Can touching a barbell in the gym get you sick with the coronavirus?

That thing you said about it the possibility of a this Being like a chronic disease, where did you get that info from?
Good question. Initially read reports 'anonymous so they don't lose their career' that once you have it, it will stay in you. I dismissed it because I want to see data - anonymous or not. Don't care for conspiracy theories. But we know, for example, that several scientists in China were disciplined for saying things public. And there are governments and organizations that have put a gag order - all info must be released officially.

Here is one: "One virologist from UK who says he wanted to remain anonymous as he might be accused of causing panic or misinformation warned that the new coronavirus can be described in basic terms as a “slow airborne killer that will never let its victim off, even if it loses the battle the first time”,implying that even in those so called recovered , they might face reinfections or that the existing viral loads in the body will eventually cause some other new chronic disease." BREAKING! LatestCoronavirus Research Reveals That The Virus Has Mutated Gene Similar To HIV and Is 1,000 Times More Potent. - Thailand Medical News

There are things indicative of persistence.

First is the question of immunity after recovery. With the common cold (4 different coronavirus), immunity is often only a year so we keep getting the common cold. If they come up with a vaccine, then perhaps it will need to done annually. There are already reports of catching this virus twice in a short time ... time should reveal why Can you catch the coronavirus twice? We don’t know yet

Then there are already documented mutations. Some say that is why symptoms vary so much. Either way, will immunity to one provide immunity to another? With a pandemic, the pool of people and sheer volume of sickness leads to more mutations.

Another concern is how long one carries the virus before getting symptoms - if they get symptoms at all. The the average is 5.6 days, the range we hear is 2-14 days. There are cases of 27, 28, and 30 days in China. So this bug is stealthy toward the immune system. There also is the issue of viral shedding (still being contagious) for up to 8 days after recovery. https://www.atsjournals.org/doi/abs/10.1164/rccm.202003-0524LE

A big red flag is the 4 HIV inserts. When sequencing this virus last January, it is basically 80% the previous SARS. But the 4 independent copies of HIV pieces may be of concern. (PDF) Uncanny similarity of unique inserts in the 2019-nCoV spike protein to HIV-1 gp120 and Gag [ As a separate point, note: "The finding of 4 unique inserts in the 2019-nCoV, all of which have identity similarity to amino acid residues in key structural proteins of HIV-1 is unlikely to be fortuitous in nature". This indicates that this virus was man made ]

A key point is in the early statement, "viral loads in the body will eventually cause some other new chronic disease". There are viruses that stay in the body permanently. Herpes and HIV are famous. Some others:
  • HPV (a STD). Once a person get it, they are more likely to get cancer in the future.
  • EBV, does the same - just pick up an Oncology reference book and many cancers list EBV as a possible cause. Now most people with EBV titers are not symptomatic. I know 4 individuals who are. One is chronically ill, averaging 2 hours a day of work for the last 10 years. The other 3 get elevated EBV titers and symptoms whenever they get a flu.
When a virus stays in the body even without overt symptoms, it adds a load to the immune system. Thus increased cancers and increased sickness during sickness.

It remains to be seen what Covid19 will do. Clearly it is more vicious that the common coronavirus. Will it be chronic in comebacks? Will it hang around afterward and add viral load?
 
Well, we will only know the true death rate years from now.

Infected people are "in progress" so we don't know the outcome yet.
And dead people is a lagging indicator as more and more people get infected by the day.

Resolved cases mean either 'recovered' or dead.

Well, there are reports of "recovered" people getting infected again, or maybe the frickin virus never actually leaves the body.

So we have
1 Dead. Pretty clear.
2 Recovered. Some may go back to infected so we'll have to wait to see their outcome.

There's a third one
3 Fucked up for life. Many people get damaged lungs or CNS.

I believe 30-50% of the world population will be infected. 10% of them will die, and another 10% will be fucked up for life.
I hope it's not actually that bad.
 
Just another misleading headline, makes it look like 10
Well, we will only know the true death rate years from now.

Infected people are "in progress" so we don't know the outcome yet.
And dead people is a lagging indicator as more and more people get infected by the day.

Resolved cases mean either 'recovered' or dead.

Well, there are reports of "recovered" people getting infected again, or maybe the frickin virus never actually leaves the body.

So we have
1 Dead. Pretty clear.
2 Recovered. Some may go back to infected so we'll have to wait to see their outcome.

There's a third one
3 Fucked up for life. Many people get damaged lungs or CNS.

I believe 30-50% of the world population will be infected. 10% of them will die, and another 10% will be fucked up for life.
I hope it's not actually that bad.
And you pulled all these percentages out of your ass with a hook
 
Good question. Initially read reports 'anonymous so they don't lose their career' that once you have it, it will stay in you. I dismissed it because I want to see data - anonymous or not. Don't care for conspiracy theories. But we know, for example, that several scientists in China were disciplined for saying things public. And there are governments and organizations that have put a gag order - all info must be released officially.

Here is one: "One virologist from UK who says he wanted to remain anonymous as he might be accused of causing panic or misinformation warned that the new coronavirus can be described in basic terms as a “slow airborne killer that will never let its victim off, even if it loses the battle the first time”,implying that even in those so called recovered , they might face reinfections or that the existing viral loads in the body will eventually cause some other new chronic disease." BREAKING! LatestCoronavirus Research Reveals That The Virus Has Mutated Gene Similar To HIV and Is 1,000 Times More Potent. - Thailand Medical News

There are things indicative of persistence.

First is the question of immunity after recovery. With the common cold (4 different coronavirus), immunity is often only a year so we keep getting the common cold. If they come up with a vaccine, then perhaps it will need to done annually. There are already reports of catching this virus twice in a short time ... time should reveal why Can you catch the coronavirus twice? We don’t know yet

Then there are already documented mutations. Some say that is why symptoms vary so much. Either way, will immunity to one provide immunity to another? With a pandemic, the pool of people and sheer volume of sickness leads to more mutations.

Another concern is how long one carries the virus before getting symptoms - if they get symptoms at all. The the average is 5.6 days, the range we hear is 2-14 days. There are cases of 27, 28, and 30 days in China. So this bug is stealthy toward the immune system. There also is the issue of viral shedding (still being contagious) for up to 8 days after recovery. https://www.atsjournals.org/doi/abs/10.1164/rccm.202003-0524LE

A big red flag is the 4 HIV inserts. When sequencing this virus last January, it is basically 80% the previous SARS. But the 4 independent copies of HIV pieces may be of concern. (PDF) Uncanny similarity of unique inserts in the 2019-nCoV spike protein to HIV-1 gp120 and Gag [ As a separate point, note: "The finding of 4 unique inserts in the 2019-nCoV, all of which have identity similarity to amino acid residues in key structural proteins of HIV-1 is unlikely to be fortuitous in nature". This indicates that this virus was man made ]

A key point is in the early statement, "viral loads in the body will eventually cause some other new chronic disease". There are viruses that stay in the body permanently. Herpes and HIV are famous. Some others:
  • HPV (a STD). Once a person get it, they are more likely to get cancer in the future.
  • EBV, does the same - just pick up an Oncology reference book and many cancers list EBV as a possible cause. Now most people with EBV titers are not symptomatic. I know 4 individuals who are. One is chronically ill, averaging 2 hours a day of work for the last 10 years. The other 3 get elevated EBV titers and symptoms whenever they get a flu.
When a virus stays in the body even without overt symptoms, it adds a load to the immune system. Thus increased cancers and increased sickness during sickness.

It remains to be seen what Covid19 will do. Clearly it is more vicious that the common coronavirus. Will it be chronic in comebacks? Will it hang around afterward and add viral load?
This is my fear by moving forward too quickly.

https://www.google.ca/amp/s/www.usn...irus-patients-test-positive-again?context=amp
 
This is a persistent/stealthy bug. There were 10% (if I remember correctly) in China that they thought were cured but were not. In order to compare, I'd like to know if influenza A or B can come back after the tests says its gone.

In all probability, the test isn't sensitive enough. But it could be the bug gets 'dormant' for a while the out again as mentioned above. Also catching a mutation that escapes current immunity could be happening.

There is a lot of optimism for vaccines (in 12-18 months). But they don't have it for other coronaviruses (common cold or SARS or MERS) - so why this one? And after about 40 years of working on an HIV vaccine ... where is it?

Granted there is a lot more motivation to get a cure or at least a treatment. Suspect antivirals will be the first reality but we'll have to be on them frequently.


I don't post this way to be morbid. Its just serious. And worrying about jobs and economy is real too. But people still want to think this will go away quickly like waking from a bad dream. Or that its just something old people will need to worry about - and they don't matter, lol.

Disasters reveal a lot about people. This is a disaster ... but one mankind will learn to deal with and resume life again, albeit there must be changes.
 
[OA] SARS-COV-2 Infection Is Likely To Be Androgen Mediated

Coronavirus disease 2019 (COVID-19) pandemic fatalities are rare before adrenarche/puberty (<10 years of age), and the vulnerability of males to severe disease has been constantly reported over the past months of pandemic. The first biological step required for potential infectivity of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is the priming of the spike proteins by transmembrane protease, serine 2 (TMPRSS2).

Although other proteases were described to activate the spikes in vitro, only TMPRSS2 activity is regarded as essential for viral spread and pathogenesis in the infected hosts. TMPRSS2 may also cleave angiotensin converting enzyme 2 (ACE2) for augmented viral entry.

Androgen receptor activity has been considered a requirement for the transcription of TMPRSS2 gene as no other known TMPRSS2 gene promoter has been described in humans to date. Male vulnerability may be further enhanced by X-linked inheritance of genetic polymorphisms (androgen receptor and ACE2 genes loci are in chromosome X).

Obvious dermatological signs of hyperactivation of androgen receptors are: pattern reduction of density of scalp hair, increased density of facial and chest hair, acne, and oily skin. Theoretically, the hyperandrogenic phenotype might correlate with COVID19 increased viral load, increased viral dissemination and severity of lung involvement (Fig1).

Studies are still required for epidemiological conclusions. Acknowledging the importance of androgens during COVID19 pandemic may offer another targeted therapy for trials, with androgen suppression to reduce host vulnerability when infection risk is high.

Wambier CG, Goren A. SARS-COV-2 infection is likely to be androgen mediated. Journal of the American Academy of Dermatology. Redirecting

Capture.PNG
 
[OA] SARS-COV-2 Infection Is Likely To Be Androgen Mediated
Theoretically, the hyperandrogenic phenotype might correlate with COVID19 increased viral load, increased viral dissemination and severity of lung involvement
There are many differences between men and women besides testosterone.
While Covid 19 death and tested-positive statistics clearly are more men, without mass testing and adjusting for other factors (estrogen, smoking, risk-taking, types of jobs, eating habits, etc.) it will remain a theory.

Looking at this WHO chart, the number are all over the place. Death from respiratory infections is 7.3% for women and 6.7% for men https://www.who.int/whr/2004/annex/topic/en/annex_2_en.pdf

A man's lifespan is shorter than a woman. But is it solely androgens mediated?

Overall the #1 cause of death is cardiovascular. #2 is cancer. How about bodybuilders? Is there evidence that they are dying primarily from infections or cancer? Seems its like everyone else: cardiovascular. The greatest risk seen with prolonged AAS use is increased cardiovascular issues - not cancer or death-by-the-bug. Is not AAS use the epitome of hyperandrogenic?
 
Sneezes play an important part in the spread of infections, but we don’t know a huge amount about how they work. Lydia Bourouiba’s lab at MIT is trying to change that, using slow motion footage and other measurements to study the fluid dynamics of sneezing. The snot-spattered experiments that show how far sneezes really spread

Ultimately, says Bourouiba, her goal with this work is to ground epidemiology and public health in physics and mathematics. When trying to keep diseases from running rampant, she says, “we want to be giving recommendations that are based on science that has been tested in the lab”. In practical terms, such insights could lead to maps showing the contamination risks in the vicinity of infected people, protective equipment optimized to shield hospital workers from specific kinds of germs, and better predictions of how diseases move through a population.

...

The video evidence contradicted conventional thinking about sneezes, which held that larger droplets would fall to the ground within 1–2 metres, and that only the smaller ones would stay aloft as airborne aerosols. Feeding her video evidence into her mathematical models, Bourouiba concluded that, thanks to the cloud dynamics, many of the larger droplets can travel up to 8 metres for a sneeze and 6 metres for a cough, depending on the environmental conditions, and stay suspended for up to 10 minutes — far enough and long enough to reach someone at the other end of a large room, not to mention the ceiling ventilation system.

 
Ortho takes on COVID ...

01:48 - I don't bench over 250 so I can prone patients. I do it to fix bones! Stop acting like such a pansy.I bet you skip leg day and wear lifting gloves.
...

03:00 - 03:02 Our muscles will atrophy!
03:04 - 03:07 It's okay. We still have protein shakes.


 


It could all be over more quickly if certain existing drugs, already known to be safe for other uses, prove effective in treating covid-19. Trials are now under way; we should know by the summer. On the flip side, it may be that only a vaccine delivers the knockout blow, and even then, we still don’t know how long one will stay effective as the virus mutates.

That means we have to prepare for a world in which there is no cure and no vaccine for a long time. There is a way to live in this world without staying permanently shut indoors. But it won’t be a return to normal; this will be, for Westerners at any rate, a new normal, with new rules of behavior and social organization, some of which will probably persist long after the crisis has ended.

In recent weeks a consensus has started to build among various groups of experts on what this new normal might look like. Some parts of the strategy will reflect the practices of contact tracing and disease monitoring adopted in the countries that have dealt best with the virus so far, such as South Korea and Singapore. Other parts are starting to emerge, such as regularly testing massive numbers of people and relaxing movement restrictions only on those who have recently tested negative or have already recovered from the virus— if indeed those people are immune, which is assumed but still not certain.

This will entail a considerable degree of surveillance and social control, though there are ways to make it less intrusive than it has been in some countries. ...
 


Kazakh bodybuilder Yuri Tolochko has revealed his planned nuptials with sex-doll fiancee Margo have fallen victim to the coronavirus pandemic. Yes, you read that right.

Muscleman Tolochko recently sent social media into a spin when he announced his plans to wed plastic lover Margo, having first shared news of their relationship back in May of 2019.

Silicone sex doll Margo has her own Instagram page run by Tolochko, where the details of their intense preparations for the big day have been shared.
 


Kazakh bodybuilder Yuri Tolochko has revealed his planned nuptials with sex-doll fiancee Margo have fallen victim to the coronavirus pandemic. Yes, you read that right.

Muscleman Tolochko recently sent social media into a spin when he announced his plans to wed plastic lover Margo, having first shared news of their relationship back in May of 2019.

Silicone sex doll Margo has her own Instagram page run by Tolochko, where the details of their intense preparations for the big day have been shared.




 
Ortho takes on COVID ...

01:48 - I don't bench over 250 so I can prone patients. I do it to fix bones! Stop acting like such a pansy.I bet you skip leg day and wear lifting gloves.
...

03:00 - 03:02 Our muscles will atrophy!
03:04 - 03:07 It's okay. We still have protein shakes.


dude, what a riot.

Bros, dismissed! Love it.
 
[OA] SARS-COV-2 Infection Is Likely To Be Androgen Mediated

Coronavirus disease 2019 (COVID-19) pandemic fatalities are rare before adrenarche/puberty (<10 years of age), and the vulnerability of males to severe disease has been constantly reported over the past months of pandemic. The first biological step required for potential infectivity of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is the priming of the spike proteins by transmembrane protease, serine 2 (TMPRSS2).

Although other proteases were described to activate the spikes in vitro, only TMPRSS2 activity is regarded as essential for viral spread and pathogenesis in the infected hosts. TMPRSS2 may also cleave angiotensin converting enzyme 2 (ACE2) for augmented viral entry.

Androgen receptor activity has been considered a requirement for the transcription of TMPRSS2 gene as no other known TMPRSS2 gene promoter has been described in humans to date. Male vulnerability may be further enhanced by X-linked inheritance of genetic polymorphisms (androgen receptor and ACE2 genes loci are in chromosome X).

Obvious dermatological signs of hyperactivation of androgen receptors are: pattern reduction of density of scalp hair, increased density of facial and chest hair, acne, and oily skin. Theoretically, the hyperandrogenic phenotype might correlate with COVID19 increased viral load, increased viral dissemination and severity of lung involvement (Fig1).

Studies are still required for epidemiological conclusions. Acknowledging the importance of androgens during COVID19 pandemic may offer another targeted therapy for trials, with androgen suppression to reduce host vulnerability when infection risk is high.

Wambier CG, Goren A. SARS-COV-2 infection is likely to be androgen mediated. Journal of the American Academy of Dermatology. Redirecting

View attachment 128424
Yo, hurry up and claim that anabolic steroids help fighting the novel coronavirus

before El Chapo does with cocaine
Intense exercise, ephedrine and cocaine increase natural killer cells needed to fight coronavirus
 
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