Stanford Pharmaceuticals

Just stopping by to say the pump from SP's anadrol is too legit2quit... Lighting sucks camel's cock in my gym, but y'all get the point.View attachment 85566

You are the first dude that I have ever seen wear a monocle while lifting. TRENDSETTER!

My eyes and brain seriously can't figure out WTF I'm looking at in this picture lol! There's so much flesh color I see a tit though lol!

Google 'speculum'. Actually, on second thought DON'T!!! You will be disturbed by much of what you see.:eek:
 
@Wunderpus Not sure if it's the lighting but you looking thicker than usual you added some thickness to your arms? Lookn good man
I've been working a lot on arms, and they have definitely grown. I lose them first when I cut, unfortunately. I've got about 1.5 months left of this mass gaining phase, so I'm hoping to continue to fill them out. Back and shoulders are moving along nicely, but arms were always a challenge... I honestly think training them multiple times a week has helped a ton....
 
Lookin as big as ever WP. How much you weighn in at?
Teetering around 240 right now. I'd love to hit 260 then cut down, not sure if that's going to happen this blast. Maybe next... I'm a bigger fan of slow and steady weight gain.
 
Can’t speak for tren A, but can for tren E. I actually considered lowering the dosage of his tren E at 600mg a week because the sides were getting intense. It’s def the most potent Tren I’ve taken, right up there with potg. I stuck it out and my body adjusted to the sides, but everything from Stanford has been on point. The anadrol is potent as well. It’s nice that Stanford doses things properly. It’s been a while since I’ve had gear this accurately dosed. Most of the time I have to OD on gear to get the desired result, but that’s not the case here.
 
I don’t post as often as I used to but I can’t stand by why others are putting themselves at risk. Lisinopril is an effective bp lowering medication but it’s much safer to go with the Ace II inhibitors if you must take bp medications. Lisinopril is an ACE I and has far more sides than the ACE II (losartan, valsartan etc..).

If you get the dry cough, clearing of the throat from lisinopril drop that shit, change meds or diet and lifestyle (if that works) and consider yourself lucky. Don’t be the guy in the ER in the middle of the night having respiratory difficulties (throat, tongue swelling).

They are different classes of drugs with different mechanisms of action. Lisinopril is an ACEI (angiotensin converting enzyme inhibitor). All the ones that end in "pril" are of the same class.
The "sartan" drugs are angiotensin II(2) receptor blockers. It is confusing how the shorthand notation uses the alphabet and roman numerals simultaneously.
Basically, the "pril" drugs prevent angiotensin 1 from converting into angiotensen 2 by inhibition of the angiotensin converting enzyme.
The "sartan" drugs don't mess with the conversion; but rather block angiotensin 2 from attaching to the cell receptors in the vessel walls.
Both are great medication classes; but the ACEI (prils) can cause a buildup of bradykinin, inducing a dry hacking cough. Emergently, ACEIs can cause angioedema (swelling of the face, and possibly the upper airway); and that is life threatening.
Generally speaking, the ARBs (sartans) have a well tolerated side effect profile with rare allergic reactions compared to the "prils".
Of note, all of the patients I have seen in my career with angioedema from ACEIs have been taking the medication for years without incident.....then, oneday they are just allergic to it.
 
They are different classes of drugs with different mechanisms of action. Lisinopril is an ACEI (angiotensin converting enzyme inhibitor). All the ones that end in "pril" are of the same class.
The "sartan" drugs are angiotensin II(2) receptor blockers. It is confusing how the shorthand notation uses the alphabet and roman numerals simultaneously.
Basically, the "pril" drugs prevent angiotensin 1 from converting into angiotensen 2 by inhibition of the angiotensin converting enzyme.
The "sartan" drugs don't mess with the conversion; but rather block angiotensin 2 from attaching to the cell receptors in the vessel walls.
Both are great medication classes; but the ACEI (prils) can cause a buildup of bradykinin, inducing a dry hacking cough. Emergently, ACEIs can cause angioedema (swelling of the face, and possibly the upper airway); and that is life threatening.
Generally speaking, the ARBs (sartans) have a well tolerated side effect profile with rare allergic reactions compared to the "prils".
Of note, all of the patients I have seen in my career with angioedema from ACEIs have been taking the medication for years without incident.....then, oneday they are just allergic to it.

Exactly, and this is why I posted about the ace 1 inhibitor lisinopril. Add me to the list of those that was taking this medication for years and suddenly had a reaction.

No other Bp medication that I encountered was as effective for me as lisinopril. After my reaction to this med, I’ve met the family members of several others that had reactions as well. My entire point was, this medication isn’t as safe as we may think. Just trying to bring awareness.
 
Exactly, and this is why I posted about the ace 1 inhibitor lisinopril. Add me to the list of those that was taking this medication for years and suddenly had a reaction.

No other Bp medication that I encountered was as effective for me as lisinopril. After my reaction to this med, I’ve met the family members of several others that had reactions as well. My entire point was, this medication isn’t as safe as we may think. Just trying to bring awareness.

Now that's a STRONG post about the SAFETY of these drugs FOUR (sic) everyONE reading this. ;):rolleyes:

Thanks for sharing StrongSafety41 !
 
Exactly, and this is why I posted about the ace 1 inhibitor lisinopril. Add me to the list of those that was taking this medication for years and suddenly had a reaction.

No other Bp medication that I encountered was as effective for me as lisinopril. After my reaction to this med, I’ve met the family members of several others that had reactions as well. My entire point was, this medication isn’t as safe as we may think. Just trying to bring awareness.

What are you taking now?
 
What are you taking now?

Now I’m taking nifedipine, it’s a calcium channel blocker. It’s seems to be keeping my bp down. I made the switch from a family practice doc to a cardiologist. Needless to say the lisinopril incident scared the shit out of me and encouraged the change of providers.
Some of these drugs have some pretty nasty sides man, just be as cautious as possible to what you’re putting in your body.
 
I'm not sure on this so if anyone knows please feel free to tell me to fuck off, but...

I think I read somewhere recently that a.i. will not prevent the conversion of dianabol to estrogen, that it is converted by some other method outside of the aromatase enzyme, hence an ai not helping as effectively as it would with regular testosterone conversion.

Again, if anyone is more familiar with this info please chime in and educate me. It's an interesting topic if I'm remembering correctly .
I believe you are thinking of Anadrol. I still love Anadrol though, and personally feel as though the bloat can be controlled.
 
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