Women of meso, whats yalls dose?

It all depends on what you can afford. I used 2 u.i. as a starting dose with Godtropin back in 2015, then 4 u.i. with black tops and later Humatrope. I didn't get any side effects from the Humatrope but all it did was lean me out a bit. Nothing earth shaking and I will never waste money on HGH again.

I agree with your statement. Many people will use HGH for the weight loss, but at the cost of it, not really worth it IMO.

Good diet, cardio, T3 with Clen will have better results faster and cheaper than HGH.

Frank
 
It all depends on what the goals are.

This is key here, and all GH-related threads should have the goals clearly stated because how one should administer their GH has goal-related dependencies.

A couple of key points here:
- Estrogen is a negative regulator of hepatic IGF-1 conversion so women are screwed in this regard (and why they have comparatively lower IGF levels than men on identical GH doses)
- Oral birth control makes this even worse
- Subcutaneous injections at night tend to create a more biologically active pharmacodynamic profile than morning injections based on multiple trials, so if only doing one injection per day, it may be wise to do it at night
 
I dont see many people getting substantial results

That's not the least bit surprising based upon existing scientific evidence, which becomes even more apparent in Gals who are run relatively low AAS dosages.

Nonetheless the anabolic effects of GH have been grossly exaggerated on PED forums and I suspect a considerable portion of the GH anabolic propaganda has it's origins in UGL pushing its sale.

. I didn't get any side effects from the Humatrope but all it did was lean me out a bit. Nothing earth shaking and I will never waste money on HGH again.

This too is entirely consistent with existing evidence
 
Fellow meso women.Got a question for yall.

Whats yalls typical dosage for hgh?
5 on 2 off? 3 on 1 off? 7 straight?
1 I.U? 1.5 I.U? Or more? A.M- P.M split?

I have seen so many different dosages for women. Just curious what yalls take on it would be pro's and con's.

Thanks


My suggestion, follow the package insert FEMALE dosing instructions of a Pharma product such as Humatrope, as its the most reliable means of limiting dosage related adverse effects.

A Pharma package insert is downloadable directly from the manufacturers web-site or indirectly thru Google.
 
Last edited:
That's not the least bit surprising based upon existing scientific evidence, which becomes even more apparent in Gals who are run relatively low AAS dosages.

Nonetheless the anabolic effects of GH have been grossly exaggerated on PED forums and I suspect a considerable portion of the GH anabolic propaganda has it's origins in UGL pushing its sale.



This too is entirely consistent with existing evidence
Anobolic effects do seem veryyy minimal with most women. Why would taking small amounts of aas reduce hgh effectiveness. Mask it ? The anti aging effect is probably more worth while if you don't mind spending the money long term. What are women like Susanne summers and christy Brinkley taking to look so good at 70 ? Besides a few surgeries. They have it figured out
 
Anobolic effects do seem veryyy minimal with most women. Why would taking small amounts of aas reduce hgh effectiveness. Mask it ? The anti aging effect is probably more worth while if you don't mind spending the money long term. What are women like Susanne summers and christy Brinkley taking to look so good at 70 ? Besides a few surgeries. They have it figured out

You have been reading to many
GH "anti-aging" blogs,

As for the MODELS "secretes" in today's society, exclusive of PS, look no further than great GENES
 
Idk quite a few dudes like Stallone holding back the years too. They are taking the good stuff

Sure it's gotta be only their secret GH concoction, rather than what I've already mentioned Good genes, Plastic surgery and MONEY
to afford it ALL!

I think if you saw them in the MORNING before their stylist, beautician, cosmetic specialist did their thing you would have a better picture of reality.
 
Sure it's gotta be only their secret GH concoction, rather than what I've already mentioned Good genes, Plastic surgery and MONEY
to afford it ALL!

I think if you saw them in the MORNING before their stylist, beautician, cosmetic specialist did their thing you would have a better picture of reality.

There's a lot of truth in your statement. I haven't seen one conclusive scientific data proving that HGH will reverse Father Time.

The fact is there's a lacking of evidence to prove HGH is the fountain of youth, and I don't need to be a doctor you say that either.

Frank
 
There's a lot of truth in your statement. I haven't seen one conclusive scientific data proving that HGH will reverse Father Time.

The fact is there's a lacking of evidence to prove HGH is the fountain of youth, and I don't need to be a doctor you say that either.

Frank

How ironic Stallone was mentioned as an icon of "youthfulness" since I recently saw two juxtaposed pictures of him in photo op PIC of and another unannounced gotcha PIStallone in one of those gossip mags.

Again the first was clearly a photo-op with him and several other "star actors" chatting with big smiles (and they all looked good) while the other was one of those morning "gotcha" PICs with Stallone doing whatever he could to hide his AVERAGE lackluster appearance.

The difference was remarkable and would make anyone believe these folk must be spending endless amounts of money on hair stylist, beauticians, and makeover artists!

To that end some have said; money "heals all wounds" - and wrinkles :)
 
Last edited:
We all must remember that "anabolism" does not equal "hypertrophy". GH is anabolic, but yet it does not directly lead to hypertrophy without the presence of AAS in the system.


Excellent point since BOTH anabolism and catabolism are required processes that occur simultaneously and independently in essentially every cell.

This is one of many reasons why
evaluating the hypertrophic effects of PEDs on SKM takes TIME

In addition markers of "anabolism" are
more readily demonstrated in cell lines with a rapid turnover rate (or reproduction rate) , such as the dermis and GI tract, while SKM
turnover approximates ZIPPO.
 
Last edited:
And knowing SKM cells do not undergo mitotic reproduction beyond the age of puberty, if not sooner, by what pathological mechanism (that which may be noted upon microscopic exam) is SKM hypertrophy possible?

1) enhanced glycogen storage

2) increased myofibril diameter or GIRTH (with few exceptions changes in myofibril LENGTH is not known to occur in humans!

3) recruitment of dormant "sarcopenic" cells

4) the development of additional
motor unit end plates

5) the loosening of cellular "gap junctions" (this process enables more fluid to distend the interstitial cell space bt cells)

6) ?

7) ?

And the million dollar question,
which of these processes are mediated or influenced by the use of PEDS, in HUMANS???
 
Last edited:
And knowing SKM cells do not undergo mitotic reproduction beyond the age of puberty, if not sooner, by what pathological mechanism (that which may be noted upon microscopic exam) is SKM hypertrophy possible?

1) enhanced glycogen storage

2) increased myofibril diameter or GIRTH (with few exceptions changes in myofibril LENGTH is not known to occur in humans!

3) recruitment of dormant "sarcopenic" cells

4) the development of additional
motor unit end plates

5) the loosening of cellular "gap junctions" (this process enables more fluid to distend the interstitial cell space bt cells)

6) ?

7) ?

And the million dollar question,
which of these processes are mediated or influenced by the use of PEDS, in HUMANS???

Very happy to have you in this board.

Honestly, having someone with scientific data and knowledge of how our cells react when a foreign substance is introduced is very important since Bro-Science continues to pop up on our board.

Frank
 
Very happy to have you in this board.

since Bro-Science continues to pop up on our board.

- THX

- In fairnes, Bro-Science originated bc of the ignorance of yesteryears "modern medicine"!

Although the 1970s era is a remote memory on my behalf
I do recall "contemporary" science denying AAS as PEDs, in spite of all the anecdotal evidence to the contrary.

-- however the impact of AAS as PEDs became apparent to all during the OLYMPIC GAMES! After several years of embarrassing losses to "commies"
modern medicine arose, with the net effect being PED, AAS in particular, evidence based research.

Fast forward to 1990s when a landmark study was published in the NEJM, to locate some of the first unequivocal evidence AAS promote SKM hypertrophy, in HUMANS!

Some 30-40 years later there's a plethora of evidence based research on many PEDs, (much of this data must be extrapolated from endocrine or infertility disorders) which has greatly diminished the imoortance of individual anecdotes and/or relevance of bro-science.

Nonetheless Bro-science continues to linger bc many prefer the "easy way" to becoming learned, not understanding being spoon fed will NEVER yield the same results or the fund of knowledge necessary to run these DRUGS in a manner that minimizes risk and optimizes benefit.
 
Last edited:
And it's the lack of a RISK vs REWARD assessment on behalf of "bro-science" that I find very troubling, as the absence of the former tends to glamorize the use of PEDs.
 
Last edited:
And knowing SKM cells do not undergo mitotic reproduction beyond the age of puberty, if not sooner, by what pathological mechanism (that which may be noted upon microscopic exam) is SKM hypertrophy possible?

1) enhanced glycogen storage

2) increased myofibril diameter or GIRTH (with few exceptions changes in myofibril LENGTH is not known to occur in humans!

3) recruitment of dormant "sarcopenic" cells

4) the development of additional
motor unit end plates

5) the loosening of cellular "gap junctions" (this process enables more fluid to distend the interstitial cell space bt cells)

6) ?

7) ?

And the million dollar question,
which of these processes are mediated or influenced by the use of PEDS, in HUMANS???


I didn't recall number SIX and SEVEN and so here they are.

6) an increase in the NUMBER of
contractile myofibril elements (actin & myosin)

7) a proportionate increase in
connective tissue stroma

Just to emphasize, the list includes only VISABLE MICROSCOPIC features believed responsible, some more than others, for SKM hypertrophy.

The responsible mechanisms, or physiologic pathways, are multi-faceted, complicated, convoluted,
and contain a variety of closed and open loop "feedback" forms of regulation.

Jim
 
Last edited:
deff a good read,not many can break it down as such. that said good genetics can only get ya so far,its pretty easy to see whos face has been stretched back, and who has some good drugs. Proper control of hormones and the right concoction of Gh can keep ya feelin and lookin younger for sure,at what cost is the ?
 
Back
Top