Post up your Natty IGF-1 Level

[Trenbolonetax]

The tirzepatide might be controlling your blood sugar... but it takes a ton of insulin to do it, because of the GH-induced insulin resistance. I like that you tested fasting insulin and not just fasting glucose - far more useful test to identify insulin resistance.

I just finished reading Ben Bikman's book Why We Get Sick, which makes a compelling argument that insulin resistance is the primary underlying cause of most chronic diseases and causes of death (heart disease, cancer, dementia, diabetes obviously). I would challenge anyone who thinks GH for the amateur BB enthusiast is a remotely good idea to read the book.

I say leave it to the pros.

https://www.amazon.com/Why-We-Get-Sick-Disease-ebook/dp/B07ZKZ2NRN/

I love the fact that Ben's name got mentioned here.

Have you checked out any of Nick Norowitz, Dom D'Agostino, Dave Feldman, or other folks in their sphere of health work on metabolism / cholesterol / keto?

Something I found pretty interesting was when I was on 16iu of GH + 15mg Tirz and a low carb diet my fasting insulin came back less than 3 (forgot the units but they are standard freedom units). That was pretty surprising to me considering I had injected 16iu of GH that same morning I got the bloodwork done.

Questions that I ponder frequently: How much is one blunting the positive impact of GH by doing IF / low carb diet / keto diet? How much is one negating the deleterious impact of GH by doing the same? Can one reap most of the fat loss and soft tissue / recovery benefits of GH despite not receiving a IGF 1 boost as a result of fasting / very low carb diet?

 

[zmurphy]

I love the fact that Ben's name got mentioned here.

Have you checked out any of Nick Norowitz, Dom D'Agostino, Dave Feldman, or other folks in their sphere of health work on metabolism / cholesterol / keto?

Something I found pretty interesting was when I was on 16iu of GH + 15mg Tirz and a low carb diet my fasting insulin came back less than 3 (forgot the units but they are standard freedom units). That was pretty surprising to me considering I had injected 16iu of GH that same morning I got the bloodwork done.

Questions that I ponder frequently: How much is one blunting the positive impact of diet? How GH by doing IF / low carb diet / keto much is one negating the deleterious impact of GH by doing the same? Can one reap most of the fat loss and soft tissue / recovery benefits of GH despite not receiving a IGF 1 boost as a result of fasting / very low carb diet?

Did you ever get an IGF-1 level on the 16iu?

 

[Trenbolonetax]

Did you ever get an IGF-1 level on the 16iu?

Yeah while on zero AAS + 16iu GH + 15mg tirz it was 218

 

[Jin23]

Something I found pretty interesting was when I was on 16iu of GH + 15mg Tirz and a low carb diet my fasting insulin came back less than 3 (forgot the units but they are standard freedom units).

I wanted to say that that sound impossible. But then:

Yeah while on zero AAS + 16iu GH + 15mg tirz it was 218

These are unmanaged diabetes numbers. I hope you stopped using gh?

Have you measured bg and insulin while beeing off of everything?

 

[Trenbolonetax]

I wanted to say that that sound impossible. But then:



These are unmanaged diabetes numbers. I hope you stopped using gh?

Have you measured bg and insulin while beeing off of everything?

I'm sorry I don't quite understand how you got to the conclusion that it's unmanaged diabetes based on the information I provided?

 

[AlexDavis43]

I love the fact that Ben's name got mentioned here.

Have you checked out any of Nick Norowitz, Dom D'Agostino, Dave Feldman, or other folks in their sphere of health work on metabolism / cholesterol / keto?

The guys who say people that respond to keto with ultra-high apoB levels are just fine. Mostly driven by Dave the engineer and Nick the fake PhD. Ben drank all the keto kool-aid. Dom is usually more sensible.

 

[FunkOdyssey]

Have you checked out any of Nick Norowitz, Dom D'Agostino, Dave Feldman, or other folks in their sphere of health work on metabolism / cholesterol / keto?

I've begun to, yes. Fascinating alternative perspectives so far that better explain and agree with observed reality.

Something I found pretty interesting was when I was on 16iu of GH + 15mg Tirz and a low carb diet my fasting insulin came back less than 3 (forgot the units but they are standard freedom units). That was pretty surprising to me considering I had injected 16iu of GH that same morning I got the bloodwork done.

That is shockingly low for 16 IU GH. I have a fasting insulin of 3 on a carnivore diet with no GH. I wonder how much of a role your elaborate / cutting edge PED stack is playing here.

Questions that I ponder frequently: How much is one blunting the positive impact of GH by doing IF / low carb diet / keto diet? How much is one negating the deleterious impact of GH by doing the same? Can one reap most of the fat loss and soft tissue / recovery benefits of GH despite not receiving a IGF 1 boost as a result of fasting / very low carb diet?

Great questions. I would guess that you are blunting both the benefits and the damage of GH with the IF / keto diet. If @Type-IIx ever releases his book, I bet the answers are in there.

 

[Trenbolonetax]

I've begun to, yes. Fascinating alternative perspectives so far that better explain and agree with observed reality.

No kidding lol.

That is shockingly low for 16 IU GH. I have a fasting insulin of 3 on a carnivore diet with no GH. I wonder how much of a role your elaborate / cutting edge PED stack is playing here.

I was completely shocked, too. At the time, there was nothing other than the 16iu GH + 15mg tirz + 20mg cardarine + 25mg enclomiphene + 80mg telmisartan + 200mcg t4; no SLUPPP, no 5Amino, nothing else. I was cycling minimum 70 miles per week, up to 150miles. And was on a pretty strict diet + deficit.

I think the fasting insulin result supports the notion that these GLP1s are true insulin sensitizing agents and don't necessarily only work by making pancreas secrete more insulin but it's rather multifaceted:

1) increase insulin independent glucose uptake (substantially)
2) directly increase insulin sensitivity (same amount of insulin has larger subsequent drop in BG)
3) increased insulin secretion compared to baseline if still necessary after above 2 points

Great questions. I would guess that you are blunting both the benefits and the damage of GH with the IF / keto diet. If @Type-IIx ever releases his book, I bet the answers are in there.

I am eagerly awaiting the book! Very excited to dig in.

I think you're right. My opinion is that I'm blunting the potential positive mass building characteristics but improving fat loss characteristics.

 

[Trenbolonetax]

The guys who say people that respond to keto with ultra-high apoB levels are just fine. Mostly driven by Dave the engineer and Nick the fake PhD. Ben drank all the keto kool-aid. Dom is usually more sensible.

I take it you're not a fan. What folks would you recommend reading about / listening to that are doing work on metabolism / cholesterol / keto?

 

[AlexDavis43]

I take it you're not a fan. What folks would you recommend reading about / listening to that are doing work on metabolism / cholesterol / keto?

Me but I'm anon here. Lol. Published a few books and studies on metabolism & keto. Many podcasts too. Those guys aren't that bad imo, btw, and I'm also pro-keto. Just not as zealous as them.