how to make transdermal testosterone?

DHT isn't the culprit in hair loss.
Estrogens and androsterone are.
Genetics is the main factor of hair loss. If you want to see where you will be later in life (or sooner with aas use) look at your mother's father and his hairline.

Androsterone just exacerbates genetic predisposition
 
Genetics is the main factor of hair loss. If you want to see where you will be later in life (or sooner with aas use) look at your mother's father and his hairline.

Androsterone just exacerbates genetic predisposition

Regardless of whether you have the genes to lose hair, high doses of androsterone and estrogens will make you thin out.
 
Regardless of whether you have the genes to lose hair, high doses of androsterone and estrogens will make you thin out.
I've noticed that most guys on TRT eventually end up with diffuse hair thinning even when they keep their estrogen level low.
Is this because androsterone levels remain constantly elevated?

Does hCG increase both estrogen and aldosterone?
I've always experienced hair loss, especially frontal hair loss from hCG.
 
Very much so.

My blood alcohol levels after applying 3ml of my solution 3 times a day is still 0.

Should I be worried about something, Chris ?

That is your systematic level though not local tissue.... are you not concerned about the ethanol toxicity locally to your testes given it's 96% ethanol? Testes are one of the most sensitive tissues in the human body. Genuine questions thats all
 
Then why do 5 alpha reductase inhibitors like finasteride and dutasteride slow down hair loss?

5ARis decreases androsterone along with a host of other androgens. This was shown in studies. Pretty much the only androgen that doesn't decrease on fin is testosterone as it increases.
 
That is your systematic level though not local tissue.... are you not concerned about the ethanol toxicity locally to your testes given it's 96% ethanol? Testes are one of the most sensitive tissues in the human body. Genuine questions thats all

The word you're looking for is "systemic", not "systematic".

My balls are doing fine, genuine answer.
 
Then why do 5 alpha reductase inhibitors like finasteride and dutasteride slow down hair loss?

5ar type II converts DHEA into androstenediol, androstenedione and androsterone.

When you inhibit 5ar type II you have no idea which androgens are lowered, maybe all of the above + DHT.
 
5ar type II converts DHEA into androstenediol, androstenedione and androsterone.

When you inhibit 5ar type II you have no idea which androgens are lowered, maybe all of the above + DHT.
Thanks.
Out of interest, have you taken a 5ar inhibitor in the past?
I was wondering if that was what drove you to experiment with these libido boosting topical formulas?
 
Thanks.
Out of interest, have you taken a 5ar inhibitor in the past?
I was wondering if that was what drove you to experiment with these libido boosting topical formulas?

Never.

The only thing that drives me towards topical formulations is the certainty that injecting anything oil-related with some benzyl benzoate in your muscles can't be good for health.
 
Does Accutane do the same?
it can. Post fin, post accutane, post AI all are very similar IMO in the sense of inhibiting certain enzyme functions
 
DMSO is completely liquid.

I have two different testosterone delivery methods:

1)There are litterally only two ingredients in this solution: testosterone base powder + liquid DMSO.

2) The second one is the cream: Testosterone base + pentravan cream + isopropyl alcohol

I alternate between the two depending on need.
Can you explain the difference between these? I have been on 200mg/ml cream for 2 years now, 3 clicks on scrotum morning and night. It was a game changer for my trt. Total test stays slightly over top range, free test extremely high, estrogen slightly elevated, but no symptoms whatsoever because my dht is probably very high as well. Just wondering how the 2 compare.
 
Can you explain the difference between these? I have been on 200mg/ml cream for 2 years now, 3 clicks on scrotum morning and night. It was a game changer for my trt. Total test stays slightly over top range, free test extremely high, estrogen slightly elevated, but no symptoms whatsoever because my dht is probably very high as well. Just wondering how the 2 compare.

Stay on the cream, DMSO is a heavy metal chelator and could make you feel worse.
Also you'd have to apply it on a skin that's always perfectly clean.
 
Does anyone have a good list of pros/cons of Test P or Test C vs. Test Base?

Esters don't make it past the skin layers when applied transdermally.

Test base is the only viable option.
 
Stay on the cream, DMSO is a heavy metal chelator and could make you feel worse.
Also you'd have to apply it on a skin that's always perfectly clean.
I think after reading this whole thread I'm more interested in the recipe that you've been talked about earlier using ethanol and myristate. I've always been fascinated and scared of dmso, even thinking about it carrying in stuff that's in tap water. I like the idea of making a transdermal with test, dht or dhea, and nandrolone base if I could find any.
 
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