Canadians talk domestic

Hi shredded

I'm new here, and on forum. I buy primo and npp from Nordic. I will start this week the cure 500 mg primo and 500 mg npp a week. My girlfrend will try anavar and primo pills.
 
Hi shredded

I'm new here, and on forum. I buy primo and npp from Nordic. I will start this week the cure 500 mg primo and 500 mg npp a week. My girlfrend will try anavar and primo pills.
Does your Gf have any previous experience running gear? If not tell her to put the brakes on before taking anything. First cycle of var is all she will need. @GearGodess would be someone to chat with before your Gf pulls the trigger. And make sure the Var is LEGIT bro lol
 
Does your Gf have any previous experience running gear? If not tell her to put the brakes on before taking anything. First cycle of var is all she will need. @GearGodess would be someone to chat with before your Gf pulls the trigger. And make sure the Var is LEGIT bro lol

@Cownan is on nordic var right now. He'll be able to tell you if it's real or not
 
@Cownan is on nordic Dr right now. He'll be able to tell you if it's real or not
Not bashing Nordic gear bro but batches can differ at times. Guys taking Var that happened to be dbol no biggie.A woman? Lol trouble! Imo if the var isn't legit pharma women must test all ug Var no exceptions.
 
Not bashing Nordic gear bro but batches can differ at times. Guys taking Var that happened to be dbol no biggie.A woman? Lol trouble! Imo if the var isn't legit pharma women must test all ug Var no exceptions.

Haha ya good point, didn't think of it from a woman's perspective
 
thanks for the Intel CdnGuy, someone else also pointed out it was a sister site of Muscle Gear, I think at least one of those sites also sells "Norma Hellas Deca" which as we know isn't even produced anymore for years.
 
thanks for the Intel CdnGuy, someone else also pointed out it was a sister site of Muscle Gear, I think at least one of those sites also sells "Norma Hellas Deca" which as we know isn't even produced anymore for years.
That's right the Norma i forgot! Saw those and started lmao SMH that shit was around when i started blasting in the early 90's haha
 
If you have bad kidney problems, you shouldn't take AAS at all.

Laboratory data and physical findings are presented in Table 2. At the time of renal biopsy, patients had variable elevations in serum creatinine, with mean serum creatinine of 3.0 mg/dl (range 1.3 to 7.8 mg/dl) and mean 24-h protein of 10.1 g/d (range 1.3 to 26.3 g/d). Among the 10 patients, five had recorded values for 24-h urine creatinine excretion, which ranged from 2.8 to 3.95 g/d. CrCl ranged from severely decreased (17.0 ml/min) to supranormal (196 ml/min) with an average of 96.2 ml/min. Because of the unusually large body surface area (BSA) of our patient population, values for 24-h urine protein excretion and CrCl that have been corrected for BSA are also presented in Table 2. Three of our 10 patients presented with full nephrotic syndrome, and two additional patients had nephrotic-range proteinuria and hypoalbuminemia in the absence of edema. Microhematuria was present in three of 10 patients, but no significant leukocyturia or casts were detected. Cholesterol levels were elevated in six of eight patients when these were measured (total cholesterol >200 mg/dl); of note, these elevations did not seem to correlate with the presence or absence of full nephrotic syndrome, reflecting known effects of anabolic steroid use on hepatic synthesis of cholesterol.17 Hematocrit at presentation was available for six of 10 patients. Mean hematocrit was 45.2% (range 41 to 50%), at the upper limit of normal range. Erythrocytosis is known to be an adverse effect of AAS use18 and has rarely been reported as a potential cause in development of FSGS.19


Table 2.

Clinical presentation
Renal biopsy findings are listed in Table 3. Sampling for light microscopy ranged from six to 61 glomeruli. Nine of 10 patients in our cohort had FSGS, four of whom displayed lesions of perihilar sclerosis and three of whom had collapsing lesions (Figure 3, A through C). One patient had no discrete lesions of segmental sclerosis but did have glomerulomegaly. Global glomerulosclerosis involved a mean of 32% of glomeruli (range 0 to 73%), and segmental sclerosis involved a mean 24% (range 0 to 47%). Tubular atrophy and interstitial fibrosis occupied a mean of 49% of the cortical area sampled (range <5 to 90%), and arteriosclerosis ranged from absent to moderate. Immunofluorescence was performed in all cases. There was weak (trace to 1+) segmental staining for IgM and C3 in areas of sclerosis in seven patients. There was no glomerular positivity for IgG, IgA, C1q, or κ or λ light chains. By electron microscopy, no significant GBM thickening was observed. No immune-type electron-dense deposits were identified in the eight cases studied; however, one case contained rare intramembranous lucencies, possibly representing the sites of resorbed deposits or remodeled GBM. FPE ranged from mild (15%) to severe (95%; Figure 3D). Electron microscopy was available for four of five cases with nephrotic-range proteinuria and hypoalbuminemia, for whom mean 89% FPE was noted (range 80 to 95%).


Table 3.

Renal biopsy findings

Figure 3.

Representative light microscopic and ultrastructural findings are shown. (A) Two hypertrophied glomeruli contain discrete segmental lesions of sclerosis and hyalinosis with adhesions to Bowman's capsule. There is prominent surrounding tubular atrophy ...
Clinical follow-up was available for eight of 10 patients (mean follow-up time 799 d; range 26 to 2127 d; Table 4). Patient 10 had severe renal impairment and extensive cortical scarring at the time of biopsy and progressed to ESRD within 1 mo of biopsy. The remaining seven patients with available follow-up have received treatment directed to the RAS in the form of an angiotensin-converting enzyme inhibitor, angiotensin II receptor blocker, and/or renin inhibitor. Patient 3, who was treated with oral prednisone, was the only patient who received any immunosuppressive therapy. All patients were encouraged to discontinue use of AASs and supplements, decrease dietary protein intake, decrease muscle mass, and reduce their exercise regimens. All seven patients experienced stabilization or improvement in serum creatinine and a reduction in proteinuria. Excluding the patient who progressed to ESRD, average creatinine decreased from 2.34 to 1.61 mg/dl, and mean 24-h urine protein declined from 9.47 to 1.83 g after discontinuation of AASs. The single patient who subsequently resumed AAS abuse developed progressive renal insufficiency and marked increase in proteinuria.

Development of Focal Segmental Glomerulosclerosis after Anabolic Steroid Abuse
 
What do i need to check on my blood test for my kidney? I dont think im having big problems but when running anadrol i feel weird my appetite decrease i dont feel that well.. with my digestion this is why i want something smoother like dbol.. i just want to keep my kidney safe so

Anadrol and super drol are the two worst oral for your health. Dbol is harsh too but not as bad. If you want something "safer" try tbol or anavar. Nothing is safe for your kidneys, even creatine and eating too much protein can stress your kidneys if you over do it.
 
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