There's so much bullshit going around about statins these days. They are wonderful drugs. Unfortunately, the way they are typically prescribed is idiotic and leads to a greater prevalence of side effects than would otherwise be necessary. All of the clinical trials were oriented toward prevention of MACE (major adverse cardiac events) i.e. folks that already had ASCVD and were likely to die from it. The problem being that MACE is a symptom and if one were prescribing it as a means to prevent ASCVD the doses would be at a much more reasonable spot on the dose response curve.
At higher doses, statins can cause fibromyalgia and insulin resistance. Folks on atorvastatin 80mg (max dose) have a 1 in 3 chance of developing type 2 diabetes. It really is nuts.
In any case, there are a couple classes of statin, lipophilic and hydrophilic. The former has a more systemic response and has a greater chance of side effects. The later is more targeted and has a smaller chance of side effects. The most popular of these are atorvastatin and rosuvastatin respectively. Typically rosuvastatin at low doses has a smaller incidence of side effects, but some folks do switch from rosuvastatin to atorvastatin with success.
For rosuvastatin, 85% of the maximal effectiveness occurs around 10mg though prescribing information suggests doses up to 40mg. A google image search for "statin dose response curve" will yield some interesting results. A 5mg dose is surprisingly effective. Some even run it EOD with success.
Personally, I run 10mg rosuvastatin, 10mg ezetimibe, and 180mg bempedoic acid. I have no side effects and have the lipid profile of a health high school student. I'm doing all this because I had a CT-CAC a few years back and discovered some calcified plaque. If I keep lipids very low for some years, it may regress. I've recently added repatha to the mix to see how that works as well.
Even on cycle, my lipids are golden. My HDL goes down some, but I supplement 4g EPA daily plus 1000mg citrus bergamot, aged garlic extract, and so on.