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Interesting, my assumptions were based on the thread that @mands started in the HGH forum.

I believe you are currently running both, is that right mands? Thoughts on combining vs cycling between the two?

If mk truly only boosts within what is naturally possible perhaps it still has value as a way to cycle off of GH while maintaining peak natural levels?
 
Let me suggest you read a few PEP evidence based studies.

Unlike AAS GH HTPA recovery only
requires a few days (maybe a couple weeks) rather than months.
 
Please correct me if I am wrong becsuse I am still learning. The serum GH level base is usually low, somewhere in the 0.1-1 range. His bloodwork read a little over 14. Not as high as I have seen but certsinly a whooping change. On top of that the IGF1 number was approaching the magic 500. If IGF1 is expensive to make and given the combination of the two tests, is it safe to say that it is probably legit HGH?
 
Please correct me if I am wrong becsuse I am still learning. The serum GH level base is usually low, somewhere in the 0.1-1 range. His bloodwork read a little over 14. Not as high as I have seen but certsinly a whooping change. On top of that the IGF1 number was approaching the magic 500. If IGF1 is expensive to make and given the combination of the two tests, is it safe to say that it is probably legit HGH?

The serum test was drawn almost exactly at the 3.5 hour mark, so it is possible that the serum score was on the way back down. Based on everything we have seen I do not believe there is any doubt that the product contains GH. The thing we are primarily testing is the dosage, since it is marketed as 24iu.

So far, things look good imo. Of course, time will tell if bloods continue to come back with solid numbers.
 
Interesting, my assumptions were based on the thread that @mands started in the HGH forum.

I believe you are currently running both, is that right mands? Thoughts on combining vs cycling between the two?

If mk truly only boosts within what is naturally possible perhaps it still has value as a way to cycle off of GH while maintaining peak natural levels?
I am actually doing opposite of what you are proposing. I am currently running 20mg MK-677, 10mg before bed and 10mg in the morning.

I had baseline IGF-1 levels in the 90's and I will get IGF-1 tested soon on MK-677. I will then add 2 iu's of GH for one week and test again. After that will increase to 5 iu's and test again.

My theory and bases on studies of MK-677 and GH that one can use these drugs synergistically each at lower amounts to increase IGF-1 levels significant and at a cheaper cost to the consumer. All while cutting down on insulin resistant by cutting down the amount of actual GH being taken.

mands
 
I am actually doing opposite of what you are proposing. I am currently running 20mg MK-677, 10mg before bed and 10mg in the morning.

I had baseline IGF-1 levels in the 90's and I will get IGF-1 tested soon on MK-677. I will then add 2 iu's of GH for one week and test again. After that will increase to 5 iu's and test again.

My theory and bases on studies of MK-677 and GH that one can use these drugs synergistically each at lower amounts to increase IGF-1 levels significant and at a cheaper cost to the consumer. All while cutting down on insulin resistant by cutting down the amount of actual GH being taken.

mands


Aha, this is what I originally thought based on your posts in the other thread. Thanks for the clarification. I will continue to watch for developments over there.
 
I am actually doing opposite of what you are proposing. I am currently running 20mg MK-677, 10mg before bed and 10mg in the morning.

I had baseline IGF-1 levels in the 90's and I will get IGF-1 tested soon on MK-677. I will then add 2 iu's of GH for one week and test again. After that will increase to 5 iu's and test again.

My theory and bases on studies of MK-677 and GH that one can use these drugs synergistically each at lower amounts to increase IGF-1 levels significant and at a cheaper cost to the consumer. All while cutting down on insulin resistant by cutting down the amount of actual GH being taken.

mands

If I understand your post correctly what you are suggesting is analogous to running a SERM while using AAS to diminish HTPA suppression.

The effects of either blunt the
benefits.

Moreover how will you prove said changes in IGF are the result of MK or GH.

Finally IMO no conclusions can be made with such limited (one person) analyses.

Best
Jim
 
If I understand your post correctly what you are suggesting is analogous to running a SERM while using AAS to diminish HTPA suppression.

The effects of either blunt the
benefits.

Moreover how will you prove said changes in IGF are the result of MK or GH.

Finally IMO no conclusions can be made with such limited (one person) analyses.

Best
Jim
I guess I don't really see it as analogous to that specifically.

I believe testing with a consistent MK dose will provide me with an pretty good idea of what MY IGF-1 levels are running 20mg a day. I want to cross reference my numbers from studies with similar dosing for comparison. I will add the GH to my existing MK dose then test, increase and test again. I'm hoping there will be a constant increase.

I agree with you completely, one such analyses isn't good for warranting a definite conclusion. It could warrant further testing for me IMO.

mands
 
I guess I don't really see it as analogous to that specifically.

I believe testing with a consistent MK dose will provide me with an pretty good idea of what MY IGF-1 levels are running 20mg a day. I want to cross reference my numbers from studies with similar dosing for comparison. I will add the GH to my existing MK dose then test, increase and test again. I'm hoping there will be a constant increase.

I agree with you completely, one such analyses isn't good for warranting a definite conclusion. It could warrant further testing for me IMO.

mands
I have to say if it proves to be effective could cut GH cost significantly.
 
I guess I don't really see it as analogous to that specifically.

I believe testing with a consistent MK dose will provide me with an pretty good idea of what MY IGF-1 levels are running 20mg a day. I want to cross reference my numbers from studies with similar dosing for comparison. I will add the GH to my existing MK dose then test, increase and test again. I'm hoping there will be a constant increase.

I agree with you completely, one such analyses isn't good for warranting a definite conclusion. It could warrant further testing for me IMO.

mands

The MOA of PEPs is they act as secretagogues stimulating the
pituitary to produce GH thereby raising IGF.

However the net of effect also increases somatostatin secretion
limiting the IGF rise.

So someone who is running supplemental GH will have HIGH
Somatostatin levels negating the
benefit of PEPs.

That being said I suppose one might could justify the use of secretagogues such as MK as an aid to "GH recovery".
 
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