Which of these is causing hair shedding?

[azx77p2p]

Finished a 12 week ostarine cycle (25mg) which is known to decimate SHBG - as expected mine was 7nmol 3 days after cycle ended.
LH and FSH were normal though with test being low.

I used nolva for 7 days at 10mg and thats when I noticed the shedding. I've stopped it but still notice 1-2 hairs every time I shower falling out.

My question is what is causing this? My theory was SHBG is low, and the test bouncing back is free and goes DHT crazy?

 

[muscletrain]

Could be super lower SHBG allowing for unbound free test. Besides that it, it's avery mild cycle.

Goodluck on the chrome dome in the future when you touch the real steroids.

 

[Cashton]

Finished a 12 week ostarine cycle (25mg) which is known to decimate SHBG - as expected mine was 7nmol 3 days after cycle ended.
LH and FSH were normal though with test being low.

I used nolva for 7 days at 10mg and thats when I noticed the shedding. I've stopped it but still notice 1-2 hairs every time I shower falling out.

My question is what is causing this? My theory was SHBG is low, and the test bouncing back is free and goes DHT crazy?

1-2 hairs is nothing, google how many actually fall out a day.

 

[Manbearpig2]

1-2 hairs is nothing, google how many actually fall out a day.

Exactly. It’s normal for hairs to fall out, you’re a human with millions of hair follicles, once you start having handfuls of hair then it’s a problem.

 

[azx77p2p]

Hmm, i guess its just alarming because its noticable hairs shedding for the first time in many years. Perhaps 1-2 was underestimation, in 1 day its likely 3-5 visibly large hairs if I run fingers through hair or when showering

Does not seem normal. But i do have a blood test pending and its been 1 month since stopping the cycle so I will see how the SHBG and test levels look.

 

[Cashton]

Hmm, i guess its just alarming because its noticable hairs shedding for the first time in many years. Perhaps 1-2 was underestimation, in 1 day its likely 3-5 visibly large hairs if I run fingers through hair or when showering

Does not seem normal. But i do have a blood test pending and its been 1 month since stopping the cycle so I will see how the SHBG and test levels look.

You likely just haven’t noticed your whole life that you lose a couple hairs a day, now that you’re on anabolics and that the hair shedding is on your mind, you notice the hears and think “oh shit, they’re falling out” but it’s likely no more than you would be losing regularly anyways, unless you’re extremely prone to hair loss. Only a percentage of people actually have the problem with MPB and baldness.

 

[GuerillaPete]

I heard a video i think it was MPMD and what he said was shocking .
He basically said once you notice it in the mirror that you have less hair it means that you lost half already .
Scary shit….

 

[Cashton]

I heard a video i think it was MPMD and what he said was shocking .
He basically said once you notice it in the mirror that you have less hair it means that you lost half already .
Scary shit….

Hair thinning is to be expected a little, mine is thinned, but it’s not falling out in any way currently.

 

[lifter6973]

Hair thinning is to be expected a little, mine is thinned, but it’s not falling out in any way currently.

That is called denial

 

[GuerillaPete]

That is called denial

Cope

 

[conqueror]

LOL. All the hair I lost was off the gear. Literally all of it. The culprit is your age.

 

[conqueror]

Finished a 12 week ostarine cycle (25mg) which is known to decimate SHBG - as expected mine was 7nmol 3 days after cycle ended.
LH and FSH were normal though with test being low.

I used nolva for 7 days at 10mg and thats when I noticed the shedding. I've stopped it but still notice 1-2 hairs every time I shower falling out.

My question is what is causing this? My theory was SHBG is low, and the test bouncing back is free and goes DHT crazy?

You’re reasoning is... incorrect I’m afraid. Just the way ostarine is expected to reduce SHBG secretion acting as an Androgen Receptor (AR) ligand via direct passage through the liver, tamoxifen will promote the opposite effect as an Estrogen Receptor (ER) ligand. Yes. Tamoxifen is actually increasing your SHBG via direct passage through the liver. It is a Selective Estrogen Receptor Modulator (SERM), not a strict Estrogen Receptor (ER) antagonist; meaning it activates the estrogen receptor on select tissue (e.g. liver) while inhibiting endogenous ligand binding elsewhere (e.g. breast tissue). Ostarine was supposed to act like that for the AR, but I never got the impression it actually did judging from the studies. It’s increased Anabolic-Androgenic differentiation, if any, over testosterone may simply be due to the fact it binds with less affinity than dihydrotestosterone where 5-alpha-reduction is bound to occur just like most other AAS other than trenbolone (and a few other 19-Nor-T(estosterone) derivatives).

 

[conqueror]

So... my tentative (serious) answer is you’re experiencing a delayed response to ostarine acting on your scalp?

 

[azx77p2p]

So... my tentative (serious) answer is you’re experiencing a delayed response to ostarine acting on your scalp?

In that case I will incorporate fin or dut into my keto shampoo regime and be wary of hair loss for future cycles. Since it seems even mild ostarine fucks my shit up :/

 

[Cashton]

That is called denial

Nah, I’ll post a pic, my hair became less full over a year or two, but hasn’t receded or become patchy.. no change except for the thickness, looks the same as it did when I was 16, or 18 or 20whatever, I’m 26 now and running gear for a while and it’s exactly the same, just less poofy.

 

[Cashton]

In that case I will incorporate fin or dut into my keto shampoo regime and be wary of hair loss for future cycles. Since it seems even mild ostarine fucks my shit up :/

That’s all I use is that anti dandruff shampoo, nizoral.

 

[conqueror]

In that case I will incorporate fin or dut into my keto shampoo regime and be wary of hair loss for future cycles. Since it seems even mild ostarine fucks my shit up :/

I wouldn’t do that, if I was you. I’ll rather be bald than experience erectile dysfunction, but that is just me.

 

[Cashton]

You’re reasoning is... incorrect I’m afraid. Just the way ostarine is expected to reduce SHBG secretion acting as an Androgen Receptor (AR) ligand via direct passage through the liver, tamoxifen will promote the opposite effect as an Estrogen Receptor (ER) ligand. Yes. Tamoxifen is actually increasing your SHBG via direct passage through the liver. It is a Selective Estrogen Receptor Modulator (SERM), not a strict Estrogen Receptor (ER) antagonist; meaning it activates the estrogen receptor on select tissue (e.g. liver) while inhibiting endogenous ligand binding elsewhere (e.g. breast tissue). Ostarine was supposed to act like that for the AR, but I never got the impression it actually did judging from the studies. It’s increased Anabolic-Androgenic differentiation, if any, over testosterone may simply be due to the fact it binds with less affinity than dihydrotestosterone where 5-alpha-reduction is bound to occur just like most other AAS other than trenbolone (and a few other 19-Nor-T(estosterone) derivatives).

Do you know how it is than that tren causes hair loss? If it’s not 5-ar, what pathway? Or does tren just “not affect hair”

 

[Cashton]

I wouldn’t do that, if I was you. I’ll rather be bald than experience erectile dysfunction, but that is just me.

I wouldn’t personally run fin or dua without having a serious reason (ie. prostate) out of those fears also but I think it’s only a fairly low percent of people who actually get those sides, most people are probably fine.

 

[conqueror]

Do you know how it is than that tren causes hair loss? If it’s not 5-ar, what pathway? Or does tren just “not affect hair”

Any sufficient amount of AAS will precipitate hair loss, if the subject is genetically predisposed for it. This will occur via normal activation of the AR. Despite the fact Trenbolone bypasses 5a-reductase potentiation unlike testosterone, it still shares about same binding affinity as DHT (product of said metabolic pathway). If the manifestation of androgenic side effects is so undesirable that you don’t mind even experiencing erectile dysfunction, no compound (that I know of) compares to nandrolone (other than just plain castration); it transforms into an inert by-product via 5a-reductase metabolism (in polar opposition to testosterone).