MESO-Rx
Anabolic Steroids
One more thing
- Thread starter sade
- Start date
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If you're like me. It kills your fucking dick and doctors think youre normal because you have normal T levels but they didn't find it unusual that your LH is elevated!! I swear to god, I've been through hell and back just to find out what the fuck was going wrong with me. I knew from the start that I needed higher levels of testosterone to feel like myself again. The UK is decades behind on testosterone issues, I'm lucky that there is a specialist over here who is very knowledgable on my condition. I got that diagnosis but am still waiting for treatment!! I swear the fucking NHS stinks of shit. I've already contacted a TRT clinic and have an appointment to get TRT prescribed but I'll have to pay for it. And the fucking bloods, prostate examinations. If I got to the clinic I could choose my own T which would be Testosterone Enanthate without a doubt. If I stick with the NHS I'll be put on Nebido. I'm unsure what to do. I have such a rare condition which doctors are unaware of. There are guys older than me with low T who are struggling to get treatment. and here I am 31 years old with a very rare condition which 99% of doctors and physicians are oblivious too.
hardasnails1973
New Member
[ame=http://www.youtube.com/watch?v=p7CaiWxKYBo]ROCKY TRIBUTE -- EYE OF THE TIGER - YouTube[/ame]
ROCKY kicks his ass........
hardasnails1973
New Member
Again mis information..
Steroids makes your pecker bigger due to increase blood flow., but may make ball smaller giving optically illusion that it grows even bigger.
[
)] Dat is indeed a classic.This is a better mix to the original I posted!!!!!!!
Bloodsport - Fight to survive by Stan Bush - YouTube!
putting the issue to rest
I'll do it. And I don't want dr scally with his shitty picture things and his words telling me that T isn't my fucking problem when he is not me and doesn't know me or my body and doesn't know what level of T will make me or break me..
I don't want or need his shitty picture posts responding to this thread.
I'll do it. And I don't want dr scally with his shitty picture things and his words telling me that T isn't my fucking problem when he is not me and doesn't know me or my body and doesn't know what level of T will make me or break me..
I don't want or need his shitty picture posts responding to this thread.
Re: putting the issue to rest
It must be the weekend
It must be the weekend
Re: putting the issue to rest
Minimum criteria required for testing to be appropriate:
Criteria (all required)
1. 46, XY karyotype AND
2. normal testosterone production AND
3. absent mullerian structures AND
4. female appearance or ambiguous genitalia with no other syndromic features
Minimum criteria required for testing to be appropriate:
Criteria (all required)
1. 46, XY karyotype AND
2. normal testosterone production AND
3. absent mullerian structures AND
4. female appearance or ambiguous genitalia with no other syndromic features
Re: putting the issue to rest
I have no idea what mullerian structues are but my genitalia is normal. Normal sized penis and testicles and 100% male appearance. My build is skinny though. I was never able to put on any muscle even after abusing steroids for years.
I have no idea what mullerian structues are but my genitalia is normal. Normal sized penis and testicles and 100% male appearance. My build is skinny though. I was never able to put on any muscle even after abusing steroids for years.
Re: putting the issue to rest
google is your friend
http://biology.westfield.ma.edu/Biol104w/class-11 (Class 11: Endocrine and Reproductive Systems | Human Biology Online: Winter Session 2012)
I think the good doctor just saved you the cost of this labwork.
google is your friend
http://biology.westfield.ma.edu/Biol104w/class-11 (Class 11: Endocrine and Reproductive Systems | Human Biology Online: Winter Session 2012)
I think the good doctor just saved you the cost of this labwork.
Re: putting the issue to rest
[ame=http://en.wikipedia.org/wiki/Mild_androgen_insensitivity_syndrome]Mild androgen insensitivity syndrome - Wikipedia, the free encyclopedia[/ame]
Mild androgen insensitivity syndrome (MAIS) is a condition that results in a mild impairment of the cell's ability to respond to androgens[1][2][3]. The degree of impairment is sufficient to impair spermatogenesis and / or the development of secondary sexual characteristics at puberty in males, but does not affect genital differentiation or development. Female genital and sexual development is not significantly affected by the insensitivity to androgens[3][4]; as such, MAIS is only diagnosed in males[1]. The clinical phenotype associated with MAIS is a normal male habitus with mild spermatogenic defect and / or reduced secondary terminal hair[1][5][6][7][8][9].
MAIS is one of three types of androgen insensitivity syndrome, which is divided into three categories that are differentiated by the degree of genital masculinization: complete androgen insensitivity syndrome (CAIS) is indicated when the external genitalia is that of a normal female, mild androgen insensitivity syndrome (MAIS) is indicated when the external genitalia is that of a normal male, and partial androgen insensitivity syndrome (PAIS) is indicated when the external genitalia is partially, but not fully masculinized [1][2][5][6][7][10][11][12][13].
Androgen insensitivity syndrome is the largest single entity that leads to 46,XY undermasculinization [1
MAIS is only diagnosed in normal phenotypic males, and is not typically investigated except in cases of male infertility [18]. MAIS has a mild presentation that often goes unnoticed and untreated [15]; even with semenological, clinical and laboratory data, it can be difficult to distinguish between men with and without MAIS, and thus a diagnosis of MAIS is not usually made without confirmation of an AR gene mutation [5]. The androgen sensitivity index (ASI), defined as the product of luteinizing hormone (LH) and testosterone (T), is frequently raised in individuals with all forms of AIS, including MAIS, although many individuals with MAIS have an ASI in the normal range [5]. Testosterone levels may be elevated despite normal levels of luteinizing hormone [15][20][24]. Conversion of testosterone (T) to dihydrotestosterone (DHT) may be impaired, although to a lesser extent than is seen in 5?-reductase deficiency [3]. A high ASI in a normal phenotypic male [45], especially when combined with azoospermia or oligospermia [5][7], decreased secondary terminal hair [26], and / or impaired conversion of T to DHT [3], can be indicative of MAIS, and may warrant genetic testing.
[ame=http://en.wikipedia.org/wiki/Mild_androgen_insensitivity_syndrome]Mild androgen insensitivity syndrome - Wikipedia, the free encyclopedia[/ame]
Mild androgen insensitivity syndrome (MAIS) is a condition that results in a mild impairment of the cell's ability to respond to androgens[1][2][3]. The degree of impairment is sufficient to impair spermatogenesis and / or the development of secondary sexual characteristics at puberty in males, but does not affect genital differentiation or development. Female genital and sexual development is not significantly affected by the insensitivity to androgens[3][4]; as such, MAIS is only diagnosed in males[1]. The clinical phenotype associated with MAIS is a normal male habitus with mild spermatogenic defect and / or reduced secondary terminal hair[1][5][6][7][8][9].
MAIS is one of three types of androgen insensitivity syndrome, which is divided into three categories that are differentiated by the degree of genital masculinization: complete androgen insensitivity syndrome (CAIS) is indicated when the external genitalia is that of a normal female, mild androgen insensitivity syndrome (MAIS) is indicated when the external genitalia is that of a normal male, and partial androgen insensitivity syndrome (PAIS) is indicated when the external genitalia is partially, but not fully masculinized [1][2][5][6][7][10][11][12][13].
Androgen insensitivity syndrome is the largest single entity that leads to 46,XY undermasculinization [1
MAIS is only diagnosed in normal phenotypic males, and is not typically investigated except in cases of male infertility [18]. MAIS has a mild presentation that often goes unnoticed and untreated [15]; even with semenological, clinical and laboratory data, it can be difficult to distinguish between men with and without MAIS, and thus a diagnosis of MAIS is not usually made without confirmation of an AR gene mutation [5]. The androgen sensitivity index (ASI), defined as the product of luteinizing hormone (LH) and testosterone (T), is frequently raised in individuals with all forms of AIS, including MAIS, although many individuals with MAIS have an ASI in the normal range [5]. Testosterone levels may be elevated despite normal levels of luteinizing hormone [15][20][24]. Conversion of testosterone (T) to dihydrotestosterone (DHT) may be impaired, although to a lesser extent than is seen in 5?-reductase deficiency [3]. A high ASI in a normal phenotypic male [45], especially when combined with azoospermia or oligospermia [5][7], decreased secondary terminal hair [26], and / or impaired conversion of T to DHT [3], can be indicative of MAIS, and may warrant genetic testing.
My last doctor said:
My last doctor said:
Re: putting the issue to rest
No disrespect, but I thought the info on Wikki was highly inaccurate, due to the fact that anyone can post and edit.
No disrespect, but I thought the info on Wikki was highly inaccurate, due to the fact that anyone can post and edit.
Re: putting the issue to rest
Yeah but my doctors notes aren't inaccurate. Read that last part of my post!
Yeah but my doctors notes aren't inaccurate. Read that last part of my post!
Re: putting the issue to rest
The info on this Wikipedia article is thoroughly sourced. In fact, just about every sentence has one or more citations to articles in medical journals. I've done plenty of research on AIS, and I can say that the article is solid (I've followed many a lead through medical journals from this article's citations).
The hospital that Sade would be going through for AIS testing does not ordinarily screen for MAIS, just CAIS and PAIS, as these are by far more common (and more concerning). This is why the criteria is focused on ambiguous / female genitalia. However, the hospital is obviously aware that some men with MAIS need testing, which is why at the end of the pdf they state "If the sample does not fulfil all the inclusion criteria and you still feel that testing should be performed please contact the molecular genetics laboratory"
So it's really this simple: if Sade's endocrinologist signs off on the paper, he'll get the test.
It is worth noting that men with MAIS do not have ambiguous genitalia, nor do they have Müllerian structures. Without this exception clause in the PDF, the hospital could not offer testing to any man with MAIS.
The info on this Wikipedia article is thoroughly sourced. In fact, just about every sentence has one or more citations to articles in medical journals. I've done plenty of research on AIS, and I can say that the article is solid (I've followed many a lead through medical journals from this article's citations).
The hospital that Sade would be going through for AIS testing does not ordinarily screen for MAIS, just CAIS and PAIS, as these are by far more common (and more concerning). This is why the criteria is focused on ambiguous / female genitalia. However, the hospital is obviously aware that some men with MAIS need testing, which is why at the end of the pdf they state "If the sample does not fulfil all the inclusion criteria and you still feel that testing should be performed please contact the molecular genetics laboratory"
So it's really this simple: if Sade's endocrinologist signs off on the paper, he'll get the test.
It is worth noting that men with MAIS do not have ambiguous genitalia, nor do they have Müllerian structures. Without this exception clause in the PDF, the hospital could not offer testing to any man with MAIS.
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