What Causes Abdominal Distension in Anabolic Steroid and HGH Users? "Palumboism"

[Silentlemon1011]

Welp
Looks like I'm fucking off work for another 17 minutes lol

 

[MuscleFreak]

Welp
Looks like I'm fucking off work for another 17 minutes lol

haha have fun bro,drink some coffee and see what u think about metformin
btw i know Luke sandoe liked it a lot and used it for years

 

[Mac11wildcat]

When you consider all this stuff you have to consider to what degrees and how.

metformin might impact mtor, but without insulin sensitivity you can’t get nutrients to muscles effectively either.

Mtor is also upregulated by testosterone too IIRC. The primary benefit of metformin is the slin sensitivity. But the other positives probably stem from the autophagy that’s mentioned. So it’s give and take. I think for muscle building, AAS + metformin is mostly take.

 

[FR0Z3N_B0MB3RRR]

When you consider all this stuff you have to consider to what degrees and how.

metformin might impact mtor, but without insulin sensitivity you can’t get nutrients to muscles effectively either.

Mtor is also upregulated by testosterone too IIRC. The primary benefit of metformin is the slin sensitivity. But the other positives probably stem from the autophagy that’s mentioned. So it’s give and take. I think for muscle building, AAS + metformin is mostly take.

you re always on Metformin so? I read about liver enzyme issues so i planned to use it after some weeks on insulin

 

[MuscleFreak]

When you consider all this stuff you have to consider to what degrees and how.

metformin might impact mtor, but without insulin sensitivity you can’t get nutrients to muscles effectively either.

Mtor is also upregulated by testosterone too IIRC. The primary benefit of metformin is the slin sensitivity. But the other positives probably stem from the autophagy that’s mentioned. So it’s give and take. I think for muscle building, AAS + metformin is mostly take.

how was your experience with metformin? does it help u stay leaner in off season?

 

[Silentlemon1011]

When you consider all this stuff you have to consider to what degrees and how.

metformin might impact mtor, but without insulin sensitivity you can’t get nutrients to muscles effectively either.

Mtor is also upregulated by testosterone too IIRC. The primary benefit of metformin is the slin sensitivity. But the other positives probably stem from the autophagy that’s mentioned. So it’s give and take. I think for muscle building, AAS + metformin is mostly take.

Thanks bro

 

[Mac11wildcat]

you re always on Metformin so? I read about liver enzyme issues so i planned to use it after some weeks on insulin

I just got bloods back and my liver and kidneys are perfect. Best they’ve ever been. Was on met the whole time.

how was your experience with metformin? does it help u stay leaner in off season?

The effect isn’t as pronounced as say, gH, but it’s there for sure. I will say met can produce a pump better than if you were to not take it. After the first couple weeks of gastro distress that’s normal, things smoothed out and I think considering how cheap it is international (and even cheaper with a script) that it’s a good addition. The health benefits outside of bodybuilding seem to be decent as well.

 

[FR0Z3N_B0MB3RRR]

So youre on met all year long? At 500mg 2 times daily? I Take sustained version By the way

 

[MuscleFreak]

I just got bloods back and my liver and kidneys are perfect. Best they’ve ever been. Was on met the whole time.



The effect isn’t as pronounced as say, gH, but it’s there for sure. I will say met can produce a pump better than if you were to not take it. After the first couple weeks of gastro distress that’s normal, things smoothed out and I think considering how cheap it is international (and even cheaper with a script) that it’s a good addition. The health benefits outside of bodybuilding seem to be decent as well.

tnx for reply brother! i have some metformin so maybe i will give it a try.

 

[Mac11wildcat]

So youre on met all year long? At 500mg 2 times daily? I Take sustained version By the way

I’ve used it up to 2g when I was concerned I had insulin resistance from poor gH and slin protocols. Right now I take 1000mg 1x at night on most says, 500 AM 1000PM on high days (high carbs and higher gH dosage + slin use)

I’d like to reduce it overall to 500-1000 but won’t do so until I see if my gH/slin protocol I’m using currently is doing well.

ive used xR and normal. The regular release is def a much better “pump” option

 

[Mayne]

I have read that Leucine would handle Metformin mTOR inhibition

 

[Mac11wildcat]

I have read that Leucine would handle Metformin mTOR inhibition

eat your god damn protein

 

[The Terminator]

It is quite common the repulsive look of the late IFBB pros, showing protrusion of their abdominal area.

There are several mechanisms that cause this phenomenon and its multifactorial.

1) It is known that HGH via somatomedin (IGF-1) causes hypertrophy/hyperplasia of skeletal muscles and rest of the tissues (lips, ears, tongue, gums).This actually takes place under high doses of somatropin (>8IU/day). Viscera (stomach, heart,lungs,liver,small and large intestines, kidneys) have a large number of receptors IGF-1, which will lead to them inflating. This effect is not immediate, it depends on the dose and time. Therefore the abuse ofr-HGH for an extended period of time creates the extrusion of the abdomen. In other words, organomegaly occur, the enlargement of each organ as a whole.

2) Accumulation of visceral fat occurs between the organs of the abdomen and the omentum. Greater and lesser omentumare layers of peritoneum surrounding the viscera and their vessels, nerves. Omentum creates bends and sinuses between the abdominal organs (peritoneal cavity).

The accumulation of fat happens mainly because of the lipogenesis (fat storage) induced by the use of insulin that is administrated synergistically with the HGH. Both act synergistically and the regulate glucose metabolism. Since somatropin raises serum glucose (through gluconeogenesis in liver) and insulin lowers serum glucose (and activate glycogen synthase enzyme in liver), they apparently have opposite effects on the metabolism of carbohydrates. Therefore, HGH causes hyperglycemia and the release of hepatic glycogen in blood.

On the other hand, insulin leads to hypoglycemia and enhances formation of liver and muscle glycogen. Insulin resistance is the decrease in body’s ability to respond properly to the metabolism of insulin and glucose, leading to a vicious circle of hyperglycemia, hyperinsulinemia and eventually the establishment of DM2 (non-insulin dependent).

As it is known, the chronic use of r-HGH decreases insulin sensitivity, resulting in a growing need of insulin that eventually will exceed the body’s ability to properly regulate it. On that case, drugs reducing insulin resistance are required (metformin). Alternatively berberine, Chromium picolinate, alpha lipoic acid and vanadyl sulphate are among the supplements that improve insulin sensitivity. While the lipolytic action of growth hormone is generally strong enough to counteract the negative effects of insulin resistance in the fatty subcutaneous cells, it does not have the same positive effect on the visceral fat depots.

The extent, to which the combination of HGH and insulin will lead to abdominal distension, differs from person to person. Factors such as the type of diet (trans fat, refined sugars create insulin spikes), cardiovascular exercise, the frequency of weight lifting, the type of insulin used and the supplements can affect the sensitivity and therefore the likelihood of developing visceral fat.

3) Exogenous insulin interferes with the electrolyte balance increasing the retention of water and sodium, therefore leading to edema and swelling of tissues. This water retention occurs not only in the subcutaneous, but on the whole body as well, which is why many people hold water in the abdomen after the injection, especially with the regular use of insulin.

4) The consumption of large portions of food is another factor of stomach distention. Therefore, the larger amounts of food consumed, the more difficult the digestion is, resulting in the distention of the intestine.

5) The chronic use of anabolic steroids (AAS) in tablet form (17 alkylated) lowers and modifies the operation of the normal intestinal flora. This change disrupts the normal bowel function and causes constipation, overproduction of gas and distension of the intestine. The use of lactobacillus acidophilus probiotic bacteria as a supplement, or the consumption of kefir and organic yogurt could be of great value. Moreover candida albicans is useful as well .

6) Digestive disorders such gluten sensitivity and lactose intolerance thatlead to the overproduction of gas in the intestine. Therefore dairy products and wheat should be avoided.

7) The excessive intake of red meat and the inadequate fiber intake burdens the colon, which inflames because of the toxins. Gus based on nitrogen is produced, giving the feeling of bloated bowel. In order to facilitate the digestion and assimilation of the gastrointestinal tract, it is necessary to consume vegetables and fruits that are rich in fiber , in order to facilitate intestinal peristalsis, thus preventing constipation and bowel distension.

8) The weakening of the transverse abdominal muscle plays also an essential role. Transverse abdominal muscle forms a tube like cavity,that ori ginates from the lumbar fascia, the last six ribs, the iliac crest, the inguinal ligament ;while it ends in linea alba with its denervation. It is located beneath the external oblique and rectus abdominis muscles. The action of the transverse abdominis helps to control the body’s stability, the cornering and bending of the trunk, the support of the abdominal wall and the production of intra-abdominal pressure. The transverse abdominal muscle twitches before the other muscles of the body do, and creates the base of the body movement. Its contraction creates a rigid cylinder because of its location and its crown shape mobilizes the thoracolumbar fascia; thus creating the conditions for the increase of the intra-abdominal pressure. It acts basically like a belt around your waist, holding the truck. Relaxation of the transverse abdominis could be the result of the technique of many exercises with a large increase of intra-abdominal pressure (squats, deadlifts, leg press, and lunges), leading to abdominal distension.

It seems necessary and obligatory to drop on weight in order for the tissues to diminish. This is something accomplished by fasting and caloric deficit, following smaller meals and minimizing insulin spikes. There are top athletes who managed to reverse this and have a more aesthetic appearance on stage. Bubble gut can ruin the V taper of the physique and particularly the front three poses (relax,lats spread,double biceps) that reveal it.

Performing respiratory exercises of vacuum, by sucking the air through diaphragmatic breathing, can control the midsection during relaxing condition. It would be wiser to perform abdominal exercises focusing on the rectus abdominis, rather than hitting the external obliques. In this way we minimize distention of waist circumference.

Mr.Olympians known for their excellent V taper were Sergio Oliva and Lee Haney.

References:

1. Multiple Effects of Growth Hormone in the Body: Is it Really the Hormone for Growth? Jesús Devesa, Cristina Almengló, and Pablo Devesa. Clin Med Insights Endocrinol Diabetes. 2016; 9: 47–71.
2. Body Fat Distribution and Insulin Resistance. Pavankumar Patel*and Nicola Abate. Nutrients. 2013 Jun; 5(6): 2019–2027.
3. The effect of 30 months of low-dose replacement therapy with recombinant human growth hormone (rhGH) on insulin and C-peptide kinetics, insulin secretion, insulin sensitivity, glucose effectiveness, and body composition in GH-deficient adults. Rosenfalck AM, Maghsoudi S, Fisker S, Jørgensen JO, Christiansen JS, Hilsted J, Vølund AA, Madsbad S.J ClinEndocrinolMetab.2000 Nov;85(11):4173-81.
4. The treatment of functional abdominal bloating and distension. Schmulson, L. Chang. Aliment PharmacolTher2011; 33: 1071–1086
5. Impacts of Gut Bacteria on Human Health and Diseases. Yu-Jie Zhang,Sha Li ,Ren-You Gan ,Tong Zhou ,Dong-Ping Xu andHua-Bin Li . J. Mol. Sci. 2015, 16(4), 7493-7519
6. Effect of craniocervical posture on abdominal muscle activities Jung Gil Su, Shin Ji Won, and HwangboGak.J Phys Ther Sci. 2016 Feb; 28(2): 654–657.

I’d say it’s most likely visceral fat from all the drugs, especially hgh and/or insulin. I’ve read a study that mentioned the possibility of roids causing this problem. It mentioned something about current roid users had more visceral fat than former and non users, but unless today’s doses are more massive I’d say it’s most likely gh/insulin causing it since old school bb’s didn’t have these guts. If those bubble guts were from backed up shat they’d have more immediate problems.

 

[Mac11wildcat]

Guess we just need to take more Anavar

 

[MuscleFreak]

Guess we just need to take more Anavar

100mg +

 

[The Terminator]

Guess we just need to take more Anavar

Go for it, but I was referring to injectable roids possibly contributing to this but the real issue being the hgh and insulin.

 

[Mac11wildcat]

Go for it, but I was referring to injectable roids possibly contributing to this but the real issue being the hgh and insulin.

I was referring to anavar eating up visceral fat

 

[The Terminator]

I was referring to anavar eating up visceral fat

Wasn’t sure if that was a response to my post, either way I just wanted to clarify I was referring to injectables not orals.

 

[Joe Morgan]

 

[master.on]

I wonder if it has to do with BBers eating more and more as they have fat burning drugs that weren't available in the 70s/80s.

I read that 70s/80s BBers did 1000-1500 calorie diets for contest prep!