For anyone else, legitimately interested in how I was able to infer that OT suppresses LH & FSH minimally, to the tune of 10% at 20 mg/d, see:
Dorner, G., Stahl, F., & Zabel, R. (1963). Vergleichende Untersuchungen über den Stoffwechsel von 4-Chlortestosteron- und Testosteronverbindungen bei Menschen. Endokrinologie. 45: 121-128.
Dorner, G. (1965). 4-chloro-Δ¹-methyltestosterone (Oralturinabol), a new orally effective anabolic agent. Dtsch. Gesundheitwes. 20: 670-674.
Holma P, Adlercreutz H. (1976). Effect of an anabolic steroid (metandienon) on plasma LH-FSH, and testosterone and on the response to intravenous administration of LRH. Acta Endocrinol (Copenh). Dec;83(4):856-64.
Based on those data, we can say that due to the effects of 4-substitution on the activity of OT it exerts very minimal effects on LH & FSH.
Since OT has only 20% of the gonadotrophic effect of Dbol (whose effects on LH, FSH at 15 mg daily have been shown to decrease LH & FSH ~50%; therefore it follows that OT only decreases LH & FSH by about ~10% [reflecting a minimal decrement in HPG axis functioning/"HPT axis suppression"] at a dose of 20 mg daily).