You're very wrong if you think 6 weeks of a long acting ester of test and then 10 days of winstrol is not enough to require PCT. That cycle will raise androgen levels above baseline, supressing natural production, for a good 7-8 weeks. Back when Dan Duchaine talked about the short cycle concept, he recommended only 2 weeks on, using a short acting ester. That's it. Bill Roberts, Blade, and others have reitereated 2 week cycles as being short enough to cause minimal HPTA suppression and little need for PCT. On the other hand, experts like Bryan Haycock have talked about cycles of 4-5 weeks being too long to not have PCT... "a waste of time" due to the loss in gains that will ensue.
Keep in mind that I am not a heavy lifting body builder relying on the ability to make the most seemless transition from cycle to off and continuing to work out at the max efficientcy possible. Regardless, there are many out there that feel the same as I do regarding 6 weeks. I did forget the fact of the steroid he stacked for a week at the end, however, I am guessing non negligible considering. I and sure that from experience, I can barely feel test by week 6, and I have tested afterwards at both 2 weeks and 8 weeks. Completely out of effective half-lives and had no suppression. There is no doubt that a six week cycle of test only is useless. Speaking for myself of course. The is also a majority that would agree with that.
Is a 6 week cycle of test E going to suppress you as much as a 12 week cycle? No. But will it suppress you enough that you should worry about PCT? I think the answer is a definite yes. Everyone I know has a clear case of testicular atrophy after 6-8 weeks on gear (when not using hCG). That alone is a big sign that PCT should be a part of your plan.
I have NEVER had any testicular atrophy at six weeks. Even at 650 mg per week avg, and ramping up, the anser for me is no. It usually takes between weeks 8-12 to start to see any nut shrinkage. I can' get them to "suck up" till week 15, and still, if I back down the dose to 4-500 mgs/wk, they will drop back down in 2 weeks flat... However, perhaps I should not be using so much personal data as everyone is different.
I don't think your idea of going right into a cut is very smart. I don't think it's very smart to reduce the loads any more than you have to, either. It makes a lot of sense to continue lifting heavy during PCT, to try to maintain the stimulus that provoked the gains seen during the cycle. A reduction in load during PCT, at the same time test levels are low and recovering, at the same time you cut calories and start dieting sounds like one bad recipe.
I'm not sure what bro told you Nolva is the devil, but I think you're very mistaken. Nolva has risks, sure, but they're not disportionate to other SERMS. Nolva is not some "crude" anti estrogen. Chemically, it's very similar in structure to clomid and toremifene. It works very stimilar to them as well.
AS far as going to cutting. What I meant was psychologincaly giving up the enlargement workout process. Not even a diet change anything down to "normal" is then maintained. I guess what I was saying about Nolva being the devil is that while the are both somewhat technically the same, the clomid being stronger I feel is too agressive by nature regardless of the dosage adjustment. AND while the do work somewhat similarly, they do also work somewhat differently. While that stament may be politically ambigous, it is true. I have also read that clomids simply has a better effect on kickstarting the body's natural systems!??? Hence the clomid is always the final, and best vehicle to set up recovery. This is also proven in the fact that most body builders will utilize clomid for PCT after attempting PCT with Nolva first. Lets just cut that chase.
As for "killing your prostate," do you have any evidence at all? A quick look on pubmed and google shows that doctors actually proscribe nolva to those who have prostate cancer. Research has also suggested a possible localized treatment for benign prostate hypertrophy. I see nothing about tamoxifen administration harming the prostate in vivo. So again, I'll ask you to post an academic reference, something that substantiates your claims as more than just a bad case of bro-lore.