Bulk/Pack on SizeWhat are your goals?
Test then, I assumeBulk/Pack on Size
Test base, then add primo or mastBulk/Pack on Size
This is spot on!!Max out to comfort doseage of test without an AI. Throw in in mast or primo to fill in. Obviously total AAS mg will depend on experience.
Just re-reading this. Presumably another difference @Type-IIx, is that Masteron disrupts signalling at the oestrogen receptor whereas Primobolan doesn’t?Mast is just a bit more androgenic, might benefit strength slightly greater vs Primo. They're virtually interchangeable.
There’s a suggestion that the following study presents evidence that Masteron “show some secondary anti-estrogenic effects” but I don’t have access to the paper and there’s no published abstract:My understanding is that Masteron negates E2 in two different ways. It has an effect similar to proviron, of occupying aromatise and preventing testosterone’s conversion. It also has a disruptive effect on genomic transcription at the E2 receptor.
Come to think of it I’ve not read any research to back this up. Could just be hearsay. I’m off to read the literature.