Palmitic acid - coconut oil

[LW64]

No progression is good news.

From an optimistic standpoint, maybe it needs to be there for a while before it reverses.

 

[ergomaniac]

The influence of the type of d... [Int J Obes Relat Metab Disord. 2002] - PubMed - NCBI
Substituting dietary saturated for monounsatura... [Diabetologia. 2001] - PubMed - NCBI
Type of dietary fat and insulin resistance. [Ann N Y Acad Sci. 2002] - PubMed - NCBI
Substitution of saturated with monounsaturated fat... [Br J Nutr. 2003] - PubMed - NCBI
Fatty acids and insulin sensi... [Curr Opin Clin Nutr Metab Care. 2008] - PubMed - NCBI

any credence to any of this. that high amounts sfas impair insulin sensitivity. ive been reading this for years. as well as too many carbs converting into sfas and hfcs making it all worse since fructose can only be stored as glycogen in one place. insulin resistance and cvd seem to be tied together.
the last one is relevant to this post. these are just some in one journal. it goes on and on. not arguing about high fat diets. just which fatty acids.

 

[zkt]

No progression is good news.

From an optimistic standpoint, maybe it needs to be there for a while before it reverses.

Yep. The glass is always full: Half air- half water. Its interesting that when I started the statin in 2006 the RICA was <50% and I ate a low fat, low to medium carb diet, more or less Ornish style and by 2009 it had increased to its current level and has held constant until the present. It was during 2010 that I was on 8mg/day medrol: no progression. During 2011 had stopped the medrol and ate Paleo; again, no progression.
I dont have much to back me up but since the immune system is intimately involved in the atherosclerotic process toning its activity down a bit might have more benefits than drawbacks.
Hell, I`ve already had most of the side effects already; the osteopenia is controlled by alendronate, bilateral glaucoma- fixed that, thinning of the dermis- that is a bit of an ongoing problem, T2D: I doubt it, but at this point its a race to the finish line anyway. lol. LOL.

 

[LW64]

The influence of the type of d... [Int J Obes Relat Metab Disord. 2002] - PubMed - NCBI
Substituting dietary saturated for monounsatura... [Diabetologia. 2001] - PubMed - NCBI
Type of dietary fat and insulin resistance. [Ann N Y Acad Sci. 2002] - PubMed - NCBI
Substitution of saturated with monounsaturated fat... [Br J Nutr. 2003] - PubMed - NCBI
Fatty acids and insulin sensi... [Curr Opin Clin Nutr Metab Care. 2008] - PubMed - NCBI

any credence to any of this. that high amounts sfas impair insulin sensitivity. ive been reading this for years. as well as too many carbs converting into sfas and hfcs making it all worse since fructose can only be stored as glycogen in one place. insulin resistance and cvd seem to be tied together.
the last one is relevant to this post. these are just some in one journal. it goes on and on. not arguing about high fat diets. just which fatty acids.

Without looking at all of it in detail ergo, there's credence to all of it. The reason for that is so long as carb intake is high (40, 50, or 60 % of total calories, which is where most of the lipophobic researchers think carb intake should be) the metabolic response to that kind of diet will be to make a 'high' fat intake dangerous when it's greater than something like 20 % of total calories.

The bottom line is that if someone wants to eat a diet where carbs are in the range of 40/50/60 % as total calories, they had better keep their fat intake low and/or make the kinds of fat substitutions they discuss in the references. Yes, there's a reduction in risk but there may be a better way...

The metabolic response picture changes completely when carbs are restricted and fat intake is increased. The reductions in CVD risk measurements are superior to those seen in low-fat diets.

 

[LW64]

Yep. The glass is always full: Half air- half water. Its interesting that when I started the statin in 2006 the RICA was <50% and I ate a low fat, low to medium carb diet, more or less Ornish style and by 2009 it had increased to its current level and has held constant until the present. It was during 2010 that I was on 8mg/day medrol: no progression. During 2011 had stopped the medrol and ate Paleo; again, no progression. LOL.

I was going to ask about your diet habits over the last few years. With Ornish, you get rid of sugar in the diet. With Paleo, you get rid of sugar in the diet and a lot of the carbs that become glucose after digestion.

 

[ergomaniac]

Without looking at all of it in detail ergo, there's credence to all of it. The reason for that is so long as carb intake is high (40, 50, or 60 % of total calories, which is where most of the lipophobic researchers think carb intake should be) the metabolic response to that kind of diet will be to make a 'high' fat intake dangerous when it's greater than something like 20 % of total calories.

The bottom line is that if someone wants to eat a diet where carbs are in the range of 40/50/60 % as total calories, they had better keep their fat intake low and/or make the kinds of fat substitutions they discuss in the references. Yes, there's a reduction in risk but there may be a better way...

The metabolic response picture changes completely when carbs are restricted and fat intake is increased. The reductions in CVD risk measurements are superior to those seen in low-fat diets.

so in other words, if youre not eating many carbs, insulin sensitivty isnt as important. me cycling carbs for athletic/ bodybuilding purposes, i find that it is.
were on two different diets i guess. sort of. i just include carbs at certain times.

 

[LW64]

so in other words, if youre not eating many carbs, insulin sensitivty isnt as important. me cycling carbs for athletic/ bodybuilding purposes, i find that it is.
were on two different diets i guess. sort of. i just include carbs at certain times.

It has more to do with where, on average, your metabolism is. I would expect that adding in carbs as a part of an excercise routine will not have the same deleterious effects that it could have if you were inactive. Since you're improving your muscle insulin sensitivity with the excercise, my guess - and it's only a guess - is that the carb loading is not a concern unless there are signs of metabolic syndrome: high BP, T2D, overweight, etc.

 

[ergomaniac]

It has more to do with where, on average, your metabolism is. I would expect that adding in carbs as a part of an excercise routine will not have the same deleterious effects that it could have if you were inactive. Since you're improving your muscle insulin sensitivity with the excercise, my guess - and it's only a guess - is that the carb loading is not a concern unless there are signs of metabolic syndrome: high BP, T2D, overweight, etc.

so exercise, particularly resistance exercise, will improve insulin sensitivity. timing those carbs eaten will prevent fat accumulation by storing them as glycogen. things like cinnamon and stevia increase insulin sensitvity as well as other supplements, i can think of a few. plus cinnamon and stevia taste good. sfas lower insulin resistance so im not looking for extras. coconut oil has other good things in it and i actually eat whole coconut. other food fats seem well balanced. same reason i dont take fish oil and eat some fish. sfas have something to do with particle size including vldls which an increase of is a marker for the begining of insulin resistance and t2d. all this seems related. this isnt a fight, im genuinely interested. some things just seem to be overlooked.