THE new "Generic" HGH Assay PAGE! AAA testing

Well the fact is bc serum Somatotropin levels are almost exclusively used as a diagnostic test for those with ailments related to raised or suppressed GH levels, any deviation is NOT EVIDENCE BASED and highly susceptible to the "anecdotes" and whims of Bro-Science.

In addition regardless of your "experiences" the reason IGF rather than Somatotropin levels are used in MEDICINE as a measure of therapeutic efficacy is it's a MUCH MORE RELIABLE ASSAY and that's an evidence based fact.

Yes Sir , I understand ,
However, We are testin to see if the HGH is indeed HGH.
Some of us in the community use or possibly abuse AAS and other substances or for some other reason Have poor HGH conversion to IGF-1.
Many have cried that there HGH is poor or underdosed based on a single IGF-1 test, when in fact it is not.
NOW I am NOT say that is always the case , But in some instances People that are not knowin and have not tested themselves and how they react or process HGH under different circumstances just dont know how to read these results or misinterpret them due to lack of Knowledge....
The HGH Serum although very crude and poor at definin quantity is a very good, fast, cheap, way to show that there is HGH in that sample bein used. There is also 1 step removed, we dont need to go through the crapshoot of conversion, Meanin,the test shows the amount of HGH in the Blood at a said amount of time after administration. So manufacture to Vial to Blood to Lab, vs. Manufacture to Vial to Blood through conversion to lab....
Now if our intrest is to test how efficiently our bodys can convert HGH into IGF-1 @ different times and on different compounds, with possibly our livers bein compromised.
So Yes Sir, it is a More reliable test in its raw form used in a controlled group used for which it was intended for. not xactly the way we are tryin to use it.
Then Yr on Doc this would b the preferred test and Yes Sir it could show that the HGh is real, and crudely give us a idea of the amount we are takin provided we know how we react on the same....


Peace
 
And that is the classic bro-science justification for
NOT relying upon evidence based
scientific studies:

Unfortunately you have fallen prey to the same rationale propagated throughout PED forums with respect to why "we can't use scientific literature", the essence of which is; "we are all as different as are our purposes".

This same "logic" led to;

- The 19Nor / Prolactin connection

- AI / SERM "E-2 rebound"

- Deca heals joints

- AAS tapering as a substitute for PCT

- SERMS or AIs cure gynecomastia

- Everyone needs the same PCT regimine

- Everyone cycling AAS needs an AI

- bla, bla, bla

Welcome to BRO-SCIENCE!
 
And that is the classic bro-science justification for
NOT relying upon evidence based
scientific studies:

Unfortunately you have fallen prey to the same rationale propagated throughout PED forums with respect to why "we can't use scientific literature", the essence of which is; "we are all as different as are our purposes".

This same "logic" led to;

- The 19Nor / Prolactin connection

- AI / SERM "E-2 rebound"

- Deca heals joints

- AAS tapering as a substitute for PCT

- SERMS or AIs cure gynecomastia

- Everyone needs the same PCT regimine

- Everyone cycling AAS needs an AI

- bla, bla, bla

Welcome to BRO-SCIENCE!


Yes Sir,
Also remember that the scientific studys may not include People that live our life style.
my point has always been this;
Know the test you are using and how you react to it at different times.
dont try to make the test do something it cannot.
there are flaws in all of them.
since we are speakin about the IGF-1 test.
I and others can b on 0 HGH and have a elevated IGF-1 test on other things....
on the opposite end.
I and others can b on HGH and get little or no elevation in IGF-1 levels.
so to use this test to try and quantify or determine whether there is HGH in a vial Is yr Bro science.
We must know much more than directions to get to a Lab Draw facility... and even if we do Know how we test. we are still using a Crude way to do it. Or in other words tryin to make the test do something it is not intended to do.
The best way to see if our HGH is HGH and the amount in a vial is accurate Lab testing. We are on our own when we try any other form.
 
- The 19Nor / Prolactin connection
Don't mean to derail the thread, but:

I got myself bloods for prolactin. It was raised significantly at 50mg of trenbolone enanthate a week. The response was dose dependent up until 1g/wk, I did not try more. Just an anecdote of mine.
 
Don't mean to derail the thread, but:

I got myself bloods for prolactin. It was raised significantly at 50mg of trenbolone enanthate a week. The response was dose dependent up until 1g/wk, I did not try more. Just an anecdote of mine.
Were you on an AI?
 
Don't mean to derail the thread, but:

I got myself bloods for prolactin. It was raised significantly at 50mg of trenbolone enanthate a week. The response was dose dependent up until 1g/wk, I did not try more. Just an anecdote of mine.


Post a COPY of both the Pre and Intra-cycle Tren LAB REPORT
 
Post a COPY of both the Pre and Intra-cycle Tren LAB REPORT
That was years ago Mr. JIM.

I had full control over the process of creating the injectable from the trenbolone, testing included.

A friend of mine, who took my bloods was convinced there's prolactinoma growing tremendously fast in my head.

I truly just wanted to share my experience and it is not worth for me to argue over it.
 
[/QUOTE]
That was years ago Mr. JIM.

I had full control over the process of creating the injectable from the trenbolone, testing included.

A friend of mine, who took my bloods was convinced there's prolactinoma growing tremendously fast in my head.

I truly just wanted to share my experience and it is not worth for me to argue over it.
So did he suggest an MRI of pituitary?
 
Did your prolactin go back to normal post cycle? You are suspect on this forum so posting bloods would help your credibility.

Yes
[/QUOTE]

Don't have them archived and don't really care about being 'suspect'. Did it out of my own curiosity.

The prolactin went back to normal, not sure how fast, though, but about two months after cessation of use it was back where it was before the cycle.

Results?[/QUOTE]

None. Didn't go there. The waiting times for MRI in the country I had my insurance in at the time were about 9 months.

I'll be happy to continue the discussion in PM, I feel bad about being completely off topic here.
 
That was years ago Mr. JIM.

I had full control over the process of creating the injectable from the trenbolone, testing included.

A friend of mine, who took my bloods was convinced there's prolactinoma growing tremendously fast in my head.

I truly just wanted to share my experience and it is not worth for me to argue over it.

I'm NOT trying to argue but I can't tell you how many times I've heard this and NOT ONCE has someone posted bloods to support such an assertion!

It's just ludicrous how gullible some are with respect to this issue. However it's not surprising many 20 something years olds in addition to "vets" are running CNS drugs (dopamine agonists) that alter neurotransmission as a consequence.

And it's all based on BRO-SCIENCE!

FYI the highest prolactin level I've seen in well over fifty draws was 36ng/ml.

(ALL were cycling a 19Nor and were convinced an "elevated Prolactin" was the cause of their varied breast changes. Neither Prolactin nor Progesterone increased more than 10%
above baseline, and some actually deceased.)

Such a level is far removed from "clinical significance" as almost all patients with SYMPTOMATIC HYPERPROLACTINEMIA have levels well ABOVE 100ng/ml.
 
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And that is the classic bro-science justification for
NOT relying upon evidence based
scientific studies:

Unfortunately you have fallen prey to the same rationale propagated throughout PED forums with respect to why "we can't use scientific literature", the essence of which is; "we are all as different as are our purposes".

This same "logic" led to;

- The 19Nor / Prolactin connection

- AI / SERM "E-2 rebound"

- Deca heals joints

- AAS tapering as a substitute for PCT

- SERMS or AIs cure gynecomastia

- Everyone needs the same PCT regimine

- Everyone cycling AAS needs an AI

- bla, bla, bla

Welcome to BRO-SCIENCE!

Wait, serms dont cure gyno? Ralox & nolva no bueno?
 
I'm NOT trying to argue but I can't tell you how many times I've heard this and NOT ONCE has someone posted bloods to support such an assertion!

It's just ludicrous how gullible some are with respect to this issue. However it's not surprising many 20 something years olds in addition to "vets" are running CNS drugs (dopamine agonists) that alter neurotransmission as a consequence.

And it's all based on BRO-SCIENCE!

FYI the highest prolactin level I've seen in well over fifty draws was 36ng/ml.

(ALL were cycling a 19Nor and were convinced an "elevated Prolactin" was the cause of their varied breast changes. Neither Prolactin nor Progesterone increased more than 10%
above baseline, and some actually deceased.)

Such a level is far removed from "clinical significance" as almost all patients with SYMPTOMATIC HYPERPROLACTINEMIA have levels well ABOVE 100ng/ml.
I completely understand your stance, I did not see any evidence supporting it either, but trenbolone gave me issues, so I checked it out myself.
Running trenbolone again, so in case I get a chance to get bloods I'll do so.

Seems impossible for me to PM you.
 
@Eman , do you have a preference for combating gyno symptoms?

I've never had gyno symptoms, ever. The only reason I even run an AI is to help keep water retention under control because I do get hit with that but I've even run dbol without so much as an itch.

So, no lol... I don't have a legitimate preference. But with that said, I keep Ralox on hand at all times in case anything does happen. Never know.
 
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