master.on
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You forgot ORAL minoxidil.
Big foot growth in a bottle.
Big foot growth in a bottle.
MESO-Rx
Anabolic Steroids
Hair Loss
- Thread starter billydidit
- Start date
Classifications of Patterned Hair Loss: A Review
Patterned hair loss is the most common cause of hair loss seen in both the sexes after puberty. Numerous classification systems have been proposed by various researchers for grading purposes.
These systems vary from the simpler systems based on recession of the hairline to the more advanced multifactorial systems based on the morphological and dynamic parameters that affect the scalp and the hair itself. Most of these preexisting systems have certain limitations.
Currently, the Hamilton-Norwood classification system for males and the Ludwig system for females are most commonly used to describe patterns of hair loss.
In this article, we review the various classification systems for patterned hair loss in both the sexes. Relevant articles were identified through searches of MEDLINE and EMBASE. Search terms included but were not limited to androgenic alopecia classification, patterned hair loss classification, male pattern baldness classification, and female pattern hair loss classification. Further publications were identified from the reference lists of the reviewed articles.
Gupta M, Mysore V. Classifications of Patterned Hair Loss: A Review. J Cutan Aesthet Surg 2016;9(1):3-12. Classifications of patterned hair loss: a review Gupta M, Mysore V - J Cutan Aesthet Surg
Patterned hair loss is the most common cause of hair loss seen in both the sexes after puberty. Numerous classification systems have been proposed by various researchers for grading purposes.
These systems vary from the simpler systems based on recession of the hairline to the more advanced multifactorial systems based on the morphological and dynamic parameters that affect the scalp and the hair itself. Most of these preexisting systems have certain limitations.
Currently, the Hamilton-Norwood classification system for males and the Ludwig system for females are most commonly used to describe patterns of hair loss.
In this article, we review the various classification systems for patterned hair loss in both the sexes. Relevant articles were identified through searches of MEDLINE and EMBASE. Search terms included but were not limited to androgenic alopecia classification, patterned hair loss classification, male pattern baldness classification, and female pattern hair loss classification. Further publications were identified from the reference lists of the reviewed articles.
Gupta M, Mysore V. Classifications of Patterned Hair Loss: A Review. J Cutan Aesthet Surg 2016;9(1):3-12. Classifications of patterned hair loss: a review Gupta M, Mysore V - J Cutan Aesthet Surg
John Bahlmer
New Member
Steroid 5α-Reductase Type 2 Deficiency.
-William's Textbook of Endicrinology, 13th ed.
Steroid 5α-reductase type 2 deficiency is also characterized by a 46,XY karyotype, normally differentiated testes, and male internal ducts but external genitalia that may be more ambiguous at birth than in 17β-HSD deficiency. There is a striking degree of virilization at puberty in patients reared as female with gonads retained. Classic features of this enzyme deficiency are summarized in Table 23-15. The description of a genetic variant in SRD5A2 in the Dominican Republic and Mexico and analysis of the biochemical and molecular features underline the importance of DHT in the development of the male phenotype.303-305 At birth, there is typically a bifid scrotum, a urogenital sinus, a blind vaginal pouch, and a clitoris-like, hypospadiac phallus. Testes differentiate normally and are located in the inguinal canal or in the labioscrotal folds. No müllerian structures are present. The wolffian ducts are stabilized so that the epididymides, vasa deferentia, and seminal vesicles are well differentiated; the ejaculatory ducts usually terminate in the blind vaginal pouch. The prostate is hypoplastic. Up to a third of cases may present with isolated hypospadias.306
Individuals assigned male gender virilize to various degrees at puberty. The voice deepens, muscle mass increases, the phallus lengthens to 4 to 8 cm, the bifid scrotum becomes rugated and pigmented, and the testes enlarge and descend into the labioscrotal folds. Postpubertal affected males do not have acne, temporal hair recession, or enlargement of the prostate, and they do not develop gynecomastia. There is normal libido with penile erections. Histologic examination of the testes shows Leydig cell hyperplasia and decreased spermatogenesis. Infertility is caused by a composite of failure to transform spermatogonia into spermatocytes, the adverse effect of a cryptorchid testis, and the specific role of DHT in regulating semen volume and viscosity.307 Nevertheless, some members of the Dominican cohort had a normal sperm count. One man fathered a child after intrauterine insemination, and two affected brothers in a Swedish family were spontaneously fertile after hypospadias repair performed in childhood.308,309
-William's Textbook of Endicrinology, 13th ed.
Steroid 5α-reductase type 2 deficiency is also characterized by a 46,XY karyotype, normally differentiated testes, and male internal ducts but external genitalia that may be more ambiguous at birth than in 17β-HSD deficiency. There is a striking degree of virilization at puberty in patients reared as female with gonads retained. Classic features of this enzyme deficiency are summarized in Table 23-15. The description of a genetic variant in SRD5A2 in the Dominican Republic and Mexico and analysis of the biochemical and molecular features underline the importance of DHT in the development of the male phenotype.303-305 At birth, there is typically a bifid scrotum, a urogenital sinus, a blind vaginal pouch, and a clitoris-like, hypospadiac phallus. Testes differentiate normally and are located in the inguinal canal or in the labioscrotal folds. No müllerian structures are present. The wolffian ducts are stabilized so that the epididymides, vasa deferentia, and seminal vesicles are well differentiated; the ejaculatory ducts usually terminate in the blind vaginal pouch. The prostate is hypoplastic. Up to a third of cases may present with isolated hypospadias.306
Individuals assigned male gender virilize to various degrees at puberty. The voice deepens, muscle mass increases, the phallus lengthens to 4 to 8 cm, the bifid scrotum becomes rugated and pigmented, and the testes enlarge and descend into the labioscrotal folds. Postpubertal affected males do not have acne, temporal hair recession, or enlargement of the prostate, and they do not develop gynecomastia. There is normal libido with penile erections. Histologic examination of the testes shows Leydig cell hyperplasia and decreased spermatogenesis. Infertility is caused by a composite of failure to transform spermatogonia into spermatocytes, the adverse effect of a cryptorchid testis, and the specific role of DHT in regulating semen volume and viscosity.307 Nevertheless, some members of the Dominican cohort had a normal sperm count. One man fathered a child after intrauterine insemination, and two affected brothers in a Swedish family were spontaneously fertile after hypospadias repair performed in childhood.308,309
John Bahlmer
New Member
Ok, so my above blurb comes from an endocrinology textbook and describes people that were born with 5 Alpha-Reductase Type II deficiency. I assume this would be equivalent to a normally born person taking high doses of finestride since birth.
Why, despite having normal to slightly elevated levels of testosterone during and after puberty, enough to allow partial virilization, do these subjects not display male-pattern baldness?
If it was simply the androgen receptor, male-level levels of testosterone should cause some level of hairloss even in people that do not have dht, shouldn't they?
Also, these people had normal libido and erections. If finestride has all of these fucked up sexual side effects these people especially should be impotent, shouldn't they?
So is testosterone even a cause of male-pattern baldness, or does DHT and other steroids have some effect that testosterone lacks independent of the so-called "androgenic rating".
Any clarification would be nice.
Why, despite having normal to slightly elevated levels of testosterone during and after puberty, enough to allow partial virilization, do these subjects not display male-pattern baldness?
If it was simply the androgen receptor, male-level levels of testosterone should cause some level of hairloss even in people that do not have dht, shouldn't they?
Also, these people had normal libido and erections. If finestride has all of these fucked up sexual side effects these people especially should be impotent, shouldn't they?
So is testosterone even a cause of male-pattern baldness, or does DHT and other steroids have some effect that testosterone lacks independent of the so-called "androgenic rating".
Any clarification would be nice.
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Johndoe
New Member
My hairline bloody hell it went down hill fast shower full of hair just destroyed it since using I now just rock the shaved head and full beard look
I have long, curly, quite distinctive hair. I really don't want to lose it! But these compounds to save it seem to cost a fortune... I don't think I can afford them on top of gear, bloods and PCT...
Johndoe
New Member
It's all about genes mate if you have a lot of bold men in your family dad brother grandad etc then gear could make you loose your hair like a Britney Spears melt down but if not it might not effect it at all.
A lovely turn of phrase!
I just spat my water out and my tshirt is wetter than Whitney's last joint!
Not much baldness in my family.
Grandparents thinning to losing it in their seventies... Dad still has loads of hair at sixty. I'm late 30s...
I just spat my water out and my tshirt is wetter than Whitney's last joint!
Not much baldness in my family.
Grandparents thinning to losing it in their seventies... Dad still has loads of hair at sixty. I'm late 30s...
Johndoe
New Member
Sounds like male pattern boldness ain't in your family then pal my hair started thinning in my teens, then i hit the gear later on in life test cycle hair was fine hit the deca and nap 50s and my hair just went poof clumps of hair on my pillow shower every place now I rock the shaved hair and beard look [emoji23] (some gear is a lot!! Harder on your hair line then others look for a post or ask about for ones to avoid
)
kpaltizer
New Member
Thanks for posting, I had just asked about this the other day but never got a good answer. I started going bald about 22 and as soon as it started showing I shaved it off. 41 now and started TRT recently and doing my 1st cycle soon. I'm obviously not worried about any falling out but because I suffer from excess dht should I look into getting my doc to put me on one of these. Would it just help decrease dht therefore increasing test levels or could I really start regrowing hair? Thx
Bill lane
New Member
Appreciate the post, Im sure tons of people have wondered about this.
Canadianhulk
New Member
Great post, just found this. My hair is thinning out a bit now that I am older so want to try this. Any other have tried this with success as OP posted?
Ive been bald for a couple years. Will any of this help regrow what has been gone for awhile?
new to the game
New Member
Thanks for sharing your experience
BuckEighty
New Member
This stuff was recommended to me by my derm. I took one month of it and stopped, because I was already spending a lot on rogaine and finasteride. Maybe I should pick some more up...
Big pump
New Member
Follicular unit transplantation is expensive, prices ranging from $4000 – $9,000. Guess if you are already bald or losing significant amounts this might be a option. Of course if you continue to use aas wouldn't it be just defeating the purpose regardless of what you use?? Hmm jmo
Big pump
New Member
AlwAys roll like Donald Trump! You can't even tell..... or the comb over. Just fn with you all. My whole family is bald so no biggie. There are good options but in the end genetics will prevail.
Bundy55
Banned
Finasteride can cause libido issues. Not minoxidil from what I understand.
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