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Out of light, at room temp, unbroken stopper, 5 years+ without significant change in potency. The alcohol acts as a preservative for the oil.

Beyond that the cheap stoppers tend to break down, but in theory, the gear itself could easily last 10 years+.
I usually refilter and recap with silicone stoppers, just for my peace of mind. Thanks brother!
 
Thanks brother!

Out of light, at room temp, unbroken stopper, 5 years+ without significant change in potency. The alcohol acts as a preservative for the oil.

Beyond that the cheap stoppers tend to break down, but in theory, the gear itself could easily last 10 years+.
I usually refilter and recap with silicone stoppers, just for my peace of mind. Thanks brother!

Make sure those are PTFE coated stoppers.
 
Out of light, at room temp, unbroken stopper, 5 years+ without significant change in potency. The alcohol acts as a preservative for the oil.

Beyond that the cheap stoppers tend to break down, but in theory, the gear itself could easily last 10 years+.

Make sure those are PTFE coated stoppers.
Idk what med labs are and I ordered KsTek now. I hope they are the good ones.
 
Sorry bro… you’re the second person I’ve convinced today. Couple reasons: 1) it’s smoother 2) more mg per ml 3) it’s cheaper 4) the endurance + pumps are insane 5) just add mast and now you have something better than primo
Just out of curiosity, what is your preferred ratio of test : EQ : mast?
 
Just out of curiosity, what is your preferred ratio of test : EQ : mast?
Depends on goals and if HCG or no HCG.

I’m trying to get faster / better cycling. So I’m low test / lowish mast / higher EQ. No estrogen issues, HCG helps create more e2.

Low test for less water retention.

210 test / 280 mast / 700 EQ.

If I was just pure BB, would be high test / med mast / high eq.

1.25g test / 600 mast / 1200 EQ

HCG + HMG cuz I’m trying to maintain fertility
 
Depends on goals and if HCG or no HCG.

I’m trying to get faster / better cycling. So I’m low test / lowish mast / higher EQ. No estrogen issues, HCG helps create more e2.

Low test for less water retention.

210 test / 280 mast / 700 EQ.

If I was just pure BB, would be high test / med mast / high eq.

1.25g test / 600 mast / 1200 EQ

HCG + HMG cuz I’m trying to maintain fertility
Ah, I see. EQ dosage can be tricky with the E2 inhibition, which is why I asked, but I'm guessing the HCG/HMG helps to mitigate that in your case. I've been hesitant to go over 300mg/week with EQ in the past because I'm paranoid of lowering my E2 too much and having to wait it out with that long ester. At the same time I hear from many people that EQ really shines at higher dosages, so I'm always debating creeping the dose up slowly over time to see how close I can get to a 1 : 1 ratio of test : EQ before significantly impacting my E2. A few months back I was at 500 test : 300 EQ and noticed my libido took a minor hit after a few weeks, so I dialed the EQ back (although I didn't get bloodwork done, so I admittedly don't have hard data to back up any claim that my E2 took a hit). Anyway, I appreciate the input. Anecdotes can be helpful to get a ballpark estimate of where most people find their sweet spot.
 
Ah, I see. EQ dosage can be tricky with the E2 inhibition, which is why I asked, but I'm guessing the HCG/HMG helps to mitigate that in your case. I've been hesitant to go over 300mg/week with EQ in the past because I'm paranoid of lowering my E2 too much and having to wait it out with that long ester. At the same time I hear from many people that EQ really shines at higher dosages, so I'm always debating creeping the dose up slowly over time to see how close I can get to a 1 : 1 ratio of test : EQ before significantly impacting my E2. A few months back I was at 500 test : 300 EQ and noticed my libido took a minor hit after a few weeks, so I dialed the EQ back (although I didn't get bloodwork done, so I admittedly don't have hard data to back up any claim that my E2 took a hit). Anyway, I appreciate the input. Anecdotes can be helpful to get a ballpark estimate of where most people find their sweet spot.
Man it’s going to be so individual. The other thing to consider is that while E2 may be low, Estrone can be sky high enough such that you don’t get low estrogen symptoms.

But once again, so individual. Primo murders my e2. TBD on EQ impact on my e2. Couple more weeks and I’ll go get bloods. But I feel amazing so that’s usually a good indicator I’m not totally fucked.
 
If the oil isn’t touching the stopper, then it shouldn’t deteriorate it at all right? I dont see medlabs say if the silicone stoppers are PTFE coated or not. KStek ones dont say either.


That link is to 2 mL vials. They also have 10 mL vials.
 
Just curious, why would you do that?
I’m female and use baby doses, so my 200mg/ml vial will last me 2-3 years. I’m not too crazy about using the same stopper for that long and figure if I can transfer into 4 2ml vials (did one cycle already), there’ll be less chance of the rubber stopper disintegrating. Full transparency, the promo I have now is from Q, and their stoppers are atrocious.
 
Man it’s going to be so individual. The other thing to consider is that while E2 may be low, Estrone can be sky high enough such that you don’t get low estrogen symptoms.

But once again, so individual. Primo murders my e2. TBD on EQ impact on my e2. Couple more weeks and I’ll go get bloods. But I feel amazing so that’s usually a good indicator I’m not totally fucked.
The relationship between EQ, E1, and E2 is still something that I've yet to find a thorough explanation of. My shallow understanding is that EQ has a very high binding affinity to aromatase, much more so than testosterone. By competing with testosterone for aromatase and "winning," EQ essentially prevents testosterone from binding to it and being converted into E2, while EQ itself will be converted into E1 via aromatase. Over time, this scenario would simultaneously raise E1 and lower E2, leading to a very high ratio of E1 : E2. This would seemingly cause some issues, but yet, as you said, the elevated E1 could also mask the usual symptoms of low E2, so it's difficult to know what is really going on under the hood without bloodwork. Adding further to the confusion, I've also read that if E1 levels are high enough, small amounts of E1 can actually be converted into E2. It's at this point that I just throw my hands up and shrug, lol.

It seems like such a crap shoot trying to juggle test and EQ dosages to find the proverbial golden ratio for an individual, and even then, that ratio could fluctuate over time within the same individual depending on various variables. And this is also assuming that test and EQ are the only compounds in the mix. So part of me gets concerned and preoccupied with all of this, but then another part of me reads the last lines of your post where you state how you feel amazing, and my mind is like, "Well, fuck it, if you're feeling great, making gains/progress, and libido is on point, who fucking cares what the bloodwork reads?" It's a delicate balance, lol, but I see your overall point on the individuality of it all.
 
can someone please send me a pic of an anavar tab? i’ve the feeling i got something wrong and would like to do a visual check, before potentially sending to a lab.
 
The relationship between EQ, E1, and E2 is still something that I've yet to find a thorough explanation of. My shallow understanding is that EQ has a very high binding affinity to aromatase, much more so than testosterone. By competing with testosterone for aromatase and "winning," EQ essentially prevents testosterone from binding to it and being converted into E2, while EQ itself will be converted into E1 via aromatase. Over time, this scenario would simultaneously raise E1 and lower E2, leading to a very high ratio of E1 : E2. This would seemingly cause some issues, but yet, as you said, the elevated E1 could also mask the usual symptoms of low E2, so it's difficult to know what is really going on under the hood without bloodwork. Adding further to the confusion, I've also read that if E1 levels are high enough, small amounts of E1 can actually be converted into E2. It's at this point that I just throw my hands up and shrug, lol.

It seems like such a crap shoot trying to juggle test and EQ dosages to find the proverbial golden ratio for an individual, and even then, that ratio could fluctuate over time within the same individual depending on various variables. And this is also assuming that test and EQ are the only compounds in the mix. So part of me gets concerned and preoccupied with all of this, but then another part of me reads the last lines of your post where you state how you feel amazing, and my mind is like, "Well, fuck it, if you're feeling great, making gains/progress, and libido is on point, who fucking cares what the bloodwork reads?" It's a delicate balance, lol, but I see your overall point on the individuality of it all.
Yep, you nailed it in a lot of places. You strike me as someone who really analyzes decisions, potentially over analyzes, and unfortunately I gotta tell you that trial and error is going to be necessary in this game.

The last part of your post, I think is the most important part.

Here’s my advice: start with a 2:1 or 1.5:1 test to EQ ratio and adjust from there. EQ long ester makes it particularly difficult to change dosing so rather start on safer side than find out it tanks your estrogen and makes you feel like shit.

Worst case? Throw some dbol and HCG in and your estrogen will come out of the gutter quick.

Best case? A 1.5:1 ratio or even less works perfectly for you.

A lot of my decisions are now “fuck it, I feel great” compared to “oh no my hormones are off in blood work so I should fix that.”
 
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