Please stop saying Anavar is a mild drug…

clearly not hepatotoxic, a 10 year badge in here reports better hdl better ldl on it! i will run it 200mg daily as my statin and you should too!
Biologically speaking its metabolized via the liver so considering it’s oxidized and reduced in the liver im guessing this reply is ragebait
 
I've been taking var for 23 days now with 300mg test cypionate. Started the Var at 40mg week one and it dried me out too much and everything was clicking and cracking. It also flattened me out too much.
The var is definitely not Winstol because I get a very big libido increase from Winstrol and a lot of a CNS stimulating effect like im drinking coffee all day.
I've had zero libido increase from the Var and it's been completely CNS neutral. In fact it has no subjective feel or side effects at all, which is excellent - I don't want to be driven to distraction with a crazy high libido for example.
I've been taking it at 20mg per day for 16 days and it's given a substantial grainy look to all my muscles, such that it's a bit freaky - in a good way. 20mg has me with nice fullness from the test also and not flattened out.
I'll get bloods done in a couple weeks and post them up.
 
The box insert says the daily dose can be up to 20mg per day for 2-4 weeks. It also states a contradiction for use in patients with renal disease. I think from that we can extrapolate that kidneys can be impacted, and because we are using it at higher doses and for longer periods of time. the impact on kidneys can be greater. The box insert also says it can cause liver and spleen problems, again, at 20mg a day for up to 4 weeks. Using more and for longer most likely increases the possibility of that.

Now pull some tylenol, aspirin and ibubrofen box insert and post it.

Then STFU and GTFO!
 
bro my ldl is 30 without drugs. I can eat pig lard or take orals and be under 40 ldl, I personally don't care. My dad literally eats sticks of butter and has a 60 LDL. Genetics.
And THIS.

You can watch your diet like a hawk, do a ton of cardio, never touch orals and have a heart attack in your 40s, while some lucky sob with the right genes can eat lard, do 0 cardio, smoke and drink and have a calcium score of ZERO.

Blame your shitty genes, not the drugs
 
It's overhyped. If you're sub10-11ish, you might see some. The mythos has grown it to the point where someone with 20+% bf will magically lose visceral fat.
I mean, it's not like your body just doesn't lose visceral fat until you're running low on subcutaneous fat. I lost more proportional visceral fat than subcutaneous fat when dropping weight in the 20%+ BF range per dexa.

Which isn't to say people should hop on anavar for visceral fat loss - GLP1 drugs exist and are better suited for this - but your point doesn't make much sense. Particularly when the studies on visceral fat loss with anavar have been on fat people.

I suspect it will do even less for you visceral fat if you're already in relatively good body composition.
Only an idiot would get on a statin for 0 reason. LDL count alone isn’t a good reason…
That is literally the entire reason to get on a statin. What are you talking about?
 
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It's overhyped. If you're sub10-11ish, you might see some. The mythos has grown it to the point where someone with 20+% bf will magically lose visceral fat.
Aren't you still fat, if you loose primarily visceral fat but are still 20+% BF?

Now pull some tylenol, aspirin and ibubrofen box insert and post it.

Then STFU and GTFO!
I am not sure if that is such a great example, of how meaningless the insert of Anavar actually is. Excluding maybe Aspirine, those are drugs one should really not take for any prolonged period of time.

That is literally the entire reason to get on a statin. What are you talking about?
The remark about statins had me a bit confused as well.
 
Greatest predictor(s) of CVE is Apob and LP(a)
Well, lowering LDL will lower ApoB

I suspect some of the confusion comes from LDL being the primary treatment target - most statins will slightly increase Lp(a) even, but still improve cvd outcomes.

PCSK9 inhibitors do lower Lp(a) a bit and there's a variety of Lp(a) targeting drugs in trials right now that show promise in very large Lp(a) reduction

It'll be possible this decade to crush both ApoB and Lp(a) levels from an "approved pharmacology" standpoint. Not sure if we'll see any of them available from china or if we'll need prescriptions and expensive shit. siRNA is less challenging than monoclonal antibodies to produce, but still a big step up from peptides. If muvalaplin pans out it's probably the most likely option - it's a fairly standard small molecule drug.
 
When it comes to the positive effects of AAS (strength, size, etc) we've seen that genetics play a very large role in the outcome. Some blow up on low doses while others can blast grams and still look natty. It's not unreasonable to assume that the negative effects also work in this way. Some can run toxic compounds and be completely fine, or recover quickly, while others get their organs ruined from low doses of so called "mild" compounds like Anavar. I'm one of those people sadly, if a drug can cause liver/kidney damage it absolutely will. I break out with forearm acne within days of implementing 20mg anavar PWO. A drug can be mild for some and harsh for others. Its not really that complicated and no point arguing whether a drug is harsh or not cause there will always be someone who was 1shot by it.
 
Laugh Celebrity GIF


Well this developed into quite the page-turner (and occasional shit-flinger). Nice work, @RockyP
 
Laugh Celebrity GIF


Well this developed into quite the page-turner (and occasional shit-flinger). Nice work, @RockyP
Let’s recap

1. Regardless of what you think, you’re wrong. Stop breathing all my oxygen

2. Under no circumstances are medications for off label use

3. Anavar is the devil. Shoot grams of Tren weekly pu$$y.

4. Most importantly @readalot is back bumping old tuna fish threads.
 
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