Hi Meso!
First post here, got an interesting question and hoping some of the members here have some experience with this.
I’m planning on the following at some point in a few months;
Weeks 1-12/14: Test-E 500mg/week
Weeks 1-4/6: Dianabol 40mg/Daily
Weeks 1-12/14: Arimidex 1mg EOD/Daily if required should Gyno be an issue
Weeks 1-12/14 - HCG 500mg/week
PCT - 2 Weeks after last Pin
Clomid 100/100/50/50
Nolva 40/40/20/20
Note: I have put Weeks 1-12/14 and Weeks 1-4/6 because the length of the cycle may be either 12 or 14 Weeks. I have enough gear for 14 weeks and will probably decide during cycle to run 12 or 14.
Quick question, shall I run HCG for the duration of the cycle and all the way through to end of the PCT or end it at the end of the cycle?
Now.. this is the interesting part for me, I have read some points regarding the use of low dosing of DNP in PCT, I’ll post a good explanation of it below, shamelessly stolen I’ll admit but as far as I can tell the science is bang on?
Please note this is my first cycle of AAS, so feel free to provide advice on the cycle plan as well as my point about DNP
24 years old,
83Kg
13%Bf
Training experience between 2-3 years.
Diet is good, not perfect, but who is?
Have used DNP before and would maybe even run it at 100mg the first week then 200mg the second week of PCT, I don’t want to lose a load of muscle and weight after my cycle, but prevent muscle loss with post cycle thyroid production of t3 etc? OR would this be better done in the two weeks between the last pin and the first day of PCT? (This is better explained in the paragraph below)
THANKS IN ADVANCE!
Post-Steroid Cycle Use of DNP:
One of the primary causes of muscle breakdown after a steroid cycle is suppressed TSH. Anabolic steroids suppress TSH, which in turn lowers T3 and T4 production by the thyroid gland. The reduction in TSH is one reason that anabolic steroids are such excellent muscle builders.
Soon after the completion of a steroid cycle, TSH up-regulates, which in turn super-stimulates the thyroid. This excess stimulation causes the thyroid to produce above normal levels of T3 and T4. This increase in thyroid hormones is highly catabolic and is the main reason why people lose muscle post-cycle.
Athletes have learned that they need to restrict T3 production post cycle to prevent muscle loss. A novel approach to achieving this goal is the use of DNP. About 80% of the body’s endogenous T3 is produced from the metabolically inactive T4 to the metabolically active T3. The de-iodinase enzyme is responsible for this conversion. It literally cleaves off an iodine molecule.
By ingesting 200mg DNP/day, the athlete can correct the over stimulated Thyroid, returning T3 levels back to normal. DNP directly blocks the production of T3 from T4 via the de-iodinase enzyme.
As a bonus, the reduction in your ATP stores because of the DNP is counter acted by an increase in the oxidation of triglycerides as an energy source. The benefit is the elimination of any potential fat-gain from the low post-cycle testosterone levels. And as DNP is non-hormonal, it has no effect on HPTA recovery.
After cessation of DNP use post-cycle, the athlete will reap the benefits of the "Anabolic Rebound Effect" which further lends credence to the use of DNP as a post-cycle ancillary for the elimination of any post-cycle muscular losses
First post here, got an interesting question and hoping some of the members here have some experience with this.
I’m planning on the following at some point in a few months;
Weeks 1-12/14: Test-E 500mg/week
Weeks 1-4/6: Dianabol 40mg/Daily
Weeks 1-12/14: Arimidex 1mg EOD/Daily if required should Gyno be an issue
Weeks 1-12/14 - HCG 500mg/week
PCT - 2 Weeks after last Pin
Clomid 100/100/50/50
Nolva 40/40/20/20
Note: I have put Weeks 1-12/14 and Weeks 1-4/6 because the length of the cycle may be either 12 or 14 Weeks. I have enough gear for 14 weeks and will probably decide during cycle to run 12 or 14.
Quick question, shall I run HCG for the duration of the cycle and all the way through to end of the PCT or end it at the end of the cycle?
Now.. this is the interesting part for me, I have read some points regarding the use of low dosing of DNP in PCT, I’ll post a good explanation of it below, shamelessly stolen I’ll admit but as far as I can tell the science is bang on?
Please note this is my first cycle of AAS, so feel free to provide advice on the cycle plan as well as my point about DNP
24 years old,
83Kg
13%Bf
Training experience between 2-3 years.
Diet is good, not perfect, but who is?
Have used DNP before and would maybe even run it at 100mg the first week then 200mg the second week of PCT, I don’t want to lose a load of muscle and weight after my cycle, but prevent muscle loss with post cycle thyroid production of t3 etc? OR would this be better done in the two weeks between the last pin and the first day of PCT? (This is better explained in the paragraph below)
THANKS IN ADVANCE!
Post-Steroid Cycle Use of DNP:
One of the primary causes of muscle breakdown after a steroid cycle is suppressed TSH. Anabolic steroids suppress TSH, which in turn lowers T3 and T4 production by the thyroid gland. The reduction in TSH is one reason that anabolic steroids are such excellent muscle builders.
Soon after the completion of a steroid cycle, TSH up-regulates, which in turn super-stimulates the thyroid. This excess stimulation causes the thyroid to produce above normal levels of T3 and T4. This increase in thyroid hormones is highly catabolic and is the main reason why people lose muscle post-cycle.
Athletes have learned that they need to restrict T3 production post cycle to prevent muscle loss. A novel approach to achieving this goal is the use of DNP. About 80% of the body’s endogenous T3 is produced from the metabolically inactive T4 to the metabolically active T3. The de-iodinase enzyme is responsible for this conversion. It literally cleaves off an iodine molecule.
By ingesting 200mg DNP/day, the athlete can correct the over stimulated Thyroid, returning T3 levels back to normal. DNP directly blocks the production of T3 from T4 via the de-iodinase enzyme.
As a bonus, the reduction in your ATP stores because of the DNP is counter acted by an increase in the oxidation of triglycerides as an energy source. The benefit is the elimination of any potential fat-gain from the low post-cycle testosterone levels. And as DNP is non-hormonal, it has no effect on HPTA recovery.
After cessation of DNP use post-cycle, the athlete will reap the benefits of the "Anabolic Rebound Effect" which further lends credence to the use of DNP as a post-cycle ancillary for the elimination of any post-cycle muscular losses
